PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24046862-4	2013	Using the NS398 specific inhibitor of COX-2, we provide evidence that reactive oxygen species by-produced by the COX-2 enzymatic activity are negligible in front of the total senescence-associated oxidative stress.	Reactive Oxygen Species	70-93	mitochondrially encoded cytochrome c oxidase II	Homo sapiens	38-43	
24046862-4	2013	Using the NS398 specific inhibitor of COX-2, we provide evidence that reactive oxygen species by-produced by the COX-2 enzymatic activity are negligible in front of the total senescence-associated oxidative stress.	Reactive Oxygen Species	70-93	mitochondrially encoded cytochrome c oxidase II	Homo sapiens	113-118	
24046862-11	2013	Taken together, these results suggest that COX-2 contributes to the establishment and maintenance of senescence of normal human fibroblasts via an independent-ROS and a dependent-PGE2/EPs intracrine pathway.	Reactive Oxygen Species	159-162	mitochondrially encoded cytochrome c oxidase II	Homo sapiens	43-48