PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
9918571-3	1999	Drugs binding to FKBP12 and inhibiting calcineurin, such as FK506 and SDZ ASM 981, dose dependently reduced the infarct volumes, determined 48 h after MCAO by both magnetic resonance imaging and triphenyltetrazolium chloride staining but only in the transient MCAO model.	triphenyltetrazolium	195-224	FKBP prolyl isomerase 1A	Rattus norvegicus	17-23
10598938-2	1999	2,3,5-Triphenyltetrazolium chloride staining showed that infarct volume of the Ad-GDNF-injected group at 24 hours after the transient MCAO was significantly smaller than that of vehicle- or Ad-LacZ-treated group.	triphenyltetrazolium	0-35	glial cell derived neurotrophic factor	Rattus norvegicus	82-86
9918571-3	1999	Drugs binding to FKBP12 and inhibiting calcineurin, such as FK506 and SDZ ASM 981, dose dependently reduced the infarct volumes, determined 48 h after MCAO by both magnetic resonance imaging and triphenyltetrazolium chloride staining but only in the transient MCAO model.	triphenyltetrazolium	195-224	H19, imprinted maternally expressed transcript (non-protein coding)	Rattus norvegicus	74-77
9742088-9	1998	Disruption of YHM2 causes a significant growth defect in the presence of nonfermentable carbon sources such as glycerol and ethanol, and the cells have defects in respiration as determined by 2,3,5,-triphenyltetrazolium chloride staining.	triphenyltetrazolium	192-228	Yhm2p	Saccharomyces cerevisiae S288C	14-18
9822733-4	1998	Brain injury volumes after 2 hr of suture insertion, as evaluated by 2,3,5-triphenyltetrazolium chloride staining at 24 hr after ischemia, revealed significantly smaller injury size in GluRepsilon1 subunit knock-out mice compared with their wild-type littermates.	triphenyltetrazolium	69-104	glutamate receptor, ionotropic, NMDA2A (epsilon 1)	Mus musculus	185-197
7978253-1	1994	A procedure was developed for activity staining of peroxidase and beta-1,3-glucanase isozymes (a double staining procedure) in the same polyacrylamide gels using 3-amino-9-ethylcarbazol for peroxidase staining and laminarine and 2,3,5-triphenyltetrazolium chloride for glucanase staining.	triphenyltetrazolium	229-264	peroxidase 1	Zea mays	51-61
8281669-6	1994	Compared with control and 40 degrees C rats, there was progressive infarct size reduction, assessed by triphenyltetrazolium chloride staining, in rats that were heat-shocked to 41 degrees C and 42 degrees C. Furthermore, there was a direct correlation between the amount of HSP72 induced and the reduction in infarct size (r = .97, P = .037).	triphenyltetrazolium	103-132	heat shock protein family A (Hsp70) member 1A	Rattus norvegicus	274-279
34514847-6	2021	The therapeutic efficacy of autotaxin/LPA receptor blockade was evaluated using triphenyl tetrazolium chloride staining, Evans blue permeability, infrared imaging, mass spectrometry, and XF24 analyzer to evaluate blood-brain barrier integrity, autotaxin activity, and mitochondrial bioenergetics.	triphenyltetrazolium	80-110	ectonucleotide pyrophosphatase/phosphodiesterase 2	Mus musculus	28-37
33854356-4	2021	Significantly reduced myocardial infarct size, decreased apoptosis ratio and upregulated SIRT1 protein expression in heart were found in swam mice by 2,3,5-triphenyltetrazolium chloride (TTC) staining of heart sections, TUNEL staining of frozen sections and Western blotting.	triphenyltetrazolium	150-185	sirtuin 1	Mus musculus	89-94
2980745-8	1987	In the case of ischemia lasting for more than 6 hours, the anti-actin antibody reaction had disappeared, corresponding to the disappearance of the TTC reaction.	triphenyltetrazolium	147-150	actin, beta	Gallus gallus	64-69
