PMID-sentid	Pub_year	Sent_text	comp_official_name	comp_offset	protein_name	organism	prot_offset
29383102-7	2017	Elevated expression of BANCR induced JNK activation, which can be decreased by the specific JNK inhibitor SP600125.	pyrazolanthrone	106-114	BRAF-activated non-protein coding RNA	Homo sapiens	23-28
31966836-0	2017	Luteolin suppresses tumor progression through lncRNA BANCR and its downstream TSHR/CCND1 signaling in thyroid carcinoma.	Luteolin	0-8	BRAF-activated non-protein coding RNA	Homo sapiens	53-58
28969673-8	2017	Interestingly, for the group of patients with the lymph node metastasis, we found the similar result that high lncRNA BANCR expression was related to poor OS (P = 0.004).	Osmium	155-157	BRAF-activated non-protein coding RNA	Homo sapiens	118-123
31966836-6	2017	We found that luteolin decreased the expression of BRAF-activated long noncoding RNA (BANCR), which further led to downregulation of TSHR and downstream oncogenic signaling.	Luteolin	14-22	BRAF-activated non-protein coding RNA	Homo sapiens	51-84
31966836-6	2017	We found that luteolin decreased the expression of BRAF-activated long noncoding RNA (BANCR), which further led to downregulation of TSHR and downstream oncogenic signaling.	Luteolin	14-22	BRAF-activated non-protein coding RNA	Homo sapiens	86-91
31966836-7	2017	Moreover, overexpression of BANCR/TSHR signaling can largely abolish the anti-tumor effects of luteolin on thyroid carcinoma in vitro and in vivo.	Luteolin	95-103	BRAF-activated non-protein coding RNA	Homo sapiens	28-33
31966836-8	2017	In conclusion, luteolin may serve as a potential important anticancer agent for thyroid carcinoma by blocking the BANCR/TSHR signaling.	Luteolin	15-23	BRAF-activated non-protein coding RNA	Homo sapiens	114-119
26296467-0	2015	Fentanyl inhibits the invasion and migration of colorectal cancer cells via inhibiting the negative regulation of Ets-1 on BANCR.	Fentanyl	0-8	BRAF-activated non-protein coding RNA	Homo sapiens	123-128
26296467-3	2015	Since studies indicates the abnormal expression of transcription factor Ets-1 and BRAF-activated lncRNA (BANCR) in CRC progress, the relationship between Ets-1 and BANCR was investigated here to illustrate the fentanyl-induced mechanism on CRC in vitro.	Fentanyl	210-218	BRAF-activated non-protein coding RNA	Homo sapiens	105-110
26296467-3	2015	Since studies indicates the abnormal expression of transcription factor Ets-1 and BRAF-activated lncRNA (BANCR) in CRC progress, the relationship between Ets-1 and BANCR was investigated here to illustrate the fentanyl-induced mechanism on CRC in vitro.	Fentanyl	210-218	BRAF-activated non-protein coding RNA	Homo sapiens	164-169
26296467-4	2015	METHODS: The expression levels of Ets-1 and BANCR were first detected in fentanyl-treated CRC cells.	Fentanyl	73-81	BRAF-activated non-protein coding RNA	Homo sapiens	44-49
26296467-8	2015	Ets-1 overexpression or co-overexpression with BANCR was further performed by plasmids transfection to show the regulatory role of Ets-1 in fentanyl-induced mechanism.	Fentanyl	140-148	BRAF-activated non-protein coding RNA	Homo sapiens	47-52
26296467-9	2015	RESULTS: Fentanyl induced BANCR upregulation and Ets-1 downregulation in CRC cells.	Fentanyl	9-17	BRAF-activated non-protein coding RNA	Homo sapiens	26-31
26296467-11	2015	Moreover, fentanyl induced less cell clone formation, as well as inhibited cell migration and invasion in vitro, while Ets-1 overexpression inhibited fentanyl-induced effects that could be reversed by BANCR co-overexpression.	Fentanyl	150-158	BRAF-activated non-protein coding RNA	Homo sapiens	201-206
26248136-8	2015	MiroRNA-9 (miR-9) targeted NF-kappaB1, and miR-9 inhibitor also reversed the effects of BANCR on gastric cancer cell growth and apoptosis.	mirorna-9	0-9	BRAF-activated non-protein coding RNA	Homo sapiens	88-93
