PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
34405716-0	2021	miR-128 participates in the pathogenesis of chronic constipation by regulating the p38alpha/M-CSF inflammatory signaling pathway.	mir-128	0-7	mitogen-activated protein kinase 14	Mus musculus	83-91
34780888-5	2022	HAND2-AS1 endogenously sponged miR-128 and suppressed its activity via sequence complementation.	mir-128	31-38	heart and neural crest derivatives expressed 2	Homo sapiens	0-5
34780888-5	2022	HAND2-AS1 endogenously sponged miR-128 and suppressed its activity via sequence complementation.	mir-128	31-38	prostaglandin D2 receptor	Homo sapiens	6-9
34780888-6	2022	Furthermore, HAND2-AS1 enhanced the expression of SIRT1 via binding to miR-128, thereby promoting ABCA1/G1 expression.	mir-128	71-78	heart and neural crest derivatives expressed 2	Homo sapiens	13-18
34780888-6	2022	Furthermore, HAND2-AS1 enhanced the expression of SIRT1 via binding to miR-128, thereby promoting ABCA1/G1 expression.	mir-128	71-78	prostaglandin D2 receptor	Homo sapiens	19-22
34780888-6	2022	Furthermore, HAND2-AS1 enhanced the expression of SIRT1 via binding to miR-128, thereby promoting ABCA1/G1 expression.	mir-128	71-78	sirtuin 1	Homo sapiens	50-55
34780888-6	2022	Furthermore, HAND2-AS1 enhanced the expression of SIRT1 via binding to miR-128, thereby promoting ABCA1/G1 expression.	mir-128	71-78	ATP binding cassette subfamily A member 1	Homo sapiens	98-103
34151409-6	2021	Here, we focused on elucidating the possible role of miR-128 in AD pathology and found that miR-128 regulates the expression of two proteins essential for synaptic transmission, SNAP-25, and synaptotagmin1 (Syt1).	mir-128	53-60	synaptosomal-associated protein 25	Mus musculus	178-185
34151409-6	2021	Here, we focused on elucidating the possible role of miR-128 in AD pathology and found that miR-128 regulates the expression of two proteins essential for synaptic transmission, SNAP-25, and synaptotagmin1 (Syt1).	mir-128	53-60	synaptotagmin I	Mus musculus	207-211
34965835-0	2021	Upregulation of miR-128 Mediates Heart Injury by Activating Wnt/beta-catenin Signaling Pathway in Heart Failure Mice.	mir-128	16-23	Wnt family member 1	Homo sapiens	60-63
34965835-0	2021	Upregulation of miR-128 Mediates Heart Injury by Activating Wnt/beta-catenin Signaling Pathway in Heart Failure Mice.	mir-128	16-23	catenin (cadherin associated protein), beta 1	Mus musculus	64-76
34965835-8	2021	Interference with miR-128 reduces Wnt1/beta-catenin expression in mouse failing hearts and ameliorates heart dysfunctional properties.	mir-128	18-25	wingless-type MMTV integration site family, member 1	Mus musculus	34-38
34965835-8	2021	Interference with miR-128 reduces Wnt1/beta-catenin expression in mouse failing hearts and ameliorates heart dysfunctional properties.	mir-128	18-25	catenin (cadherin associated protein), beta 1	Mus musculus	39-51
34965835-9	2021	We identified miR-128 directly targets to Axin1, an inhibitor of Wnt/beta-catenin signaling, and suppresses its inhibition on Wnt1/beta-catenin.	mir-128	14-21	axin 1	Mus musculus	42-47
34965835-9	2021	We identified miR-128 directly targets to Axin1, an inhibitor of Wnt/beta-catenin signaling, and suppresses its inhibition on Wnt1/beta-catenin.	mir-128	14-21	Wnt family member 1	Homo sapiens	65-68
34965835-9	2021	We identified miR-128 directly targets to Axin1, an inhibitor of Wnt/beta-catenin signaling, and suppresses its inhibition on Wnt1/beta-catenin.	mir-128	14-21	catenin (cadherin associated protein), beta 1	Mus musculus	69-81
34965835-9	2021	We identified miR-128 directly targets to Axin1, an inhibitor of Wnt/beta-catenin signaling, and suppresses its inhibition on Wnt1/beta-catenin.	mir-128	14-21	wingless-type MMTV integration site family, member 1	Mus musculus	126-130
34965835-9	2021	We identified miR-128 directly targets to Axin1, an inhibitor of Wnt/beta-catenin signaling, and suppresses its inhibition on Wnt1/beta-catenin.	mir-128	14-21	catenin (cadherin associated protein), beta 1	Mus musculus	131-143
34965835-10	2021	Our study provides evidence indicating miR-128 as an inducer of HF and cardiac hypertrophy by enhancing Wnt1/beta-catenin in an Axin1-dependent nature.	mir-128	39-46	wingless-type MMTV integration site family, member 1	Mus musculus	104-108
34965835-10	2021	Our study provides evidence indicating miR-128 as an inducer of HF and cardiac hypertrophy by enhancing Wnt1/beta-catenin in an Axin1-dependent nature.	mir-128	39-46	catenin (cadherin associated protein), beta 1	Mus musculus	109-121
34965835-10	2021	Our study provides evidence indicating miR-128 as an inducer of HF and cardiac hypertrophy by enhancing Wnt1/beta-catenin in an Axin1-dependent nature.	mir-128	39-46	axin 1	Mus musculus	128-133
34812491-14	2021	This study elaborated the mechanism by which Gs-Rb1 alleviates hepatotoxicity induced by foodborne 3-MCPD by stimulating autophagic flux via miR-128-targeted TFEB, which provides a reliable theoretical basis and target for the use of natural substances to reduce the harm of food processing pollutants on the human body.	mir-128	141-148	RB transcriptional corepressor 1	Homo sapiens	48-51
34812491-14	2021	This study elaborated the mechanism by which Gs-Rb1 alleviates hepatotoxicity induced by foodborne 3-MCPD by stimulating autophagic flux via miR-128-targeted TFEB, which provides a reliable theoretical basis and target for the use of natural substances to reduce the harm of food processing pollutants on the human body.	mir-128	141-148	transcription factor EB	Homo sapiens	158-162
34405716-9	2021	Furthermore, co-culture assays indicated that regulating expression of miR-128 in colonic epithelial cells induced the secretion of IL-6 and TNF-alpha by macrophages.	mir-128	71-78	interleukin 6	Mus musculus	132-136
