PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9230198-4	1997	In the present study, using this in vitro model of hepatocarcinogenesis, we demonstrated that phenobarbital, a tumor promoter of rodent hepatocarcinogenesis, triggers remarkable apoptosis specifically in the c-myc-transfected CHST8 cells at 39 degrees C, which show abundant c-myc expression despite growth arrest.	Phenobarbital	94-107	carbohydrate sulfotransferase 8	Mus musculus	226-231	
9230198-5	1997	Dissociation of p53 proteins from degrading T antigens followed by a phenobarbital and c-myc-dependent, 15-fold induction of Bax protein, known to activate the apoptotic pathway downstream of p53, occurred in association with this phenomenon.	Phenobarbital	69-82	transformation related protein 53, pseudogene	Mus musculus	16-19	
9230198-5	1997	Dissociation of p53 proteins from degrading T antigens followed by a phenobarbital and c-myc-dependent, 15-fold induction of Bax protein, known to activate the apoptotic pathway downstream of p53, occurred in association with this phenomenon.	Phenobarbital	69-82	BCL2-associated X protein	Mus musculus	125-128	
9230198-5	1997	Dissociation of p53 proteins from degrading T antigens followed by a phenobarbital and c-myc-dependent, 15-fold induction of Bax protein, known to activate the apoptotic pathway downstream of p53, occurred in association with this phenomenon.	Phenobarbital	69-82	transformation related protein 53, pseudogene	Mus musculus	192-195	
9230198-6	1997	The effects of phenobarbital and c-myc in increasing Bax on shifting the temperature from 33 degrees C to 39 degrees C were additive, with both having similar degrees of influence on the protein level.	Phenobarbital	15-28	BCL2-associated X protein	Mus musculus	53-56	
9230198-7	1997	Interestingly, subsequent introduction of an activated c-H-ras oncogene into the c-myc-transfected CHST8 cells resulted not only in escape from the growth arrest at 39 degrees C but also in complete inhibition of the phenobarbital-inducible apoptosis along with de novo induction of the Bax antagonist, Bcl-2.	Phenobarbital	217-230	Harvey rat sarcoma virus oncogene	Mus musculus	55-62	
9230198-7	1997	Interestingly, subsequent introduction of an activated c-H-ras oncogene into the c-myc-transfected CHST8 cells resulted not only in escape from the growth arrest at 39 degrees C but also in complete inhibition of the phenobarbital-inducible apoptosis along with de novo induction of the Bax antagonist, Bcl-2.	Phenobarbital	217-230	carbohydrate sulfotransferase 8	Mus musculus	99-104	
9230198-7	1997	Interestingly, subsequent introduction of an activated c-H-ras oncogene into the c-myc-transfected CHST8 cells resulted not only in escape from the growth arrest at 39 degrees C but also in complete inhibition of the phenobarbital-inducible apoptosis along with de novo induction of the Bax antagonist, Bcl-2.	Phenobarbital	217-230	BCL2-associated X protein	Mus musculus	287-290	
9230198-7	1997	Interestingly, subsequent introduction of an activated c-H-ras oncogene into the c-myc-transfected CHST8 cells resulted not only in escape from the growth arrest at 39 degrees C but also in complete inhibition of the phenobarbital-inducible apoptosis along with de novo induction of the Bax antagonist, Bcl-2.	Phenobarbital	217-230	B cell leukemia/lymphoma 2	Mus musculus	303-308	
9230198-8	1997	These findings strongly suggest that the phenobarbital-inducible apoptosis is mediated by Bax.	Phenobarbital	41-54	BCL2-associated X protein	Mus musculus	90-93