PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2002334-2	1991	Chronic (3-72-h) agonist (nicotine or carbamylcholine) treatment of cells led to a complete (TE671) or nearly complete (PC12) loss of functional nAChR responses, which is referred to as "functional inactivation."	Carbachol	38-53	cholinergic receptor nicotinic alpha 4 subunit	Homo sapiens	145-150	
2002334-8	1991	Recovery of TE671 cell nAChR function following treatment with carbamylcholine, nicotine, or d-TC occurred with half-times of 1-3 days whether cells were maintained in situ or harvested and replated after removal of ligand.	Carbachol	63-78	cholinergic receptor nicotinic alpha 4 subunit	Homo sapiens	23-28