34405716-9	2021	Furthermore, co-culture assays indicated that regulating expression of miR-128 in colonic epithelial cells induced the secretion of IL-6 and TNF-alpha by macrophages.	mir-128	71-78	tumor necrosis factor	Mus musculus	141-150
34405716-10	2021	In conclusion, our study demonstrated that miR-128 regulated the p38alpha/M-CSF signaling pathway to promote chronic inflammatory responses and changes in the immune microenvironment of the colon, thereby offering potential insights into the pathogenesis of CC and therapeutic targets for its treatment.	mir-128	43-50	mitogen-activated protein kinase 14	Mus musculus	65-73
34092219-12	2021	CONCLUSIONS: Overall, down-regulating SNHG16 alleviated the sepsis-mediated ALI by regulating miR-128-3p/HMGB3.	mir-128	94-101	high mobility group box 3	Rattus norvegicus	105-110
34384932-5	2021	We, herein using in silico analysis followed by in vitro experiments in C2C12 myoblasts, showed that miR-128 reduces mitochondrial biogenesis by directly targeting PGC1alpha.	mir-128	101-108	PPARG coactivator 1 alpha	Homo sapiens	164-173
34384932-6	2021	The expression of downstream genes, nuclear respiratory factors 1 and 2 (NRF1 and NRF2, respectively), and mitochondrial transcription factor A (TFAM) were decreased in C2C12 myoblasts upon overexpression of miR-128.	mir-128	208-215	nuclear respiratory factor 1	Homo sapiens	36-71
34384932-6	2021	The expression of downstream genes, nuclear respiratory factors 1 and 2 (NRF1 and NRF2, respectively), and mitochondrial transcription factor A (TFAM) were decreased in C2C12 myoblasts upon overexpression of miR-128.	mir-128	208-215	nuclear respiratory factor 1	Homo sapiens	73-77
34384932-6	2021	The expression of downstream genes, nuclear respiratory factors 1 and 2 (NRF1 and NRF2, respectively), and mitochondrial transcription factor A (TFAM) were decreased in C2C12 myoblasts upon overexpression of miR-128.	mir-128	208-215	NFE2 like bZIP transcription factor 2	Homo sapiens	82-86
34384932-6	2021	The expression of downstream genes, nuclear respiratory factors 1 and 2 (NRF1 and NRF2, respectively), and mitochondrial transcription factor A (TFAM) were decreased in C2C12 myoblasts upon overexpression of miR-128.	mir-128	208-215	transcription factor A, mitochondrial	Homo sapiens	107-143
34384932-6	2021	The expression of downstream genes, nuclear respiratory factors 1 and 2 (NRF1 and NRF2, respectively), and mitochondrial transcription factor A (TFAM) were decreased in C2C12 myoblasts upon overexpression of miR-128.	mir-128	208-215	transcription factor A, mitochondrial	Homo sapiens	145-149
34384932-7	2021	Also, miR-128 is shown to promote mitochondrial dysfunction by directly targeting NADH Dehydrogenase (Ubiquinone) Fe-S Protein 4 (NDUFS4).	mir-128	6-13	NADH:ubiquinone oxidoreductase subunit S4	Homo sapiens	130-136
34384932-8	2021	The mitochondrial dynamics and morphology were impaired post miR-128 overexpression, as revealed by downregulation of fusion proteins (mitofusin1 and 2, i.e., MFN1 and MFN2, respectively) and upregulation of fission protein (dynamin-related protein 1, i.e., DRP1).	mir-128	61-68	mitofusin 1	Homo sapiens	135-151
34384932-8	2021	The mitochondrial dynamics and morphology were impaired post miR-128 overexpression, as revealed by downregulation of fusion proteins (mitofusin1 and 2, i.e., MFN1 and MFN2, respectively) and upregulation of fission protein (dynamin-related protein 1, i.e., DRP1).	mir-128	61-68	mitofusin 1	Homo sapiens	159-163
34384932-8	2021	The mitochondrial dynamics and morphology were impaired post miR-128 overexpression, as revealed by downregulation of fusion proteins (mitofusin1 and 2, i.e., MFN1 and MFN2, respectively) and upregulation of fission protein (dynamin-related protein 1, i.e., DRP1).	mir-128	61-68	mitofusin 2	Homo sapiens	168-172
34384932-8	2021	The mitochondrial dynamics and morphology were impaired post miR-128 overexpression, as revealed by downregulation of fusion proteins (mitofusin1 and 2, i.e., MFN1 and MFN2, respectively) and upregulation of fission protein (dynamin-related protein 1, i.e., DRP1).	mir-128	61-68	dynamin 1 like	Homo sapiens	225-250
34384932-8	2021	The mitochondrial dynamics and morphology were impaired post miR-128 overexpression, as revealed by downregulation of fusion proteins (mitofusin1 and 2, i.e., MFN1 and MFN2, respectively) and upregulation of fission protein (dynamin-related protein 1, i.e., DRP1).	mir-128	61-68	dynamin 1 like	Homo sapiens	258-262
34108447-9	2021	Furthermore, PHF6 overexpression affected IEC-6 cells by activating PI3K/AKT signaling which was mediated by the miR-128-3p/PHF6 axis.	mir-128	113-120	PHD finger protein 6	Rattus norvegicus	13-17
34108447-9	2021	Furthermore, PHF6 overexpression affected IEC-6 cells by activating PI3K/AKT signaling which was mediated by the miR-128-3p/PHF6 axis.	mir-128	113-120	AKT serine/threonine kinase 1	Rattus norvegicus	73-76
34108447-9	2021	Furthermore, PHF6 overexpression affected IEC-6 cells by activating PI3K/AKT signaling which was mediated by the miR-128-3p/PHF6 axis.	mir-128	113-120	PHD finger protein 6	Rattus norvegicus	124-128
34646427-0	2021	Downregulation of miR-128 Ameliorates Ang II-Induced Cardiac Remodeling via SIRT1/PIK3R1 Multiple Targets.	mir-128	18-25	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	38-44
34646427-0	2021	Downregulation of miR-128 Ameliorates Ang II-Induced Cardiac Remodeling via SIRT1/PIK3R1 Multiple Targets.	mir-128	18-25	sirtuin 1	Mus musculus	76-81
34646427-0	2021	Downregulation of miR-128 Ameliorates Ang II-Induced Cardiac Remodeling via SIRT1/PIK3R1 Multiple Targets.	mir-128	18-25	phosphoinositide-3-kinase regulatory subunit 1	Mus musculus	82-88
34646427-3	2021	The aim of the present study was to investigate the role of miR-128 in chronic angiotensin II (Ang II) infusion-induced cardiac remodeling and its underlying mechanism.	mir-128	60-67	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	79-93
