PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26659468-0	2016	OCT1 mediates hepatic uptake of sumatriptan and loss-of-function OCT1 polymorphisms affect sumatriptan pharmacokinetics.	Sumatriptan	32-43	solute carrier family 22 member 1	Homo sapiens	0-4	
26659468-0	2016	OCT1 mediates hepatic uptake of sumatriptan and loss-of-function OCT1 polymorphisms affect sumatriptan pharmacokinetics.	Sumatriptan	91-102	solute carrier family 22 member 1	Homo sapiens	0-4	
26659468-0	2016	OCT1 mediates hepatic uptake of sumatriptan and loss-of-function OCT1 polymorphisms affect sumatriptan pharmacokinetics.	Sumatriptan	91-102	solute carrier family 22 member 1	Homo sapiens	65-69	
26659468-3	2016	OCT1 transported sumatriptan with high capacity and sumatriptan uptake into human hepatocytes was strongly inhibited by the OCT1 inhibitor MPP(+) .	Sumatriptan	17-28	solute carrier family 22 member 1	Homo sapiens	0-4	
26659468-3	2016	OCT1 transported sumatriptan with high capacity and sumatriptan uptake into human hepatocytes was strongly inhibited by the OCT1 inhibitor MPP(+) .	Sumatriptan	52-63	solute carrier family 22 member 1	Homo sapiens	124-128	
26659468-7	2016	In conclusion, OCT1 is a high-capacity transporter of sumatriptan and polymorphisms causing OCT1 deficiency have similar effects on sumatriptan pharmacokinetics as those observed in subjects with liver impairment.	Sumatriptan	54-65	solute carrier family 22 member 1	Homo sapiens	15-19	
26659468-7	2016	In conclusion, OCT1 is a high-capacity transporter of sumatriptan and polymorphisms causing OCT1 deficiency have similar effects on sumatriptan pharmacokinetics as those observed in subjects with liver impairment.	Sumatriptan	132-143	solute carrier family 22 member 1	Homo sapiens	92-96