6366242-10	1984	In dogs with 3 to 24 h occlusion immunostaining was significantly reduced for both CK-B and CK-M in regions confirmed to be necrotic by triphenyl tetrazolium chloride (TTC) and H & E staining.	triphenyltetrazolium	136-166	Creatine kinase B chain	Canis lupus familiaris	83-87
6366242-10	1984	In dogs with 3 to 24 h occlusion immunostaining was significantly reduced for both CK-B and CK-M in regions confirmed to be necrotic by triphenyl tetrazolium chloride (TTC) and H & E staining.	triphenyltetrazolium	136-166	creatine kinase, M-type	Canis lupus familiaris	92-96
6366242-10	1984	In dogs with 3 to 24 h occlusion immunostaining was significantly reduced for both CK-B and CK-M in regions confirmed to be necrotic by triphenyl tetrazolium chloride (TTC) and H & E staining.	triphenyltetrazolium	168-171	Creatine kinase B chain	Canis lupus familiaris	83-87
6366242-10	1984	In dogs with 3 to 24 h occlusion immunostaining was significantly reduced for both CK-B and CK-M in regions confirmed to be necrotic by triphenyl tetrazolium chloride (TTC) and H & E staining.	triphenyltetrazolium	168-171	creatine kinase, M-type	Canis lupus familiaris	92-96
5855390-0	1965	[Triphenyltetrazolium chloride--an aid in the examination of urinary tract infections?	triphenyltetrazolium	1-30	activation induced cytidine deaminase	Homo sapiens	35-38
2539540-0	1989	Evaluation of the effect of evan"s blue and triphenyltetrazolium chloride dyes on myeloperoxidase activity in canine cardiac tissue.	triphenyltetrazolium	44-73	myeloperoxidase	Canis lupus familiaris	82-97
3352297-7	1988	Infarct size measured by triphenyltetrazolium chloride staining showed significant difference (p less than 0.001) between animals treated with superoxide dismutase and catalase (0.9% +/- 0.1%) and control animals (61% +/- 70%).	triphenyltetrazolium	25-54	catalase	Ovis aries	168-176
34013967-10	2021	RESULTS: TTC staining showed a smaller brain injury area in the Olig2-RNAi group than in the RNAi-negative control group.	triphenyltetrazolium	9-12	oligodendrocyte transcription factor 2	Rattus norvegicus	64-69
33854356-4	2021	Significantly reduced myocardial infarct size, decreased apoptosis ratio and upregulated SIRT1 protein expression in heart were found in swam mice by 2,3,5-triphenyltetrazolium chloride (TTC) staining of heart sections, TUNEL staining of frozen sections and Western blotting.	triphenyltetrazolium	187-190	sirtuin 1	Mus musculus	89-94
33854356-7	2021	Importantly, the data of TTC staining, TUNEL staining, Western blotting, echocardiography and histopathological staining revealed that mice with inducible cardiac SIRT1-knockout blocked the protective effects of exercise preconditioning on myocardial infarct size, myocardial apoptosis, adverse ventricular remodeling, cardiac fibrosis and cardiac dysfunction after MI/R injury, simultaneously exercise-induced expression of myocardial antioxidant stress factors was hindered which was detected by immunohistochemical analysis.	triphenyltetrazolium	25-28	sirtuin 1	Mus musculus	163-168
33386278-7	2021	However, cell growth and ethanol and succinate production of the ura3-mutated strain BY4741, which also exhibited no TTC staining activity, was altered compared to those of BY4741U, indicating that the fermentation profile varies among strains that show no TTC staining activity.	triphenyltetrazolium	117-120	orotidine-5'-phosphate decarboxylase	Saccharomyces cerevisiae S288C	65-69
32138732-9	2020	TTC staining showed that the infracted area of whole brain was significantly attenuated in treated with sh-MALAT1 group in vivo.	triphenyltetrazolium	0-3	metastasis associated lung adenocarcinoma transcript 1 (non-coding RNA)	Mus musculus	107-113