34646427-3	2021	The aim of the present study was to investigate the role of miR-128 in chronic angiotensin II (Ang II) infusion-induced cardiac remodeling and its underlying mechanism.	mir-128	60-67	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	95-101
34646427-7	2021	Our results showed that chronic Ang II delivery for 28 days induced cardiac dysfunction, hypertrophy, fibrosis, apoptosis, and oxidative stress in the mice, while the miR-128 expression was notably enhanced in the left ventricle.	mir-128	167-174	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	32-38
34646427-8	2021	Silencing miR-128 in the hearts of mice ameliorated Ang II-induced cardiac dysfunction, hypertrophy, fibrosis apoptosis, and oxidative stress injury.	mir-128	10-17	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	52-58
34646427-9	2021	Moreover, Ang II induced excessive autophagy in the mouse hearts, which was suppressed by miR-128 knockdown.	mir-128	90-97	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	10-16
34646427-10	2021	In cultured cells, Ang II treatment induced a marked elevation in the miR-128 expression.	mir-128	70-77	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	19-25
34646427-11	2021	Downregulation of miR-128 in the cells by transfection with miR-128 antagomir attenuated Ang II-induced apoptosis and oxidative injury probably via directly targeting on the SIRT1/p53 pathway.	mir-128	18-25	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	89-95
34646427-11	2021	Downregulation of miR-128 in the cells by transfection with miR-128 antagomir attenuated Ang II-induced apoptosis and oxidative injury probably via directly targeting on the SIRT1/p53 pathway.	mir-128	18-25	sirtuin 1	Mus musculus	174-179
34646427-11	2021	Downregulation of miR-128 in the cells by transfection with miR-128 antagomir attenuated Ang II-induced apoptosis and oxidative injury probably via directly targeting on the SIRT1/p53 pathway.	mir-128	18-25	transformation related protein 53, pseudogene	Mus musculus	180-183
34646427-11	2021	Downregulation of miR-128 in the cells by transfection with miR-128 antagomir attenuated Ang II-induced apoptosis and oxidative injury probably via directly targeting on the SIRT1/p53 pathway.	mir-128	60-67	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	89-95
34646427-11	2021	Downregulation of miR-128 in the cells by transfection with miR-128 antagomir attenuated Ang II-induced apoptosis and oxidative injury probably via directly targeting on the SIRT1/p53 pathway.	mir-128	60-67	sirtuin 1	Mus musculus	174-179
34646427-11	2021	Downregulation of miR-128 in the cells by transfection with miR-128 antagomir attenuated Ang II-induced apoptosis and oxidative injury probably via directly targeting on the SIRT1/p53 pathway.	mir-128	60-67	transformation related protein 53, pseudogene	Mus musculus	180-183
34646427-12	2021	Intriguingly, we found that miR-128 inhibition activated PIK3R1/Akt/mTOR pathway and thereby significantly damped Ang II-stimulated pathological autophagy in cardiomyocytes, which consequently mitigated cell oxidative stress and apoptosis.	mir-128	28-35	phosphoinositide-3-kinase regulatory subunit 1	Mus musculus	57-63
34646427-12	2021	Intriguingly, we found that miR-128 inhibition activated PIK3R1/Akt/mTOR pathway and thereby significantly damped Ang II-stimulated pathological autophagy in cardiomyocytes, which consequently mitigated cell oxidative stress and apoptosis.	mir-128	28-35	thymoma viral proto-oncogene 1	Mus musculus	64-67
34646427-12	2021	Intriguingly, we found that miR-128 inhibition activated PIK3R1/Akt/mTOR pathway and thereby significantly damped Ang II-stimulated pathological autophagy in cardiomyocytes, which consequently mitigated cell oxidative stress and apoptosis.	mir-128	28-35	mechanistic target of rapamycin kinase	Mus musculus	68-72
34646427-12	2021	Intriguingly, we found that miR-128 inhibition activated PIK3R1/Akt/mTOR pathway and thereby significantly damped Ang II-stimulated pathological autophagy in cardiomyocytes, which consequently mitigated cell oxidative stress and apoptosis.	mir-128	28-35	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	114-120
34646427-13	2021	In conclusion, downregulation of miR-128 ameliorates Ang II-provoked cardiac oxidative stress, hypertrophy, fibrosis, apoptosis, and dysfunction in mice, likely through targeting on PIK3R1/Akt/mTORC1 and/or SIRT1/p53 pathways.	mir-128	33-40	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	53-59
34646427-13	2021	In conclusion, downregulation of miR-128 ameliorates Ang II-provoked cardiac oxidative stress, hypertrophy, fibrosis, apoptosis, and dysfunction in mice, likely through targeting on PIK3R1/Akt/mTORC1 and/or SIRT1/p53 pathways.	mir-128	33-40	phosphoinositide-3-kinase regulatory subunit 1	Mus musculus	182-188
34646427-13	2021	In conclusion, downregulation of miR-128 ameliorates Ang II-provoked cardiac oxidative stress, hypertrophy, fibrosis, apoptosis, and dysfunction in mice, likely through targeting on PIK3R1/Akt/mTORC1 and/or SIRT1/p53 pathways.	mir-128	33-40	thymoma viral proto-oncogene 1	Mus musculus	189-192
34646427-13	2021	In conclusion, downregulation of miR-128 ameliorates Ang II-provoked cardiac oxidative stress, hypertrophy, fibrosis, apoptosis, and dysfunction in mice, likely through targeting on PIK3R1/Akt/mTORC1 and/or SIRT1/p53 pathways.	mir-128	33-40	CREB regulated transcription coactivator 1	Mus musculus	193-199
34646427-13	2021	In conclusion, downregulation of miR-128 ameliorates Ang II-provoked cardiac oxidative stress, hypertrophy, fibrosis, apoptosis, and dysfunction in mice, likely through targeting on PIK3R1/Akt/mTORC1 and/or SIRT1/p53 pathways.	mir-128	33-40	sirtuin 1	Mus musculus	207-212
34646427-13	2021	In conclusion, downregulation of miR-128 ameliorates Ang II-provoked cardiac oxidative stress, hypertrophy, fibrosis, apoptosis, and dysfunction in mice, likely through targeting on PIK3R1/Akt/mTORC1 and/or SIRT1/p53 pathways.	mir-128	33-40	transformation related protein 53, pseudogene	Mus musculus	213-216
