PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16790503-0	2006	Biotin-responsive basal ganglia disease-linked mutations inhibit thiamine transport via hTHTR2: biotin is not a substrate for hTHTR2.	Biotin	0-6	solute carrier family 19 member 3	Homo sapiens	88-94	
16790503-0	2006	Biotin-responsive basal ganglia disease-linked mutations inhibit thiamine transport via hTHTR2: biotin is not a substrate for hTHTR2.	Biotin	96-102	solute carrier family 19 member 3	Homo sapiens	88-94	
16790503-4	2006	Recently, two hTHTR2 mutants (G23V, T422A) have been discovered in clinical kindreds manifesting biotin-responsive basal ganglia disease (BBGD): the symptoms of which are alleviated by biotin administration.	Biotin	97-103	solute carrier family 19 member 3	Homo sapiens	14-20	
16790503-4	2006	Recently, two hTHTR2 mutants (G23V, T422A) have been discovered in clinical kindreds manifesting biotin-responsive basal ganglia disease (BBGD): the symptoms of which are alleviated by biotin administration.	Biotin	185-191	solute carrier family 19 member 3	Homo sapiens	14-20	
16790503-8	2006	Furthermore, biotin accumulation was not detectable in cells overexpressing either the full length hTHTR2 or the clinically relevant hTHTR2 mutants, yet was demonstrable in the same assay using cells overexpressing the human sodium-dependent multivitamin transporter, a known biotin transporter.	Biotin	13-19	solute carrier family 19 member 3	Homo sapiens	99-105	
16790503-8	2006	Furthermore, biotin accumulation was not detectable in cells overexpressing either the full length hTHTR2 or the clinically relevant hTHTR2 mutants, yet was demonstrable in the same assay using cells overexpressing the human sodium-dependent multivitamin transporter, a known biotin transporter.	Biotin	13-19	solute carrier family 19 member 3	Homo sapiens	133-139