33576461-7	2021	The triphenyltetrazolium chloride, Nissl staining and immunohistochemistry results further demonstrated that miR-150 knockdown improved the activity of hippocampal neurons in VD model rats compared with the antagomiR NC group.	triphenyltetrazolium	4-33	microRNA 150	Rattus norvegicus	109-116
33436714-10	2021	Plasma CT-proAVP (r = 0.771, p = 0.025) and IGF-1 (r = -0.775, p = 0.024) demonstrated a strong correlation with TTC staining area.	triphenyltetrazolium	113-116	insulin-like growth factor 1	Rattus norvegicus	44-49
31799679-11	2019	TTC staining results revealed that lncRNA GAS5 knockdown could remarkably reduce the CI area of rats in CI group (p<0.05).	triphenyltetrazolium	0-3	growth arrest specific 5	Rattus norvegicus	42-46
30809120-0	2019	SAT: Free Software for the Semi-Automated Analysis of Rodent Brain Sections With 2,3,5-Triphenyltetrazolium Chloride Staining.	triphenyltetrazolium	81-116	spermidine/spermine N1-acetyl transferase 1	Mus musculus	0-3
29978354-8	2019	Brain infarct volume in TTC (2,3,5-triphenyltetrazolium chloride)-stained brain sections was significantly smaller 24 h after MCAO in PAI-1-deficient mice (- 31%), but not in PAI-2-deficient mice (- 6%).	triphenyltetrazolium	24-27	serine (or cysteine) peptidase inhibitor, clade E, member 1	Mus musculus	134-139
29978354-8	2019	Brain infarct volume in TTC (2,3,5-triphenyltetrazolium chloride)-stained brain sections was significantly smaller 24 h after MCAO in PAI-1-deficient mice (- 31%), but not in PAI-2-deficient mice (- 6%).	triphenyltetrazolium	29-64	serine (or cysteine) peptidase inhibitor, clade E, member 1	Mus musculus	134-139
31832520-12	2019	TTC staining showed treatment with rno-miR-124-3p agomir reduced brain infarction.	triphenyltetrazolium	0-3	microRNA 124-3	Rattus norvegicus	35-49
31552299-4	2019	Measurement of 2,3,5-triphenyltetrazolium chloride (TTC) or the MAP2-negative area in a mouse model of middle cerebral artery occlusion (MCAO) revealed that Slc38a1 deficiency decreased infarct size.	triphenyltetrazolium	15-50	solute carrier family 38, member 1	Mus musculus	157-164
31552299-4	2019	Measurement of 2,3,5-triphenyltetrazolium chloride (TTC) or the MAP2-negative area in a mouse model of middle cerebral artery occlusion (MCAO) revealed that Slc38a1 deficiency decreased infarct size.	triphenyltetrazolium	52-55	solute carrier family 38, member 1	Mus musculus	157-164
29788103-7	2018	Post-ischaemic treatment with IL-1beta antibody reduced infarct size, cerebral oedema and improved neurological performance as assessed by 2,3,5-triphenyltetrazolium chloride staining, Bederson and RotaRod tests.	triphenyltetrazolium	139-174	interleukin 1 beta	Mus musculus	30-38
27040712-6	2016	In PAFR deficiency, we observed a significant improvement in the neurological deficits, which were associated with a reduction of brain infarcted area as evaluated by triphenyltetrazolium chloride (TTC).	triphenyltetrazolium	167-196	platelet-activating factor receptor	Mus musculus	3-7
29906552-4	2018	Our data indicated that IL-33 deficiency exacerbated the neurological dysfunction caused by cerebral ischemia-reperfusion injury in mice and led to the formation of larger cerebral infarct volume as shown by 2,3,5-triphenyltetrazolium chloride staining and magnetic resonance imaging.	triphenyltetrazolium	208-243	interleukin 33	Mus musculus	24-29
28637776-8	2017	Although there was no significant difference in the area at risk, the area of vital myocardium was markedly larger in mice treated with RNase-1 compared with controls, as detected by Evans blue and 2,3,5-triphenyltetrazolium chloride staining.	triphenyltetrazolium	198-233	ribonuclease, RNase A family, 1 (pancreatic)	Mus musculus	136-143