34296305-0	2021	Upregulation of miR-128 inhibits neuronal cell apoptosis following spinal cord injury via FasL downregulation by repressing ULK1.	mir-128	16-23	unc-51 like autophagy activating kinase 1	Rattus norvegicus	124-128
34296305-10	2021	Overexpression of miR-128 or downregulation of ULK1 inhibited neuronal cell apoptosis and inflammation as evidenced by an increased BBB score and more neurons and nestin-positive cells, but reduced expression of inflammatory and apoptosis-related factors.	mir-128	18-25	nestin	Rattus norvegicus	163-169
34296305-11	2021	ULK1 was negatively regulated by miR-128, whereas FasL was positively regulated by ULK1.	mir-128	33-40	unc-51 like autophagy activating kinase 1	Rattus norvegicus	0-4
34296305-13	2021	In conclusion, the upregulation of miR-128 depresses neuronal cell apoptosis by downregulating ULK1, thereby attenuating SCI via the downregulation of FasL.	mir-128	35-42	unc-51 like autophagy activating kinase 1	Rattus norvegicus	95-99
34296305-13	2021	In conclusion, the upregulation of miR-128 depresses neuronal cell apoptosis by downregulating ULK1, thereby attenuating SCI via the downregulation of FasL.	mir-128	35-42	Fas ligand	Rattus norvegicus	151-155
34553041-6	2021	In addition, BMI-1, ABCC-5, E2F3, and c-MET were identified as candidate targets of miR-128, and the overexpression of miR-128 significantly reduced mRNA/protein levels of BMI-1, ABCC-5, E2F3, and c-MET in A549 and H460 cells.	mir-128	84-91	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	13-18
34553041-6	2021	In addition, BMI-1, ABCC-5, E2F3, and c-MET were identified as candidate targets of miR-128, and the overexpression of miR-128 significantly reduced mRNA/protein levels of BMI-1, ABCC-5, E2F3, and c-MET in A549 and H460 cells.	mir-128	84-91	ATP binding cassette subfamily C member 5	Homo sapiens	20-26
34553041-6	2021	In addition, BMI-1, ABCC-5, E2F3, and c-MET were identified as candidate targets of miR-128, and the overexpression of miR-128 significantly reduced mRNA/protein levels of BMI-1, ABCC-5, E2F3, and c-MET in A549 and H460 cells.	mir-128	84-91	E2F transcription factor 3	Homo sapiens	28-32
34553041-6	2021	In addition, BMI-1, ABCC-5, E2F3, and c-MET were identified as candidate targets of miR-128, and the overexpression of miR-128 significantly reduced mRNA/protein levels of BMI-1, ABCC-5, E2F3, and c-MET in A549 and H460 cells.	mir-128	84-91	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	38-43
34553041-6	2021	In addition, BMI-1, ABCC-5, E2F3, and c-MET were identified as candidate targets of miR-128, and the overexpression of miR-128 significantly reduced mRNA/protein levels of BMI-1, ABCC-5, E2F3, and c-MET in A549 and H460 cells.	mir-128	84-91	E2F transcription factor 3	Homo sapiens	187-191
34553041-6	2021	In addition, BMI-1, ABCC-5, E2F3, and c-MET were identified as candidate targets of miR-128, and the overexpression of miR-128 significantly reduced mRNA/protein levels of BMI-1, ABCC-5, E2F3, and c-MET in A549 and H460 cells.	mir-128	84-91	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	197-202
34124052-4	2021	Furthermore, we identified Ppp1cc as a direct target gene of miR-128 regulated by the PERK signaling pathway and showed that its repression is critical for a feedback loop that regulates the activity of UPR-associated signaling pathways, leading to cell migration, cell fusion, endoplasmic reticulum expansion, and myotube formation during myoblast differentiation.	mir-128	61-68	protein phosphatase 1 catalytic subunit gamma	Mus musculus	27-33
34124052-4	2021	Furthermore, we identified Ppp1cc as a direct target gene of miR-128 regulated by the PERK signaling pathway and showed that its repression is critical for a feedback loop that regulates the activity of UPR-associated signaling pathways, leading to cell migration, cell fusion, endoplasmic reticulum expansion, and myotube formation during myoblast differentiation.	mir-128	61-68	eukaryotic translation initiation factor 2 alpha kinase 3	Mus musculus	86-90
35358540-6	2022	Finally, functional assays carried out in vitro proved SNHG16 could modulate GIST cell proliferation, migration, invasion and apoptosis via miR-128-3p/CASC3 axis.	mir-128	140-147	small nucleolar RNA host gene 16	Homo sapiens	55-61
35587369-10	2022	These results suggested that KCNQ1OT1 regulates the osteoclast differentiation via the miR-128-3p/NFAT5 axis.	mir-128	87-94	KCNQ1 overlapping transcript 1	Mus musculus	29-37
35587369-10	2022	These results suggested that KCNQ1OT1 regulates the osteoclast differentiation via the miR-128-3p/NFAT5 axis.	mir-128	87-94	nuclear factor of activated T cells 5	Mus musculus	98-103
35252056-13	2022	Conclusion: The present results indicate that the miR-128-3p/c-Met axis may be potential therapeutic targets for circumventing lenvatinib resistance in HCC and warrant further investigation.	mir-128	50-57	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	61-66
33994812-14	2021	MiR-128 was predicted to be a target of HCP5.	mir-128	0-7	HLA complex P5	Homo sapiens	40-44
35142579-6	2022	NG-R1 treatment reduced miR-128-2-5p expression in the lung tissue, increased Tollip expression, inhibited HMGB1, TLR4, TRAF6, IRAK1, MyD88, NF-kappaB65, and p-IkappaB-alpha expression levels, suppressed NF-kappaB65 and the TLR4 expression levels, reduced MPO activity, reduced TNF-alpha, IL-1beta, IL-6, and ICAM-1 levels in BALF, and alleviated SAP-induced ALI.	mir-128	24-31	reticulon 4 receptor	Rattus norvegicus	0-5
35082915-0	2022	lncRNA-NEAT1 Sponges miR-128 to Promote Inflammatory Reaction and Phenotypic Transformation of Airway Smooth Muscle Cells.	mir-128	21-28	nuclear paraspeckle assembly transcript 1	Homo sapiens	7-12