26248136-8	2015	MiroRNA-9 (miR-9) targeted NF-kappaB1, and miR-9 inhibitor also reversed the effects of BANCR on gastric cancer cell growth and apoptosis.	mir-9	11-16	BRAF-activated non-protein coding RNA	Homo sapiens	88-93
26296467-13	2015	Furthermore, the regulatory role of Ets-1 on BANCR influenced fentanyl-induced mechanism, indicating their potential application in the therapeutic treatment of CRC.	Fentanyl	62-70	BRAF-activated non-protein coding RNA	Homo sapiens	45-50
34512168-0	2021	Rutin attenuates Sorafenib-induced Chemoresistance and Autophagy in Hepatocellular Carcinoma by regulating BANCR/miRNA-590-5P/OLR1 Axis.	Rutin	0-5	BRAF-activated non-protein coding RNA	Homo sapiens	107-112
25013510-7	2014	Further investigation into the underlying mechanisms responsible for the migratory effects revealed that BANCR induced the epithelial-mesenchymal transition (EMT) through an MEK/extracellular signal-regulated kinase-dependent mechanism as treatment with the MEK inhibitor, U0126 decreased migration and reversed the EMT in the BANCR-overexpressed HCT116 cells.	U 0126	273-278	BRAF-activated non-protein coding RNA	Homo sapiens	105-110
25013510-7	2014	Further investigation into the underlying mechanisms responsible for the migratory effects revealed that BANCR induced the epithelial-mesenchymal transition (EMT) through an MEK/extracellular signal-regulated kinase-dependent mechanism as treatment with the MEK inhibitor, U0126 decreased migration and reversed the EMT in the BANCR-overexpressed HCT116 cells.	U 0126	273-278	BRAF-activated non-protein coding RNA	Homo sapiens	327-332
24967732-6	2014	Moreover, combination treatment of BANCR knockdown and suppression ERK1/2 or JNK (induced by specific inhibitors U0126 or SP600125 respectively) produced synergistic inhibitory effects in vitro.	U 0126	113-118	BRAF-activated non-protein coding RNA	Homo sapiens	35-40
24967732-6	2014	Moreover, combination treatment of BANCR knockdown and suppression ERK1/2 or JNK (induced by specific inhibitors U0126 or SP600125 respectively) produced synergistic inhibitory effects in vitro.	pyrazolanthrone	122-130	BRAF-activated non-protein coding RNA	Homo sapiens	35-40
25893737-8	2015	These results indicated that baicalein inhibited osteosarcoma cell proliferation and promoted apoptosis by targeting c-MYC gene through Wnt signaling, in which JNK and BANCR were also involved as well as beta-catenin, suggesting a new potential mechanism for us to better understand the inhibiting effect of baicalein on osteosarcoma.	baicalein	29-38	BRAF-activated non-protein coding RNA	Homo sapiens	168-173
34512168-0	2021	Rutin attenuates Sorafenib-induced Chemoresistance and Autophagy in Hepatocellular Carcinoma by regulating BANCR/miRNA-590-5P/OLR1 Axis.	Sorafenib	17-26	BRAF-activated non-protein coding RNA	Homo sapiens	107-112
34512168-7	2021	Our results indicated that rutin treatment attenuates autophagy and BANCR expression in SO resistance cells.	Rutin	27-32	BRAF-activated non-protein coding RNA	Homo sapiens	68-73
34512168-11	2021	Furthermore, in vivo study demonstrated that rutin could inhibit autophagy through the BANCR/miRNA-590-5P/OLR1 axis.	Rutin	45-50	BRAF-activated non-protein coding RNA	Homo sapiens	87-92
34512168-12	2021	Our findings suggest that rutin could regulate autophagy by regulating BANCR/miRNA-590-5P/OLR1 axis.	Rutin	26-31	BRAF-activated non-protein coding RNA	Homo sapiens	71-76
33336751-1	2020	OBJECTIVE: To investigate the therapeutic effects of ticagrelor and clopidogrel on patients with acute myocardial infarction (AMI) and its effect of lncRNA BANCR.	Ticagrelor	53-63	BRAF-activated non-protein coding RNA	Homo sapiens	156-161
34522245-9	2021	In addition, upregulated lncRNA BANCR reduced or postponed cell sensitivity to cisplatin by enhancing cell proliferation in TU686 and TU177 cells.	Cisplatin	79-88	BRAF-activated non-protein coding RNA	Homo sapiens	32-37
