PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30207165-2	2018	We previously showed the pathogenic effects of MPO-ANCA that activate MPO to trigger an oxidative burst mainly through HOCl production, contributing to endothelial injury and lung fibrosis.	Hypochlorous Acid	119-123	myeloperoxidase	Homo sapiens	47-50	
30207165-2	2018	We previously showed the pathogenic effects of MPO-ANCA that activate MPO to trigger an oxidative burst mainly through HOCl production, contributing to endothelial injury and lung fibrosis.	Hypochlorous Acid	119-123	myeloperoxidase	Homo sapiens	70-73	
30207165-7	2018	MPA patients" sera displayed higher HOCl production by MPO ( P < 0.001), higher AOPP ( P < 0.001) and thiol ( P < 0.01) levels, compared with healthy subjects.	Hypochlorous Acid	36-40	myeloperoxidase	Homo sapiens	55-58	
30207165-11	2018	Our findings suggest that HOCl production by activated MPO could contribute to the very early stage of glomerulonephritis, whereas thiol may exert a protective effect against the development of renal vasculitis and glomerulosclerosis.	Hypochlorous Acid	26-30	myeloperoxidase	Homo sapiens	55-58