PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18829552-0	2008	ATM and the Mre11-Rad50-Nbs1 complex respond to nucleoside analogue-induced stalled replication forks and contribute to drug resistance.	Nucleosides	48-58	ATM serine/threonine kinase	Homo sapiens	0-3	
18829552-0	2008	ATM and the Mre11-Rad50-Nbs1 complex respond to nucleoside analogue-induced stalled replication forks and contribute to drug resistance.	Nucleosides	48-58	MRE11 homolog, double strand break repair nuclease	Homo sapiens	12-17	
18829552-0	2008	ATM and the Mre11-Rad50-Nbs1 complex respond to nucleoside analogue-induced stalled replication forks and contribute to drug resistance.	Nucleosides	48-58	RAD50 double strand break repair protein	Homo sapiens	18-23	
18829552-0	2008	ATM and the Mre11-Rad50-Nbs1 complex respond to nucleoside analogue-induced stalled replication forks and contribute to drug resistance.	Nucleosides	48-58	nibrin	Homo sapiens	24-28	
18829552-1	2008	The Mre11-Rad50-Nbs1 complex and autophosphorylated Ser(1981)-ATM are involved in recognizing and repairing DNA damage, such as double-strand breaks (DSB).	Serine	52-55	ATM serine/threonine kinase	Homo sapiens	62-65	
18829552-5	2008	Cellular responses to both DSBs and stalled replication forks are marked by H2AX phosphorylation on Ser(139) (gamma-H2AX), which forms nuclear foci at sites of DNA damage.	Serine	100-103	H2A.X variant histone	Homo sapiens	76-80	
18829552-5	2008	Cellular responses to both DSBs and stalled replication forks are marked by H2AX phosphorylation on Ser(139) (gamma-H2AX), which forms nuclear foci at sites of DNA damage.	Serine	100-103	H2A.X variant histone	Homo sapiens	116-120	
18829552-7	2008	Using gamma-H2AX as a marker for changes in chromatin structure, we show that Mre11, Rad50, Nbs1, and phosphorylated ATM respond to nucleoside analogue-induced stalled replication forks by forming nuclear foci that colocalize with gamma-H2AX within 2 hours.	Nucleosides	132-142	H2A.X variant histone	Homo sapiens	12-16	
18829552-7	2008	Using gamma-H2AX as a marker for changes in chromatin structure, we show that Mre11, Rad50, Nbs1, and phosphorylated ATM respond to nucleoside analogue-induced stalled replication forks by forming nuclear foci that colocalize with gamma-H2AX within 2 hours.	Nucleosides	132-142	MRE11 homolog, double strand break repair nuclease	Homo sapiens	78-83	
18829552-7	2008	Using gamma-H2AX as a marker for changes in chromatin structure, we show that Mre11, Rad50, Nbs1, and phosphorylated ATM respond to nucleoside analogue-induced stalled replication forks by forming nuclear foci that colocalize with gamma-H2AX within 2 hours.	Nucleosides	132-142	RAD50 double strand break repair protein	Homo sapiens	85-90	
18829552-7	2008	Using gamma-H2AX as a marker for changes in chromatin structure, we show that Mre11, Rad50, Nbs1, and phosphorylated ATM respond to nucleoside analogue-induced stalled replication forks by forming nuclear foci that colocalize with gamma-H2AX within 2 hours.	Nucleosides	132-142	nibrin	Homo sapiens	92-96	
18829552-7	2008	Using gamma-H2AX as a marker for changes in chromatin structure, we show that Mre11, Rad50, Nbs1, and phosphorylated ATM respond to nucleoside analogue-induced stalled replication forks by forming nuclear foci that colocalize with gamma-H2AX within 2 hours.	Nucleosides	132-142	ATM serine/threonine kinase	Homo sapiens	117-120	
18829552-7	2008	Using gamma-H2AX as a marker for changes in chromatin structure, we show that Mre11, Rad50, Nbs1, and phosphorylated ATM respond to nucleoside analogue-induced stalled replication forks by forming nuclear foci that colocalize with gamma-H2AX within 2 hours.	Nucleosides	132-142	H2A.X variant histone	Homo sapiens	237-241	
18829552-9	2008	Deficiencies in ATM, Mre11, or Rad50 led to a 2- to 5-fold increase in clonogenic sensitization to gemcitabine, whereas Nbs1 and H2AX deficiency did not affect reproductive growth.	gemcitabine	99-110	ATM serine/threonine kinase	Homo sapiens	16-19	
18829552-9	2008	Deficiencies in ATM, Mre11, or Rad50 led to a 2- to 5-fold increase in clonogenic sensitization to gemcitabine, whereas Nbs1 and H2AX deficiency did not affect reproductive growth.	gemcitabine	99-110	MRE11 homolog, double strand break repair nuclease	Homo sapiens	21-26	
18829552-9	2008	Deficiencies in ATM, Mre11, or Rad50 led to a 2- to 5-fold increase in clonogenic sensitization to gemcitabine, whereas Nbs1 and H2AX deficiency did not affect reproductive growth.	gemcitabine	99-110	RAD50 double strand break repair protein	Homo sapiens	31-36