28294064-7	2017	RESULTS: In CCR5 deficiency, we observed a significant improvement in the neurological deficits associated with decreased brain infarcted area as evaluated by triphenyltetrazolium chloride (TTC).	triphenyltetrazolium	159-188	chemokine (C-C motif) receptor 5	Mus musculus	12-16
28294064-7	2017	RESULTS: In CCR5 deficiency, we observed a significant improvement in the neurological deficits associated with decreased brain infarcted area as evaluated by triphenyltetrazolium chloride (TTC).	triphenyltetrazolium	190-193	chemokine (C-C motif) receptor 5	Mus musculus	12-16
29547524-6	2018	The myocardial infarct area/area at risk (IA/AAR), evaluated using Evans blue (EB) and 2,3,5-triphenyltetrazolium chloride (TTC) staining, was significantly smaller in SeP KO mice than in WT mice.	triphenyltetrazolium	87-122	selenoprotein P	Mus musculus	168-171
29547524-6	2018	The myocardial infarct area/area at risk (IA/AAR), evaluated using Evans blue (EB) and 2,3,5-triphenyltetrazolium chloride (TTC) staining, was significantly smaller in SeP KO mice than in WT mice.	triphenyltetrazolium	124-127	selenoprotein P	Mus musculus	168-171
27040712-6	2016	In PAFR deficiency, we observed a significant improvement in the neurological deficits, which were associated with a reduction of brain infarcted area as evaluated by triphenyltetrazolium chloride (TTC).	triphenyltetrazolium	198-201	platelet-activating factor receptor	Mus musculus	3-7
26408350-1	2016	The archetypal triphenyl tetrazolium chloride (tetrazolium red) has been used in spectrophotometric microplate assays for 2-hydroxy ketones and, by extension, for the activity of enzymes including aminotransferase, transketolase, and pyruvate decarboxylase.	triphenyltetrazolium	15-45	transketolase	Homo sapiens	215-228
26851772-8	2016	The histochemical localization of thrombin activity following tMCAo was in good correlation with the infarct areas per triphenyltetrazolium chloride staining and to thrombin activity measured biochemically in tissue punches (85 +- 35 and 20 +- 3 mU/ml, in the cortical and striatum areas respectively, compared to 7 +- 2 and 13 +- 2 mU/ml, in the corresponding contralateral areas; mean +- SEM; p<0.05).	triphenyltetrazolium	119-148	coagulation factor II	Mus musculus	34-42
24213611-7	2014	Cardiac troponin I (cTnI), a specific marker of myocardial injury, was significantly elevated at 6 h (41 +- 6 ng/ml, n = 4, P < 0.05 vs. baseline) and returned to baseline after 72 h. Triphenyltetrazolium chloride staining revealed transmural anterolateral infarcts targeting 25 +- 3% of the left ventricle at day 1 with a decrease to 20 +- 3% at day 40 (n = 6 for each group, P < 0.01 vs. day 1).	triphenyltetrazolium	187-216	troponin I3, cardiac type	Rattus norvegicus	0-18
25633836-6	2015	Histochemical analysis using Evan"s Blue, triphenyl-tetrazolium chloride and TUNEL staining, demonstrated that myocardial injury and cardiomyocyte apoptosis induced I/R injury were significantly attenuated in PHD3 knockout mice.	triphenyltetrazolium	42-72	egl-9 family hypoxia-inducible factor 3	Mus musculus	209-213
24477823-8	2014	Compared with the EGFP-MSCs group, the EGFP-hRAMP1-MSCs group had a significantly smaller infarcted area and improved cardiac function by 28 days after cell transplantation, as detected by triphenyltetrazolium chloride staining and echocardiography.	triphenyltetrazolium	189-218	receptor activity modifying protein 1	Homo sapiens	44-50
24366244-9	2014	The infarction area was significantly decreased following treatment with this concentration of PP1-12, as revealed by 2,3,5-triphenyltetrazolium chloride (TTC) staining.	triphenyltetrazolium	118-153	neuropeptide Y receptor Y4	Rattus norvegicus	95-101
24366244-9	2014	The infarction area was significantly decreased following treatment with this concentration of PP1-12, as revealed by 2,3,5-triphenyltetrazolium chloride (TTC) staining.	triphenyltetrazolium	155-158	neuropeptide Y receptor Y4	Rattus norvegicus	95-101
