PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9401960-0	1997	Evidence of two mechanisms for the activation of the glucose transporter GLUT1 by anisomycin: p38(MAP kinase) activation and protein synthesis inhibition in mammalian cells.	Anisomycin	82-92	solute carrier family 2 member 1	Homo sapiens	73-78	
9401960-0	1997	Evidence of two mechanisms for the activation of the glucose transporter GLUT1 by anisomycin: p38(MAP kinase) activation and protein synthesis inhibition in mammalian cells.	Anisomycin	82-92	mitogen-activated protein kinase 14	Homo sapiens	94-108	
9401960-2	1997	Inhibitors of protein synthesis stimulate sugar transport in mammalian cells through activation of plasma membrane GLUT1, the housekeeping isoform of the glucose transporter.	Sugars	42-47	solute carrier family 2 member 1	Homo sapiens	115-120	
9401960-7	1997	In contrast, stimulation by anisomycin, a potent Jun-NH2-terminal kinase (JNK) agonist, exhibited no lag phase.	Anisomycin	28-38	mitogen-activated protein kinase 8	Homo sapiens	49-72	
9401960-7	1997	In contrast, stimulation by anisomycin, a potent Jun-NH2-terminal kinase (JNK) agonist, exhibited no lag phase.	Anisomycin	28-38	mitogen-activated protein kinase 8	Homo sapiens	74-77	
9401960-11	1997	The stimulation of transport by a low concentration of anisomycin (0.3 microM) was transient, peaked at 30-60 min and it was inhibited (IC50 < 1 microM) by SB203580, which indicates that its mediator is not JNK, but the homologous p38(MAP kinase) (p38(MAPK)).	Anisomycin	55-65	mitogen-activated protein kinase 8	Homo sapiens	210-213	
9401960-11	1997	The stimulation of transport by a low concentration of anisomycin (0.3 microM) was transient, peaked at 30-60 min and it was inhibited (IC50 < 1 microM) by SB203580, which indicates that its mediator is not JNK, but the homologous p38(MAP kinase) (p38(MAPK)).	Anisomycin	55-65	mitogen-activated protein kinase 14	Homo sapiens	234-249	
9401960-11	1997	The stimulation of transport by a low concentration of anisomycin (0.3 microM) was transient, peaked at 30-60 min and it was inhibited (IC50 < 1 microM) by SB203580, which indicates that its mediator is not JNK, but the homologous p38(MAP kinase) (p38(MAPK)).	Anisomycin	55-65	mitogen-activated protein kinase 14	Homo sapiens	251-260	
9401960-11	1997	The stimulation of transport by a low concentration of anisomycin (0.3 microM) was transient, peaked at 30-60 min and it was inhibited (IC50 < 1 microM) by SB203580, which indicates that its mediator is not JNK, but the homologous p38(MAP kinase) (p38(MAPK)).	SB 203580	159-167	mitogen-activated protein kinase 8	Homo sapiens	210-213	
9401960-11	1997	The stimulation of transport by a low concentration of anisomycin (0.3 microM) was transient, peaked at 30-60 min and it was inhibited (IC50 < 1 microM) by SB203580, which indicates that its mediator is not JNK, but the homologous p38(MAP kinase) (p38(MAPK)).	SB 203580	159-167	mitogen-activated protein kinase 14	Homo sapiens	234-249	
9401960-11	1997	The stimulation of transport by a low concentration of anisomycin (0.3 microM) was transient, peaked at 30-60 min and it was inhibited (IC50 < 1 microM) by SB203580, which indicates that its mediator is not JNK, but the homologous p38(MAP kinase) (p38(MAPK)).	SB 203580	159-167	mitogen-activated protein kinase 14	Homo sapiens	251-260	
9401960-14	1997	Exposure to anisomycin resulted in rapid activation of p38(MAPK).	Anisomycin	12-22	mitogen-activated protein kinase 14	Homo sapiens	55-58	
9401960-14	1997	Exposure to anisomycin resulted in rapid activation of p38(MAPK).	Anisomycin	12-22	mitogen-activated protein kinase 14	Homo sapiens	59-63	
9401960-15	1997	Activation of both p38(MAPK) and GLUT1 by 0.3 microM anisomycin was cancelled by puromycin.	Anisomycin	53-63	mitogen-activated protein kinase 14	Homo sapiens	19-28	
9401960-15	1997	Activation of both p38(MAPK) and GLUT1 by 0.3 microM anisomycin was cancelled by puromycin.	Anisomycin	53-63	solute carrier family 2 member 1	Homo sapiens	33-38	
9401960-15	1997	Activation of both p38(MAPK) and GLUT1 by 0.3 microM anisomycin was cancelled by puromycin.	Puromycin	81-90	mitogen-activated protein kinase 14	Homo sapiens	19-28	
9401960-15	1997	Activation of both p38(MAPK) and GLUT1 by 0.3 microM anisomycin was cancelled by puromycin.	Puromycin	81-90	solute carrier family 2 member 1	Homo sapiens	33-38	
9401960-17	1997	We conclude that the activation of GLUT1 in response to anisomycin includes two components: a delayed component involving translational inhibition and a fast, puromycin-inhibitable component that is secondary to activation of p38(MAPK).	Anisomycin	56-66	solute carrier family 2 member 1	Homo sapiens	35-40	
9401960-17	1997	We conclude that the activation of GLUT1 in response to anisomycin includes two components: a delayed component involving translational inhibition and a fast, puromycin-inhibitable component that is secondary to activation of p38(MAPK).	Anisomycin	56-66	mitogen-activated protein kinase 14	Homo sapiens	226-229	
9401960-17	1997	We conclude that the activation of GLUT1 in response to anisomycin includes two components: a delayed component involving translational inhibition and a fast, puromycin-inhibitable component that is secondary to activation of p38(MAPK).	Anisomycin	56-66	mitogen-activated protein kinase 14	Homo sapiens	230-234	
9401960-17	1997	We conclude that the activation of GLUT1 in response to anisomycin includes two components: a delayed component involving translational inhibition and a fast, puromycin-inhibitable component that is secondary to activation of p38(MAPK).	Puromycin	159-168	solute carrier family 2 member 1	Homo sapiens	35-40