PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
668812-1	1978	The bisquaternary mono-oxime HI-6, and to a lesser extent HS-6, caused functional recovery of neuromuscular transmission in vivo and in vitro when given 60 min after soman, i.e. when the soman-cholinesterase (AChE) complex is said to be fully "aged".	mono-oxime hi-6	18-33	butyrylcholinesterase	Rattus norvegicus	193-207	
668812-1	1978	The bisquaternary mono-oxime HI-6, and to a lesser extent HS-6, caused functional recovery of neuromuscular transmission in vivo and in vitro when given 60 min after soman, i.e. when the soman-cholinesterase (AChE) complex is said to be fully "aged".	mono-oxime hi-6	18-33	acetylcholinesterase	Rattus norvegicus	209-213	
668812-6	1978	In experiments with isolated rat phrenic nerve-diaphragm preparations, HI-6 given 60 min after soman produced functional recovery which could be abolished by a second dose of soman, suggesting that HI-6 had reactivated the AChE and that this enzyme was then reinhibited by the second dose of soman.	asoxime chloride	71-75	acetylcholinesterase	Rattus norvegicus	223-227	
668812-7	1978	HI-6 reactivates purified bovine erythrocyte AChE when added immediately after inhibition by soman, but does not reactivate tabun-inhibited AChE.	asoxime chloride	0-4	acetylcholinesterase	Bos taurus	45-49