PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2011401-2	1991	Treating JURKAT cells with the phorbol ester tetradecanoyl phorbol acetate (TPA) inhibited their proliferation and induced expression of the gene for the interleukin 2 receptor alpha chain (IL2R-alpha), consistent with previous reports.	Tetradecanoylphorbol Acetate	76-79	interleukin 2 receptor subunit alpha	Homo sapiens	190-200	
2011401-4	1991	In contrast, accumulation of mRNAs for the C-FOS, C-JUN, and EGR-1 genes increased markedly in TPA-treated cells and preceded the induction of IL2R-alpha mRNA.	Tetradecanoylphorbol Acetate	95-98	interleukin 2 receptor subunit alpha	Homo sapiens	143-153	
2011401-7	1991	Despite sustained expression of C-MYC, BCL2, or the combination of these protooncogenes, TPA continued to inhibit JURKAT cell growth and to induce IL2R expression.	Tetradecanoylphorbol Acetate	89-92	interleukin 2 receptor subunit alpha	Homo sapiens	147-151