24213611-7	2014	Cardiac troponin I (cTnI), a specific marker of myocardial injury, was significantly elevated at 6 h (41 +- 6 ng/ml, n = 4, P < 0.05 vs. baseline) and returned to baseline after 72 h. Triphenyltetrazolium chloride staining revealed transmural anterolateral infarcts targeting 25 +- 3% of the left ventricle at day 1 with a decrease to 20 +- 3% at day 40 (n = 6 for each group, P < 0.01 vs. day 1).	triphenyltetrazolium	187-216	troponin I3, cardiac type	Rattus norvegicus	20-24
23410819-9	2013	Infarction size by 2,3,5-triphenyltetrazolium chloride (TTC) staining was smaller in the Bcl-2, but not in the other groups as compared to wild-type mice.	triphenyltetrazolium	19-54	B cell leukemia/lymphoma 2	Mus musculus	89-94
24672780-7	2014	Triphenyltetrazolium chloride staining and behavioral analysis revealed greater functional recovery in animals with MSCs-BDNF compared with the other groups.	triphenyltetrazolium	0-29	brain derived neurotrophic factor	Homo sapiens	121-125
23410819-9	2013	Infarction size by 2,3,5-triphenyltetrazolium chloride (TTC) staining was smaller in the Bcl-2, but not in the other groups as compared to wild-type mice.	triphenyltetrazolium	56-59	B cell leukemia/lymphoma 2	Mus musculus	89-94
23585565-6	2013	2,3,5-Triphenyltetrazolium chloride, TUNEL, and Nissl staining indicated that an up-regulation of CYGB resulted in reduced acute brain injury.	triphenyltetrazolium	0-35	cytoglobin	Rattus norvegicus	98-102
20412616-9	2010	Cerebral infarction analyzed using 2,3,5-triphenyl-tetrazolium chloride staining confirmed that the VEGF gels significantly and potently reduced the lesion volume.	triphenyltetrazolium	35-71	vascular endothelial growth factor A	Rattus norvegicus	100-104
22707562-6	2012	IL-11 treatment reduced I/R injury, analyzed by triphenyl tetrazolium chloride staining [PBS, 46.7 +- 14.4%; IL-11 (20 mug/kg), 28.6 +- 7.5% in the ratio of infarct to risk area].	triphenyltetrazolium	48-78	interleukin 11	Mus musculus	0-5
23123139-13	2013	Based on triphenyltetrazolium chloride staining and autoradiograph image data, the hotspot uptake may be associated primarily with the ischemic penumbra, in which high apoptotic activity was observed by cleaved caspase-3 immunocytochemical staining.	triphenyltetrazolium	9-38	caspase 3	Rattus norvegicus	211-220
22462051-1	2011	Magnetic resonance tomography, staining with triphenyltetrazolium chloride, and tests for evaluation of functional disturbances "cylinder" and "limb stimulation" showed that daily intraperitoneal injection of dipeptide mimetic of nerve growth factor GK-2 (1 mg/kg) for 6 days to rats with experimental focal ischemia provoked by unilateral intravascular occlusion of a branch of the middle cerebral artery significantly improved neurological deficit and decreased the infarction area.	triphenyltetrazolium	45-74	nerve growth factor	Rattus norvegicus	230-249
22462051-1	2011	Magnetic resonance tomography, staining with triphenyltetrazolium chloride, and tests for evaluation of functional disturbances "cylinder" and "limb stimulation" showed that daily intraperitoneal injection of dipeptide mimetic of nerve growth factor GK-2 (1 mg/kg) for 6 days to rats with experimental focal ischemia provoked by unilateral intravascular occlusion of a branch of the middle cerebral artery significantly improved neurological deficit and decreased the infarction area.	triphenyltetrazolium	45-74	glycerol kinase 2	Rattus norvegicus	250-254
19399876-6	2009	Triphenyltetrazolium chloride (TTC) staining indicated that pretreatment with MANF significantly reduced the volume of infarction at 2 days after MCAo.	triphenyltetrazolium	0-29	mesencephalic astrocyte-derived neurotrophic factor	Rattus norvegicus	78-82
