PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25695262-0	2015	Expression of the platelet-activating factor receptor enhances benzyl isothiocyanate-induced apoptosis in murine and human melanoma cells.	benzyl isothiocyanate	63-84	platelet-activating factor receptor	Mus musculus	18-53	
25695262-5	2015	Activation of PAF-R, using the PAF-R agonist, 1-hexadecyl-2-N-methylcarbamoyl-3-glycerophosphocholine, induced an increase in the proliferation of B16-PAF-R cells compared with the B16-MSCV cells.	1-hexadecyl-2-n-methylcarbamoyl-3-glycerophosphocholine	46-101	platelet activating factor receptor	Homo sapiens	14-19	
25695262-5	2015	Activation of PAF-R, using the PAF-R agonist, 1-hexadecyl-2-N-methylcarbamoyl-3-glycerophosphocholine, induced an increase in the proliferation of B16-PAF-R cells compared with the B16-MSCV cells.	1-hexadecyl-2-n-methylcarbamoyl-3-glycerophosphocholine	46-101	platelet activating factor receptor	Homo sapiens	31-36	
25695262-5	2015	Activation of PAF-R, using the PAF-R agonist, 1-hexadecyl-2-N-methylcarbamoyl-3-glycerophosphocholine, induced an increase in the proliferation of B16-PAF-R cells compared with the B16-MSCV cells.	1-hexadecyl-2-n-methylcarbamoyl-3-glycerophosphocholine	46-101	platelet activating factor receptor	Homo sapiens	147-156	
25695262-8	2015	The results revealed that treatment with BITC decreased the survival rate of the PAF-R-positive and negative murine and human melanoma cells.	benzyl isothiocyanate	41-45	platelet-activating factor receptor	Mus musculus	81-86	
25695262-10	2015	In order to determine the underlying mechanism, flow cytometric analysis was used, which demonstrated a significant increase in the generation of reactive oxygen species (ROS) in the B16-PAF-R cells compared with the B16-MSCV cells, which enhanced apoptosis by BITC, as measured by increased caspase-3/7 luminescence.	Reactive Oxygen Species	146-169	platelet activating factor receptor	Homo sapiens	183-192	
25695262-10	2015	In order to determine the underlying mechanism, flow cytometric analysis was used, which demonstrated a significant increase in the generation of reactive oxygen species (ROS) in the B16-PAF-R cells compared with the B16-MSCV cells, which enhanced apoptosis by BITC, as measured by increased caspase-3/7 luminescence.	Reactive Oxygen Species	171-174	platelet activating factor receptor	Homo sapiens	183-192	
25695262-11	2015	Notably, the BITC-mediated decreased cell survival rate, increased ROS and increased apoptosis in the B16-PAF-R cells were significantly attenuated by the antioxidant, vitamin C, indicating ROS involvement.	Ascorbic Acid	168-177	platelet activating factor receptor	Homo sapiens	102-111	
25695262-11	2015	Notably, the BITC-mediated decreased cell survival rate, increased ROS and increased apoptosis in the B16-PAF-R cells were significantly attenuated by the antioxidant, vitamin C, indicating ROS involvement.	Reactive Oxygen Species	190-193	platelet activating factor receptor	Homo sapiens	102-111