PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22128160-5	2012	Furthermore, pharmacological and mutagenesis assays revealed that protein kinase C (PKC) and high K(+) depolarization increase RET surface levels through phosphorylation of the Thr(675) residue in the Box1 motif.	Threonine	177-180	ret proto-oncogene	Homo sapiens	127-130	
22128160-6	2012	Finally, we found that the phosphorylation status of the Thr(675) residue influences RET mediated response to GDNF stimulation.	Threonine	57-60	ret proto-oncogene	Homo sapiens	85-88