PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33387788-0	2021	Dexamethasone inhibits SARS-CoV-2 spike pseudotyped virus viropexis by binding to ACE2.	Dexamethasone	0-13	angiotensin converting enzyme 2	Homo sapiens	82-86	
33387788-4	2021	Molecular docking results revealed that DEX occupied with active binding site of ACE2 of SARS-CoV-2 spike protein.	Dexamethasone	40-43	angiotensin converting enzyme 2	Homo sapiens	81-85	
33387788-5	2021	Surface plasmon resonance (SPR) results showed that KD value between DEX and ACE2 was (9.03 +- 0.78) e-6 M. Cell membrane chromatography (CMC) results uncovered that DEX had a chromatographic retention.	Dexamethasone	69-72	angiotensin converting enzyme 2	Homo sapiens	77-81	
33387788-5	2021	Surface plasmon resonance (SPR) results showed that KD value between DEX and ACE2 was (9.03 +- 0.78) e-6 M. Cell membrane chromatography (CMC) results uncovered that DEX had a chromatographic retention.	Dexamethasone	166-169	angiotensin converting enzyme 2	Homo sapiens	77-81	
33387788-6	2021	DEX was found out to inhibiting the viropexis into ACE2h cells using SARS-CoV-2 spike pseudotyped virus.	Dexamethasone	0-3	angiotensin converting enzyme 2	Homo sapiens	51-55	
33387788-7	2021	Therefore, DEX inhibits the entrance of SARS-CoV-2 spike pseudotyped virus into cell by binding to ACE2.	Dexamethasone	11-14	angiotensin converting enzyme 2	Homo sapiens	99-103