34522245-11	2021	After cisplatin treatment, down-regulation of BANCR could consequently attenuate TU686-DDP-R and TU177-DDP-R cell proliferation, and the expression of MRP1, Bcl-2, and p-PKB was decreased and Bax was increased.	Cisplatin	6-15	BRAF-activated non-protein coding RNA	Homo sapiens	46-51
34522245-12	2021	Conclusions: Down-regulation of BANCR reverses cisplatin resistance of cisplatin-resistant LSCC cell lines.	Cisplatin	47-56	BRAF-activated non-protein coding RNA	Homo sapiens	32-37
34522245-12	2021	Conclusions: Down-regulation of BANCR reverses cisplatin resistance of cisplatin-resistant LSCC cell lines.	Cisplatin	71-80	BRAF-activated non-protein coding RNA	Homo sapiens	32-37
33880577-10	2021	Notably, U0126, a specific MEK inhibitor, decreased MEK and ERK expression, and blocked the promotive effects of BANCR overexpression on the proliferation, migration and invasion of ESCC cells.	U 0126	9-14	BRAF-activated non-protein coding RNA	Homo sapiens	113-118
33469371-8	2021	Ly3214996, an inhibitor of ERK signal pathway, was administered to assess the effects of BANCR overexpression on gastric cancer cell with cisplatin-treated resistance.	Cisplatin	138-147	BRAF-activated non-protein coding RNA	Homo sapiens	89-94
33469371-9	2021	Moreover, the role of BANCR in cisplatin resistance of gastric cancer was validated in xenograft mouse models in vivo.	Cisplatin	31-40	BRAF-activated non-protein coding RNA	Homo sapiens	22-27
33469371-11	2021	Furthermore, we found that BANCR overexpression promoted gastric cancer cell resistance to cisplatin in vitro.	Cisplatin	91-100	BRAF-activated non-protein coding RNA	Homo sapiens	27-32
33469371-12	2021	Ly3214996 treatment abolished the BANCR overexpression-mediated gastric cancer cell cisplatin resistance via regulating the phosphorylation of ERK protein.	LY3214996	0-9	BRAF-activated non-protein coding RNA	Homo sapiens	34-39
33469371-12	2021	Ly3214996 treatment abolished the BANCR overexpression-mediated gastric cancer cell cisplatin resistance via regulating the phosphorylation of ERK protein.	Cisplatin	84-93	BRAF-activated non-protein coding RNA	Homo sapiens	34-39
33469371-14	2021	Conclusion: BANCR promoted cisplatin resistance of gastric cancer cells by activating ERK1/2 pathway.	Cisplatin	27-36	BRAF-activated non-protein coding RNA	Homo sapiens	12-17
31007747-8	2019	However, lncRNA BANCR overexpression inhibited the apoptosis of ARPE-19 cells under high-glucose conditions.	Glucose	89-96	BRAF-activated non-protein coding RNA	Homo sapiens	16-21
32945416-14	2020	On the whole, the findings of the present study demonstrate that BANCR inhibition blocks ESCC progression by inactivating the IGF1R/Raf/MEK/ERK pathway by sponging miR-338-3p.	mir-338-3p	164-174	BRAF-activated non-protein coding RNA	Homo sapiens	65-70
31482398-10	2020	Besides, miR-204-3p and Wnt/beta-catenin signalling pathway were found to participate in the oncogenic effects of BANCR on RB cell line by Hoechst staining, cell Counting Kit-8 (CCK-8) assay, wound healing assay, transwell assay, and Western blot analysis in vitro.	hoechst	139-146	BRAF-activated non-protein coding RNA	Homo sapiens	114-119
30996220-7	2019	High-glucose treatment led to upregulated expression of BANCR in the human retinal pigment epithelial cell line ARPE-19.	Glucose	5-12	BRAF-activated non-protein coding RNA	Homo sapiens	56-61
30996220-8	2019	Overexpression of BANCR promoted and siRNA silencing inhibited the apoptosis of cells in the human retinal pigment epithelial cell line ARPE-19 in a high-glucose environment.	Glucose	154-161	BRAF-activated non-protein coding RNA	Homo sapiens	18-23
30144787-0	2018	LncRNA BANCR promotes tumorigenesis and enhances adriamycin resistance in colorectal cancer.	Doxorubicin	49-59	BRAF-activated non-protein coding RNA	Homo sapiens	7-12
30144787-3	2018	The roles and molecular basis of long non-coding RNA BRAF-activated noncoding RNA (BANCR) in CRC progression and adriamycin (ADR) resistance have not been extensively identified.	Doxorubicin	113-123	BRAF-activated non-protein coding RNA	Homo sapiens	83-88