PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28392448-4	2017	Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons.	Lactic Acid	75-82	solute carrier family 16 member 1	Homo sapiens	26-55	
28392448-4	2017	Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons.	Lactic Acid	75-82	solute carrier family 16 member 1	Homo sapiens	57-61	
28392448-4	2017	Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons.	Lactic Acid	75-82	solute carrier family 16 member 1	Homo sapiens	139-143	
28392448-4	2017	Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons.	Lactic Acid	192-199	solute carrier family 16 member 1	Homo sapiens	26-55	
28392448-4	2017	Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons.	Lactic Acid	192-199	solute carrier family 16 member 1	Homo sapiens	57-61