PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9268688-1	1997	Maintenance of rats on a vitamin A-deficient diet resulting in undetectable levels of plasma retinol and significant reductions in relative testes weight compared to age-matched controls leads to the loss of liver membrane-bound low affinity glucocorticoid binding site (LAGS) activity without any effects on the levels of constitutively expressed CYP3A2 protein.	Vitamin A	25-34	cytochrome P450, family 3, subfamily a, polypeptide 2	Rattus norvegicus	348-354	
9268688-4	1997	Retinol acetate administration alone induces CYP3A2 protein to apparent maximal levels since dexamethasone 21-phosphate does not further increase the induction response.	retinol acetate	0-15	cytochrome P450, family 3, subfamily a, polypeptide 2	Rattus norvegicus	45-51	
9268688-5	1997	However, CYP3A2 remains inducible to dexamethasone 21-phosphate in vitamin A-deficient rats.	dexamethasone 21-phosphate	37-63	cytochrome P450, family 3, subfamily a, polypeptide 2	Rattus norvegicus	9-15	
9268688-5	1997	However, CYP3A2 remains inducible to dexamethasone 21-phosphate in vitamin A-deficient rats.	Vitamin A	67-76	cytochrome P450, family 3, subfamily a, polypeptide 2	Rattus norvegicus	9-15	
9268688-6	1997	These data suggest that vitamin A status affects the expression of LAGS and CYP3A2 but that glucocorticoids regulate the induction of CYP3A2 by a mechanism(s) independent of their interaction with the LAGS.	Vitamin A	24-33	cytochrome P450, family 3, subfamily a, polypeptide 2	Rattus norvegicus	76-82