35082915-12	2022	Conclusion: lncRNA-NEAT1 sponges miR-128 to boost PDGF-BB-induced inflammatory reaction and phenotypic transformation of ASMCs to aggravate the occurrence and development of childhood bronchial asthma.	mir-128	33-40	nuclear paraspeckle assembly transcript 1	Homo sapiens	19-24
35059433-0	2021	Exosome-Transmitted miR-128 Targets CCL18 to Inhibit the Proliferation and Metastasis of Urothelial Carcinoma.	mir-128	20-27	C-C motif chemokine ligand 18	Homo sapiens	36-41
35059433-10	2021	The exosome-transmitted miR-128 can inhibit cell proliferation (p < 0.05), invasion (p < 0.05), and migration (p < 0.05) in UCs, and these effects can be reversed by CCL18.	mir-128	24-31	C-C motif chemokine ligand 18	Homo sapiens	166-171
35059433-11	2021	In terms of apoptosis, miR-128 was able to promote the occurrence of BUC T24 apoptosis (p < 0.05), which can also be reversed by CCL18.	mir-128	23-30	C-C motif chemokine ligand 18	Homo sapiens	129-134
35059433-13	2021	Conclusion: The miR-128 inhibits the proliferation, invasion, migration of UCs, and promotes its apoptosis by regulating CCL18 secretion.	mir-128	16-23	C-C motif chemokine ligand 18	Homo sapiens	121-126
35000531-7	2022	Meanwhile, CCL2 down-regulating significantly repressed the proliferation, migration, and invasion by regulating miR-128.	mir-128	113-120	C-C motif chemokine ligand 2	Homo sapiens	11-15
35000531-8	2022	In addition, we proved miR-128 was a direct target of CCL2 through double luciferase assay and bioinformatics analysis.	mir-128	23-30	C-C motif chemokine ligand 2	Homo sapiens	54-58
35000531-10	2022	More importantly, miR-128 could reverse the effects of lncRNA CCL2 knocked down.	mir-128	18-25	C-C motif chemokine ligand 2	Homo sapiens	62-66
35000531-12	2022	Meanwhile, miR-128 mimic and the knockout of CCL2 distinctly decreased PARP2 protein level.	mir-128	11-18	poly(ADP-ribose) polymerase 2	Homo sapiens	71-76
35000531-13	2022	Additionally, luciferase report experiments certificated that PARP2 targeted miR-128, implying PARP2 directly interacted with miR-128 in gastric cancer.	mir-128	77-84	poly(ADP-ribose) polymerase 2	Homo sapiens	62-67
35000531-13	2022	Additionally, luciferase report experiments certificated that PARP2 targeted miR-128, implying PARP2 directly interacted with miR-128 in gastric cancer.	mir-128	77-84	poly(ADP-ribose) polymerase 2	Homo sapiens	95-100
35000531-13	2022	Additionally, luciferase report experiments certificated that PARP2 targeted miR-128, implying PARP2 directly interacted with miR-128 in gastric cancer.	mir-128	126-133	poly(ADP-ribose) polymerase 2	Homo sapiens	62-67
35000531-13	2022	Additionally, luciferase report experiments certificated that PARP2 targeted miR-128, implying PARP2 directly interacted with miR-128 in gastric cancer.	mir-128	126-133	poly(ADP-ribose) polymerase 2	Homo sapiens	95-100
35000531-14	2022	More interestingly, the downregulation of PARP could reverse the trend triggered by miR-128 inhibitor in gastric tumor.	mir-128	84-91	collagen type XI alpha 2 chain	Homo sapiens	42-46
33750005-9	2021	miR-128 has been reported to inhibit dendritic growth and branching in mouse brain neurons, which directly opposes the novel functions of R3HDM1.	mir-128	0-7	R3H domain containing 1	Mus musculus	138-144
33743142-0	2021	B7-H3, Negatively Regulated by miR-128, Promotes Colorectal Cancer Cell Proliferation and Migration.	mir-128	31-38	CD276 antigen	Mus musculus	0-5
33743142-8	2021	Besides, the regulatory relationship between miR-128 and B7-H3 was validated through dual-luciferase reporter gene assay, qRT-PCR, and western blotting.	mir-128	45-52	CD276 antigen	Mus musculus	57-62
33743142-11	2021	Moreover, B7-H3 was identified as a target of miR-128, and miR-128 negatively regulated B7-H3 expression in CRC cells.	mir-128	46-53	CD276 antigen	Mus musculus	10-15
33743142-11	2021	Moreover, B7-H3 was identified as a target of miR-128, and miR-128 negatively regulated B7-H3 expression in CRC cells.	mir-128	59-66	CD276 antigen	Mus musculus	88-93
33743142-13	2021	Besides, B7-H3 expression is negatively regulated by miR-128 in CRC.	mir-128	53-60	CD276 antigen	Mus musculus	9-14
34047500-0	2021	Mechanism of ARPP21 antagonistic intron miR-128 on neurological function repair after stroke.	mir-128	40-47	cAMP regulated phosphoprotein 21	Rattus norvegicus	13-19
34047500-2	2021	ARPP21 is a conserved host gene of miR-128 controlling neurodevelopmental functions.	mir-128	35-42	cAMP regulated phosphoprotein 21	Rattus norvegicus	0-6
34047500-3	2021	This study investigated the mechanism of ARPP21 antagonistic intron miR-128 on neurological function repair after stroke.	mir-128	68-75	cAMP regulated phosphoprotein 21	Rattus norvegicus	41-47
34047500-8	2021	The binding relationship between miR-128 and CREB1 was verified.	mir-128	33-40	cAMP responsive element binding protein 1	Mus musculus	45-50
34047500-14	2021	miR-128 mimic could reverse these effects of pc-ARPP21 on neurons and astrocytes.	mir-128	0-7	cAMP regulated phosphoprotein 21	Homo sapiens	48-54
34047500-15	2021	miR-128 targeted CREB1 and reduced BDNF secretion.	mir-128	0-7	cAMP responsive element binding protein 1	Homo sapiens	17-22
34047500-15	2021	miR-128 targeted CREB1 and reduced BDNF secretion.	mir-128	0-7	brain derived neurotrophic factor	Homo sapiens	35-39
34047500-17	2021	CONCLUSION: ARPP21 upregulated CREB1 and BDNF expressions by antagonizing miR-128, thus inhibiting neuronal apoptosis and promoting neurological function repair after stroke.	mir-128	74-81	cAMP regulated phosphoprotein 21	Rattus norvegicus	12-18
34047500-17	2021	CONCLUSION: ARPP21 upregulated CREB1 and BDNF expressions by antagonizing miR-128, thus inhibiting neuronal apoptosis and promoting neurological function repair after stroke.	mir-128	74-81	cAMP responsive element binding protein 1	Rattus norvegicus	31-36
