PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
457783-0	1979	Tunicamycin-mediated depletion of insulin receptors in 3T3-L1 adipocytes.	Tunicamycin	0-11	insulin	Homo sapiens	34-41	
457783-1	1979	Tunicamycin, an antibiotic that inhibits protein glycosylation, elicited a rapid depletion of insulin binding activity at the surface of 3T3-L1 adipocytes.	Tunicamycin	0-11	insulin	Homo sapiens	94-101	
457783-2	1979	Disappearance of insulin receptors occurred more rapidly in the presence of tunicamycin than when protein synthesis was inhibited by cycloheximide and was accompanied by a diminution in sensitivity of the adipocytes to the acute effects of insulin and anti-insulin receptor antibody on hexose uptake and metabolism.	Tunicamycin	76-87	insulin	Homo sapiens	17-24	
457783-2	1979	Disappearance of insulin receptors occurred more rapidly in the presence of tunicamycin than when protein synthesis was inhibited by cycloheximide and was accompanied by a diminution in sensitivity of the adipocytes to the acute effects of insulin and anti-insulin receptor antibody on hexose uptake and metabolism.	Tunicamycin	76-87	insulin	Homo sapiens	240-247	
457783-2	1979	Disappearance of insulin receptors occurred more rapidly in the presence of tunicamycin than when protein synthesis was inhibited by cycloheximide and was accompanied by a diminution in sensitivity of the adipocytes to the acute effects of insulin and anti-insulin receptor antibody on hexose uptake and metabolism.	Tunicamycin	76-87	insulin	Homo sapiens	240-247