19470883-7	2009	Moreover, tumor necrosis factor-alpha inhibition rescued the hemolysis-induced brain injury, evaluated with the method of 2,3,5-triphenyltetrazolium chloride and by the histological analysis of pyknotic nuclei.	triphenyltetrazolium	122-157	tumor necrosis factor	Homo sapiens	10-37
19399876-6	2009	Triphenyltetrazolium chloride (TTC) staining indicated that pretreatment with MANF significantly reduced the volume of infarction at 2 days after MCAo.	triphenyltetrazolium	31-34	mesencephalic astrocyte-derived neurotrophic factor	Rattus norvegicus	78-82
17882163-6	2008	The intrastriatal injection of recombinant glutathione S-transferase-tagged human DJ-1 (GST-DJ-1) markedly reduced infarct size in 2,3,5-triphenyltetrazolium chloride staining at 3 days after MCAO.	triphenyltetrazolium	131-166	Parkinsonism associated deglycase	Homo sapiens	82-86
19038254-2	2009	METHODS: Employing LDF, relative regional cerebral blood flow (rCBF) was continuously monitored during the first 2 h following ES and correlated with 24 h 2,3,5-triphenyltetrazolium chloride (TTC)-staining of corrected infarct volume.	triphenyltetrazolium	155-190	CCAAT/enhancer binding protein zeta	Rattus norvegicus	63-67
18359516-9	2008	The infarct volume in the alpha-MSH group was smaller compared to the saline treated group as demonstrated both by MRI and TTC staining.	triphenyltetrazolium	123-126	proopiomelanocortin	Rattus norvegicus	26-35
18707079-4	2008	Dynamic light scattering (DLS) showed a higher tendency to undergo intermolecular aggregation for P(SS 1- co-MA 1) in the presence of TTC, a result that could be related with a higher tendency for TTC to form hydrophobic ion pairs as a consequence of single stacking with the benzene sulfonate groups (BS) of this polyelectrolyte.	triphenyltetrazolium	134-137	major histocompatibility complex, class II, DR beta 1	Homo sapiens	100-104
17882163-6	2008	The intrastriatal injection of recombinant glutathione S-transferase-tagged human DJ-1 (GST-DJ-1) markedly reduced infarct size in 2,3,5-triphenyltetrazolium chloride staining at 3 days after MCAO.	triphenyltetrazolium	131-166	Parkinsonism associated deglycase	Homo sapiens	88-96
16616052-2	2006	NRG-1(3.0 ng/kg) was applied intravascularly 10 min before middle cerebral artery occlusion (MCAO) and then focal cerebral ischemia for 90 min and reperfusion for 24 h. The rats were scored post-reperfusion for neurological deficits and infarct volume in the brain was assessed by 2,3,5-triphenyltetrazolium chloride(TTC).	triphenyltetrazolium	281-316	neuregulin 1	Rattus norvegicus	0-5
17497673-6	2007	TTC-staining showed a significant reduction of infarct volume in the G-CSF-treated group (**P < 0.01).	triphenyltetrazolium	0-3	colony stimulating factor 3	Rattus norvegicus	69-74
16574892-5	2006	Triphenyltetrazolium chloride staining revealed that G-CSF markedly reduced the infarct size.	triphenyltetrazolium	0-29	colony stimulating factor 3	Rattus norvegicus	53-58
15718373-6	2005	Epo treatment produced significant reductions in the mean infarct area and volume at 1 and 3 d after FCI, demonstrated by 2,3,5-triphenyltetrazolium chloride staining.	triphenyltetrazolium	122-157	erythropoietin	Rattus norvegicus	0-3
15936510-3	2005	In female CD-1 mice injected 30 min before MCAO, NGP1-01 (20 mg/kg) reduced infarct area by 42.6% (P < 0.05) compared to vehicle-treated controls as visualized by 2,3,5-triphenyltetrazolium chloride (TTC) staining.	triphenyltetrazolium	166-201	guanine nucleotide binding protein-like 2 (nucleolar)	Mus musculus	49-53