34047500-17	2021	CONCLUSION: ARPP21 upregulated CREB1 and BDNF expressions by antagonizing miR-128, thus inhibiting neuronal apoptosis and promoting neurological function repair after stroke.	mir-128	74-81	brain-derived neurotrophic factor	Rattus norvegicus	41-45
33993846-5	2021	miR-128 was decreased in GC patients and sponged by HCP5.	mir-128	0-7	HLA complex P5	Homo sapiens	52-56
33993846-6	2021	HMGA2 was targeted by miR-128 and was increased in GC patients.	mir-128	22-29	high mobility group AT-hook 2	Homo sapiens	0-5
33993846-7	2021	HCP5 aggravated the resistance of GC cells to DDP in vitro by elevating HMGA2 expression via sponging miR-128.	mir-128	102-109	HLA complex P5	Homo sapiens	0-4
33993846-7	2021	HCP5 aggravated the resistance of GC cells to DDP in vitro by elevating HMGA2 expression via sponging miR-128.	mir-128	102-109	high mobility group AT-hook 2	Homo sapiens	72-77
33994812-15	2021	It was demonstrated that HCP5 directly bound to miR-128 and regulated its expression in glioma cells.	mir-128	48-55	HLA complex P5	Homo sapiens	25-29
33994812-16	2021	Furthermore, the effects of HCP5 knockdown on radiosensitivity of glioma cells were attenuated by the inhibitor of miR-128.	mir-128	115-122	HLA complex P5	Homo sapiens	28-32
33986789-13	2021	Conclusions: miR-128-3p inhibition attenuated DOX-related acute cardiac injury via the regulation of PPAR-gamma in mice.	mir-128	13-20	peroxisome proliferator activated receptor gamma	Mus musculus	101-111
33826315-4	2021	miR-128 can directly target PPARGC1A and inhibit its expression.	mir-128	0-7	PPARG coactivator 1 alpha	Bos taurus	28-36
33622342-9	2021	Moreover, miR-128 mimics partially abrogated the effect of LINC00477 on granulosa cells.	mir-128	10-17	long intergenic non-protein coding RNA 477	Homo sapiens	59-68
33848670-13	2021	Knockdown of miR-128 restored the altered proliferation, migration and invasion and the expression of ZEB1 and E-cadherin caused by knockdown of PVT1.	mir-128	13-20	zinc finger E-box binding homeobox 1	Homo sapiens	102-106
33848670-13	2021	Knockdown of miR-128 restored the altered proliferation, migration and invasion and the expression of ZEB1 and E-cadherin caused by knockdown of PVT1.	mir-128	13-20	cadherin 1	Homo sapiens	111-121
33848670-13	2021	Knockdown of miR-128 restored the altered proliferation, migration and invasion and the expression of ZEB1 and E-cadherin caused by knockdown of PVT1.	mir-128	13-20	Pvt1 oncogene	Homo sapiens	145-149
33746773-9	2021	Either overexpression of miR-128 or downregulation of beta-catenin by CWP232228, a beta-catenin-specific antagonist, blocked the promoting roles of ETS1 in cells.	mir-128	25-32	E26 avian leukemia oncogene 1, 5' domain	Mus musculus	148-152
33746773-10	2021	To conclude, this study provided evidence that ETS1 suppresses miR-128 transcription to activate the following HOXA13/beta-catenin axis, therefore promoting osteogenic differentiation ability of MC3T3-E1 cells.	mir-128	63-70	E26 avian leukemia oncogene 1, 5' domain	Mus musculus	47-51
33746773-10	2021	To conclude, this study provided evidence that ETS1 suppresses miR-128 transcription to activate the following HOXA13/beta-catenin axis, therefore promoting osteogenic differentiation ability of MC3T3-E1 cells.	mir-128	63-70	homeobox A13	Mus musculus	111-117
33746773-10	2021	To conclude, this study provided evidence that ETS1 suppresses miR-128 transcription to activate the following HOXA13/beta-catenin axis, therefore promoting osteogenic differentiation ability of MC3T3-E1 cells.	mir-128	63-70	catenin (cadherin associated protein), beta 1	Mus musculus	118-130
33347907-5	2021	In this study, miR-128 was identified as a negative regulator to participate in the NF-kappaB signaling pathway by targeting TAB2 in miiuy croaker.	mir-128	15-22	nuclear factor kappa B subunit 1	Homo sapiens	84-93
33347907-9	2021	Particularly, we found that miR-128 can regulate TAB2-mediated NF-kappaB signaling pathways.	mir-128	28-35	nuclear factor kappa B subunit 1	Homo sapiens	63-72
33347907-10	2021	In summary, our results indicate that miR-128 plays a critical role in suppressing inflammatory responses by regulating the TAB2-mediated NF-kappaB signaling pathway in miiuy croaker.	mir-128	38-45	nuclear factor kappa B subunit 1	Homo sapiens	138-147
32772776-0	2021	miR-128 regulated the proliferation and autophagy in porcine adipose-derived stem cells through targeting the JNK signaling pathway.	mir-128	0-7	mitogen-activated protein kinase 8	Homo sapiens	110-113
32772776-7	2021	However, followed by the block of the JNK signaling pathway using SP600125 inhibitor, the effects of miR-128 on the proliferation, apoptosis and autophagy of porcine ASCs were significantly suppressed.	mir-128	101-108	mitogen-activated protein kinase 8	Homo sapiens	38-41
32772776-8	2021	CONCLUSION: It is demonstrated that the miR-128-JNK signaling pathway is a potential therapeutic target for the treatment of obesity.	mir-128	40-47	mitogen-activated protein kinase 8	Homo sapiens	48-51
33571908-0	2021	miR-128 regulates epilepsy sensitivity in mice by suppressing SNAP-25 and SYT1 expression in the hippocampus.	mir-128	0-7	synaptosomal-associated protein 25	Mus musculus	62-69
33571908-0	2021	miR-128 regulates epilepsy sensitivity in mice by suppressing SNAP-25 and SYT1 expression in the hippocampus.	mir-128	0-7	synaptotagmin I	Mus musculus	74-78
33571908-3	2021	Both SNAP-25 and SYT1 are regulated by miR-128 in vitro and in vivo.	mir-128	39-46	synaptosomal-associated protein 25	Mus musculus	5-12
33571908-3	2021	Both SNAP-25 and SYT1 are regulated by miR-128 in vitro and in vivo.	mir-128	39-46	synaptotagmin I	Mus musculus	17-21
33571908-4	2021	Overexpressing miR-128 in cultured neurons decreased neurotransmitter released by suppressing SNAP-25 and SYT1 expression.	mir-128	15-22	synaptosomal-associated protein 25	Mus musculus	94-101
33571908-4	2021	Overexpressing miR-128 in cultured neurons decreased neurotransmitter released by suppressing SNAP-25 and SYT1 expression.	mir-128	15-22	synaptotagmin I	Mus musculus	106-110
