PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23675509-4	2013	Treating the IGF-1R deficient myoblasts with H2O2 resulted in significantly higher phosphorylation of Akt as compared to cells having normal expression of IGF-1R.	Hydrogen Peroxide	45-49	insulin-like growth factor I receptor	Mus musculus	13-19	
23675509-4	2013	Treating the IGF-1R deficient myoblasts with H2O2 resulted in significantly higher phosphorylation of Akt as compared to cells having normal expression of IGF-1R.	Hydrogen Peroxide	45-49	insulin-like growth factor I receptor	Mus musculus	155-161	
23675509-8	2013	However, in NIH-3T3 fibroblasts and MC3T3-E1 osteoblasts, the loss of IGF-1R by siRNA directed knockdown was associated with reduced levels of phosphorylated Akt on treatment with H2O2 or UV as compared to control cells and these cells showed more apoptosis.	Hydrogen Peroxide	180-184	insulin-like growth factor I receptor	Mus musculus	70-76