15936510-3	2005	In female CD-1 mice injected 30 min before MCAO, NGP1-01 (20 mg/kg) reduced infarct area by 42.6% (P < 0.05) compared to vehicle-treated controls as visualized by 2,3,5-triphenyltetrazolium chloride (TTC) staining.	triphenyltetrazolium	203-206	guanine nucleotide binding protein-like 2 (nucleolar)	Mus musculus	49-53
12973018-3	2003	After 2 hours of left middle cerebral artery occlusion and 1 day of reperfusion, a 33% smaller infarct (P < 0.05) was observed by 2,3,5-triphenyltetrazolium staining in the brains of E2F1-null mice compared with their E2F1+/+ and +/- littermates.	triphenyltetrazolium	133-159	E2F transcription factor 1	Mus musculus	186-190
15545913-4	2004	Further, 2,3,5-triphenyltetrazolium chloride staining showed that FGF-2 gene transfer decreased infarct volume by 44% as compared with that in the vehicle-treated groups at 2 days after MCAO.	triphenyltetrazolium	9-44	fibroblast growth factor 2	Rattus norvegicus	66-71
14707023-6	2004	Overexpression of the HGF gene resulted in a significant decrease in the infarcted brain area as assessed by triphenyltetrazolium chloride staining, whereas rats transfected with control vector exhibited a wide area of brain death after 24 hours of ischemia.	triphenyltetrazolium	109-138	hepatocyte growth factor	Rattus norvegicus	22-25
11382412-2	2001	Treatment with GDNF significantly reduced the infarct volume stained with 2,3,5-triphenyltetrazolium chloride (TTC) when GDNF was topically applied at 0 and 1 h of reperfusion, but became insignificant at 3 h as compared to vehicle group.	triphenyltetrazolium	74-109	glial cell derived neurotrophic factor	Rattus norvegicus	15-19
11522595-6	2001	Infarct size (determined at the end of reperfusion by triphenyltetrazolium chloride staining) was reduced by angiotensin II in a concentration-dependent manner (from a control value of 27+/-3 to 18+/-4% and 9+/-3% of the LV, respectively).	triphenyltetrazolium	54-83	angiotensinogen	Rattus norvegicus	109-123
12858036-3	2003	Triphenyltetrazolium chloride (TTC) staining of viable brain tissue revealed that the rAAV-delivered IL-1ra gene could rescue the brain tissues from ischemia-induced injury.	triphenyltetrazolium	0-29	interleukin 1 receptor antagonist	Rattus norvegicus	101-107
12858036-3	2003	Triphenyltetrazolium chloride (TTC) staining of viable brain tissue revealed that the rAAV-delivered IL-1ra gene could rescue the brain tissues from ischemia-induced injury.	triphenyltetrazolium	31-34	interleukin 1 receptor antagonist	Rattus norvegicus	101-107
11382412-2	2001	Treatment with GDNF significantly reduced the infarct volume stained with 2,3,5-triphenyltetrazolium chloride (TTC) when GDNF was topically applied at 0 and 1 h of reperfusion, but became insignificant at 3 h as compared to vehicle group.	triphenyltetrazolium	74-109	glial cell derived neurotrophic factor	Rattus norvegicus	121-125
11382412-2	2001	Treatment with GDNF significantly reduced the infarct volume stained with 2,3,5-triphenyltetrazolium chloride (TTC) when GDNF was topically applied at 0 and 1 h of reperfusion, but became insignificant at 3 h as compared to vehicle group.	triphenyltetrazolium	111-114	glial cell derived neurotrophic factor	Rattus norvegicus	15-19
11382412-2	2001	Treatment with GDNF significantly reduced the infarct volume stained with 2,3,5-triphenyltetrazolium chloride (TTC) when GDNF was topically applied at 0 and 1 h of reperfusion, but became insignificant at 3 h as compared to vehicle group.	triphenyltetrazolium	111-114	glial cell derived neurotrophic factor	Rattus norvegicus	121-125
11085892-3	2000	Triphenyltetrazolium chloride tissue stain analysis revealed that the rAAV-delivered GDNF gene could rescue the brain tissues from ischemia-induced injury.	triphenyltetrazolium	0-29	glial cell derived neurotrophic factor	Rattus norvegicus	85-89