33622342-10	2021	CONCLUSION: LINC00477 may function as a ceRNA to inhibit proliferation and apoptosis of granulosa cells by modulating miR-128 expression.	mir-128	118-125	long intergenic non-protein coding RNA 477	Homo sapiens	12-21
33379338-10	2020	Notably, miR-128 mediated SSR-enhanced HeLa and CaSki cells" adhesion and metastasis through suppressed ITGA5, ITGB5, sLex, CEACAM-6, MMP9, and MMP23 transcript levels.	mir-128	9-16	integrin subunit alpha 5	Homo sapiens	104-109
33542630-10	2021	The serum miR-128 levels in AD patients were positively correlated with the serum IL-1beta (r=0.798, P<0.01) and serum TNF-alpha levels (r=0.733, P<0.01).	mir-128	10-17	interleukin 1 alpha	Homo sapiens	82-90
33542630-10	2021	The serum miR-128 levels in AD patients were positively correlated with the serum IL-1beta (r=0.798, P<0.01) and serum TNF-alpha levels (r=0.733, P<0.01).	mir-128	10-17	tumor necrosis factor	Homo sapiens	119-128
33472642-6	2021	Genetic knockdown of miR-128 in bone neoplasms cells suppressed the activation of the Wnt/beta-catenin and epithelial-mesenchymal transition (EMT) signaling pathways.	mir-128	21-28	catenin beta 1	Homo sapiens	90-102
33472642-9	2021	CONCLUSIONS: These results define a tumor-regulated function for miR-128 in bone neoplasms by down-regulation of the Wnt/beta-catenin and EMT signal pathways, which provided a potential target for bone neoplasms gene therapy.	mir-128	65-72	catenin beta 1	Homo sapiens	121-133
33080252-5	2021	Computational analysis identified the transcription factor EYA4 as a candidate target consistent with the observation that miR-128 and EYA4 display similar expression profiles.	mir-128	123-130	EYA transcriptional coactivator and phosphatase 4	Gallus gallus	59-63
33080252-7	2021	In vivo experiments also suggest that EYA4 is regulated by miR-128.	mir-128	59-66	EYA transcriptional coactivator and phosphatase 4	Gallus gallus	38-42
33080252-11	2021	Our results reveal that miR-128 is involved in regulating skeletal myogenesis by directly targeting EYA4 with indirect effects on other PSED members, including SIX4 and PAX3.	mir-128	24-31	EYA transcriptional coactivator and phosphatase 4	Gallus gallus	100-104
33080252-11	2021	Our results reveal that miR-128 is involved in regulating skeletal myogenesis by directly targeting EYA4 with indirect effects on other PSED members, including SIX4 and PAX3.	mir-128	24-31	SIX homeobox 5	Gallus gallus	160-164
33080252-11	2021	Our results reveal that miR-128 is involved in regulating skeletal myogenesis by directly targeting EYA4 with indirect effects on other PSED members, including SIX4 and PAX3.	mir-128	24-31	paired box 3	Gallus gallus	169-173
33080252-12	2021	Hence, the inhibitory effect on myogenesis observed after miR-128 knockdown was rescued by concomitant knockdown of PAX3.	mir-128	58-65	paired box 3	Gallus gallus	116-120
33379338-10	2020	Notably, miR-128 mediated SSR-enhanced HeLa and CaSki cells" adhesion and metastasis through suppressed ITGA5, ITGB5, sLex, CEACAM-6, MMP9, and MMP23 transcript levels.	mir-128	9-16	integrin subunit beta 5	Homo sapiens	111-116
33379338-10	2020	Notably, miR-128 mediated SSR-enhanced HeLa and CaSki cells" adhesion and metastasis through suppressed ITGA5, ITGB5, sLex, CEACAM-6, MMP9, and MMP23 transcript levels.	mir-128	9-16	CEA cell adhesion molecule 6	Homo sapiens	124-132
33379338-10	2020	Notably, miR-128 mediated SSR-enhanced HeLa and CaSki cells" adhesion and metastasis through suppressed ITGA5, ITGB5, sLex, CEACAM-6, MMP9, and MMP23 transcript levels.	mir-128	9-16	matrix metallopeptidase 9	Homo sapiens	134-138
33379338-10	2020	Notably, miR-128 mediated SSR-enhanced HeLa and CaSki cells" adhesion and metastasis through suppressed ITGA5, ITGB5, sLex, CEACAM-6, MMP9, and MMP23 transcript levels.	mir-128	9-16	matrix metallopeptidase 23B	Homo sapiens	144-149
33167678-6	2020	Direct binding of miR-128 to PVT1 was investigated using a probe pulldown assay.	mir-128	18-25	Pvt1 oncogene	Mus musculus	29-33
33167678-10	2020	Mechanistically, we found that PVT1 competes endogenously with miR-128 in the regulation of vascular endothelial growth factor C (VEGFC) expression, which is significantly associated with an unfavorable prognosis in lung cancer.	mir-128	63-70	vascular endothelial growth factor C	Homo sapiens	92-128
33167678-10	2020	Mechanistically, we found that PVT1 competes endogenously with miR-128 in the regulation of vascular endothelial growth factor C (VEGFC) expression, which is significantly associated with an unfavorable prognosis in lung cancer.	mir-128	63-70	vascular endothelial growth factor C	Homo sapiens	130-135
33167678-11	2020	We identified that copy number amplification significantly contributes to the high level of PVT1 transcripts in lung cancer, which promotes cell proliferation and metastatic behavior via modulating VEGFC expression by endogenous competition with miR-128.	mir-128	246-253	Pvt1 oncogene	Homo sapiens	92-96
33167678-11	2020	We identified that copy number amplification significantly contributes to the high level of PVT1 transcripts in lung cancer, which promotes cell proliferation and metastatic behavior via modulating VEGFC expression by endogenous competition with miR-128.	mir-128	246-253	vascular endothelial growth factor C	Homo sapiens	198-203
33116855-10	2020	HOXA10 as the target of miR-128/miR-142 was verified in OSCC cells.	mir-128	24-31	homeobox A10	Homo sapiens	0-6
33116617-13	2020	GOLM1 was a target of miR-128 and was negatively regulated by miR-128.	mir-128	22-29	golgi membrane protein 1	Mus musculus	0-5
33116617-13	2020	GOLM1 was a target of miR-128 and was negatively regulated by miR-128.	mir-128	62-69	golgi membrane protein 1	Mus musculus	0-5
