PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34419609-14	2021	Moreover, HCZP treatment significantly downregulated respiratory system resistance, and the expressions of IL-4, IL-5, IL-13 and IgE, as well as significantly improved inflammatory cell infiltration in lung tissues.	hczp	10-14	interleukin 4	Homo sapiens	107-111	
34419609-14	2021	Moreover, HCZP treatment significantly downregulated respiratory system resistance, and the expressions of IL-4, IL-5, IL-13 and IgE, as well as significantly improved inflammatory cell infiltration in lung tissues.	hczp	10-14	interleukin 5	Homo sapiens	113-117	
34419609-14	2021	Moreover, HCZP treatment significantly downregulated respiratory system resistance, and the expressions of IL-4, IL-5, IL-13 and IgE, as well as significantly improved inflammatory cell infiltration in lung tissues.	hczp	10-14	interleukin 13	Homo sapiens	119-124	
34419609-14	2021	Moreover, HCZP treatment significantly downregulated respiratory system resistance, and the expressions of IL-4, IL-5, IL-13 and IgE, as well as significantly improved inflammatory cell infiltration in lung tissues.	hczp	10-14	immunoglobulin heavy constant epsilon	Homo sapiens	129-132	
34419609-15	2021	Moreover, the protein expression of p-PI3K, p-JNK and p-p38 in lung tissues decreased after HCZP treatment.	hczp	92-96	mitogen-activated protein kinase 8	Homo sapiens	46-49	
34419609-15	2021	Moreover, the protein expression of p-PI3K, p-JNK and p-p38 in lung tissues decreased after HCZP treatment.	hczp	92-96	mitogen-activated protein kinase 14	Homo sapiens	56-59	
34419609-16	2021	CONCLUSION: HCZP significantly inhibited the OVA-induced inflammatory response via the PI3K-Akt and Fc epsilon RI signaling pathways.	hczp	12-16	AKT serine/threonine kinase 1	Homo sapiens	92-95	
34419609-16	2021	CONCLUSION: HCZP significantly inhibited the OVA-induced inflammatory response via the PI3K-Akt and Fc epsilon RI signaling pathways.	hczp	12-16	Fc epsilon receptor Ia	Homo sapiens	100-113