PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10201532-3	1999	Interleukin-4 deficient BALB/C mice sensitized to 2,4-dinitrochlorobenzene showed after challenge a significant reduction in magnitude and duration of the contact hypersensitivity response in comparison with wild-type mice.	Dinitrochlorobenzene	50-74	interleukin 4	Mus musculus	0-13	
10201532-8	1999	This indicates that the contact hypersensitivity response to 2,4-dinitrochlorobenzene and Oxazolone may partly evolve on different pathways being dependent and independent of interleukin-4.	Dinitrochlorobenzene	61-85	interleukin 4	Mus musculus	175-188	
10201532-9	1999	Our results clearly show that the complete loss of endogenous interleukin-4 expression in BALB/C mice is associated with an impaired manifestation of contact hypersensitivity response to 2,4-dinitrochlorobenzene, implying an important proinflammatory function of this cytokine.	Dinitrochlorobenzene	187-211	interleukin 4	Mus musculus	62-75