PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33479510-7	2022	Remarkably, a number of these behavioral deficits could be rescued by the administration of mu-opioid and D2 dopamine receptor (D2R) antagonists: naltrexone and haloperidol, respectively, suggesting that loss of neuropsychiatric manifestations in mice lacking GPR139 are driven by opioidergic and dopaminergic hyper-functionality.	Haloperidol	161-172	dopamine receptor D2	Mus musculus	106-126	
33479510-7	2022	Remarkably, a number of these behavioral deficits could be rescued by the administration of mu-opioid and D2 dopamine receptor (D2R) antagonists: naltrexone and haloperidol, respectively, suggesting that loss of neuropsychiatric manifestations in mice lacking GPR139 are driven by opioidergic and dopaminergic hyper-functionality.	Haloperidol	161-172	dopamine receptor D2	Mus musculus	128-131