PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27242527-0	2016	Rifaximin Improves Clostridium difficile Toxin A-Induced Toxicity in Caco-2 Cells by the PXR-Dependent TLR4/MyD88/NF-kappaB Pathway.	Rifaximin	0-9	toll like receptor 4	Homo sapiens	103-107	
27242527-8	2016	The expression of TLR4 (-33, -50, and -75%), MyD88 (-29, -60, and -81%) and TAK1 (-37, -63, and -79%) were also reduced with rifaximin versus TcdA treatment.	Rifaximin	125-134	toll like receptor 4	Homo sapiens	18-22	
27242527-10	2016	CONCLUSION: Rifaximin improved TcdA-induced toxicity in Caco-2 cells by the PXR-dependent TLR4/MyD88/NF-kappaB pathway mechanism, and may be useful in the treatment of CDIs.	Rifaximin	12-21	toll like receptor 4	Homo sapiens	90-94