PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9580328-4	1998	Overexpression of Bcl-2 and pretreatment with either the immunosuppressant cyclosporin A or the glutathione precursor N-acetyl-L-cysteine blocked deltapsi(m) disruption and apoptosis, but not the generation of ROS induced by these compounds.	Cyclosporine	75-88	BCL2 apoptosis regulator	Homo sapiens	18-23	
9580328-4	1998	Overexpression of Bcl-2 and pretreatment with either the immunosuppressant cyclosporin A or the glutathione precursor N-acetyl-L-cysteine blocked deltapsi(m) disruption and apoptosis, but not the generation of ROS induced by these compounds.	Glutathione	96-107	BCL2 apoptosis regulator	Homo sapiens	18-23	
9580328-4	1998	Overexpression of Bcl-2 and pretreatment with either the immunosuppressant cyclosporin A or the glutathione precursor N-acetyl-L-cysteine blocked deltapsi(m) disruption and apoptosis, but not the generation of ROS induced by these compounds.	Acetylcysteine	118-137	BCL2 apoptosis regulator	Homo sapiens	18-23	
9580328-4	1998	Overexpression of Bcl-2 and pretreatment with either the immunosuppressant cyclosporin A or the glutathione precursor N-acetyl-L-cysteine blocked deltapsi(m) disruption and apoptosis, but not the generation of ROS induced by these compounds.	Reactive Oxygen Species	210-213	BCL2 apoptosis regulator	Homo sapiens	18-23	
9580328-5	1998	Capsaicin and resiniferatoxin were found to activate both isoforms of c-jun-NH2-kinase (JNK), with a maximal activity after 30 min of treatment.	Capsaicin	0-9	mitogen-activated protein kinase 8	Homo sapiens	70-86	
9580328-5	1998	Capsaicin and resiniferatoxin were found to activate both isoforms of c-jun-NH2-kinase (JNK), with a maximal activity after 30 min of treatment.	Capsaicin	0-9	mitogen-activated protein kinase 8	Homo sapiens	88-91	
9580328-5	1998	Capsaicin and resiniferatoxin were found to activate both isoforms of c-jun-NH2-kinase (JNK), with a maximal activity after 30 min of treatment.	resiniferatoxin	14-29	mitogen-activated protein kinase 8	Homo sapiens	70-86	
9580328-5	1998	Capsaicin and resiniferatoxin were found to activate both isoforms of c-jun-NH2-kinase (JNK), with a maximal activity after 30 min of treatment.	resiniferatoxin	14-29	mitogen-activated protein kinase 8	Homo sapiens	88-91	
9580328-8	1998	We suggest that ROS generation by inhibition of the NADH-dependent plasma membrane electron transport system resulted in the oxidation of mitochondrial megachannel pores that allows for the disruption of deltapsi(m) and apoptosis, and that AP-1 activation is not required for vanilloid-induced apoptosis.	Reactive Oxygen Species	16-19	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	240-244	
9580328-8	1998	We suggest that ROS generation by inhibition of the NADH-dependent plasma membrane electron transport system resulted in the oxidation of mitochondrial megachannel pores that allows for the disruption of deltapsi(m) and apoptosis, and that AP-1 activation is not required for vanilloid-induced apoptosis.	NAD	52-56	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	240-244