33116855-13	2020	Conclusion: MiR-128/miR-142 suppressed OSCC tumorigenesis and metastasis by targeting HOXA10, providing a new promising therapeutic approach for OSCC patient diagnosis and treatment.	mir-128	12-19	homeobox A10	Homo sapiens	86-92
32993809-0	2020	MiR-128 suppresses metastatic capacity by targeting metadherin in breast cancer cells.	mir-128	0-7	metadherin	Homo sapiens	52-62
32993809-9	2020	Moreover, knockdown of MTDH in MDA-MB-231 cells obviously impaired the migration and invasion capabilities, whereas re-expression of MTDH abrogated the suppressive effect caused by miR-128.	mir-128	181-188	metadherin	Homo sapiens	133-137
32486927-8	2020	Moreover, overexpression of Rnd3 could reverse the activation of NF-kappaB caused by miR-128-3p agomir during liver I/R injury.	mir-128	85-92	Rho family GTPase 3	Mus musculus	28-32
32958647-0	2020	Astroglial FMRP deficiency cell-autonomously up-regulates miR-128 and disrupts developmental astroglial mGluR5 signaling.	mir-128	58-65	fragile X messenger ribonucleoprotein 1	Homo sapiens	11-15
32739259-0	2020	Obesity-related hypoxia via miR-128 decreases insulin-receptor expression in human and mouse adipose tissue promoting systemic insulin resistance.	mir-128	28-35	insulin receptor	Homo sapiens	46-62
32739259-0	2020	Obesity-related hypoxia via miR-128 decreases insulin-receptor expression in human and mouse adipose tissue promoting systemic insulin resistance.	mir-128	28-35	insulin	Homo sapiens	46-53
33277837-0	2020	IRF-1 mediated long non-coding RNA PVT1-214 promotes oxaliplatin resistance of colorectal cancer via miR-128 inhibition.	mir-128	101-108	interferon regulatory factor 1	Homo sapiens	0-5
32486927-8	2020	Moreover, overexpression of Rnd3 could reverse the activation of NF-kappaB caused by miR-128-3p agomir during liver I/R injury.	mir-128	85-92	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	65-74
32218208-4	2020	The expression of PAICS was regulated by miR-128 and transcriptionally activated by Myc in CRC cells.	mir-128	41-48	phosphoribosylaminoimidazole carboxylase and phosphoribosylaminoimidazolesuccinocarboxamide synthase	Homo sapiens	18-23
32678046-12	2020	CONCLUSION: The high expression of miR-128 can down-regulate the expression of CB1, promote the activation of NF-KB and p-JNK, increase the level of apoptosis and promote the occurrence of diabetic bladder disease.	mir-128	35-42	cannabinoid receptor 1	Rattus norvegicus	79-82
32678046-12	2020	CONCLUSION: The high expression of miR-128 can down-regulate the expression of CB1, promote the activation of NF-KB and p-JNK, increase the level of apoptosis and promote the occurrence of diabetic bladder disease.	mir-128	35-42	RELA proto-oncogene, NF-kB subunit	Rattus norvegicus	110-115
32678046-12	2020	CONCLUSION: The high expression of miR-128 can down-regulate the expression of CB1, promote the activation of NF-KB and p-JNK, increase the level of apoptosis and promote the occurrence of diabetic bladder disease.	mir-128	35-42	mitogen-activated protein kinase 8	Rattus norvegicus	122-125
32536914-6	2020	miR-128 was inversely correlated with cluster of differentiation 47 (CD47), which was positively related to zinc finger E-box-binding homeobox 1 (ZEB1) in PDAC.	mir-128	0-7	zinc finger E-box binding homeobox 1	Mus musculus	108-144
32536914-6	2020	miR-128 was inversely correlated with cluster of differentiation 47 (CD47), which was positively related to zinc finger E-box-binding homeobox 1 (ZEB1) in PDAC.	mir-128	0-7	zinc finger E-box binding homeobox 1	Mus musculus	146-150
32222058-0	2020	Mossy cell synaptic dysfunction causes memory imprecision via miR-128 inhibition of STIM2 in Alzheimer"s disease mouse model.	mir-128	62-69	stromal interaction molecule 2	Mus musculus	84-89
32222058-4	2020	Silencing miR-128 or disrupting miR-128 binding to STIM2 evokes STIM2 expression, restores synaptic function, and rescues memory imprecision in AD mice.	mir-128	10-17	stromal interaction molecule 2	Mus musculus	64-69
32222058-4	2020	Silencing miR-128 or disrupting miR-128 binding to STIM2 evokes STIM2 expression, restores synaptic function, and rescues memory imprecision in AD mice.	mir-128	32-39	stromal interaction molecule 2	Mus musculus	51-56
32222058-4	2020	Silencing miR-128 or disrupting miR-128 binding to STIM2 evokes STIM2 expression, restores synaptic function, and rescues memory imprecision in AD mice.	mir-128	32-39	stromal interaction molecule 2	Mus musculus	64-69
32377170-12	2020	MIR4435-2HG knockdown inhibited proliferation, invasion and migration but induced apoptosis of OC cells via miR-128-3p/CDK14 axis.	mir-128	108-115	MIR4435-2 host gene	Homo sapiens	0-11
32678046-2	2020	In this study we investigate the mechanism of miR-128 targeting CB1 expression to mediate the occurrence of diabetic bladder disease.	mir-128	46-53	cannabinoid receptor 1	Rattus norvegicus	64-67
32678046-9	2020	miR-128 could target the inhibition of CB1 expression, and high expression of CB1 could antagonize miR-128 against diabetic bladder.	mir-128	0-7	cannabinoid receptor 1	Rattus norvegicus	39-42
32678046-9	2020	miR-128 could target the inhibition of CB1 expression, and high expression of CB1 could antagonize miR-128 against diabetic bladder.	mir-128	99-106	cannabinoid receptor 1	Rattus norvegicus	78-81
32678046-10	2020	In the diabetic bladder, miR-128 can regulate the expression of NF-KB and p-JNK through CB1 and affect the level of apoptosis.	mir-128	25-32	RELA proto-oncogene, NF-kB subunit	Rattus norvegicus	64-69
32678046-10	2020	In the diabetic bladder, miR-128 can regulate the expression of NF-KB and p-JNK through CB1 and affect the level of apoptosis.	mir-128	25-32	mitogen-activated protein kinase 8	Rattus norvegicus	76-79
32678046-10	2020	In the diabetic bladder, miR-128 can regulate the expression of NF-KB and p-JNK through CB1 and affect the level of apoptosis.	mir-128	25-32	cannabinoid receptor 1	Rattus norvegicus	88-91
32678046-11	2020	miR-128 regulates NF-KB/p-JNK through CB1, thus affecting the occurrence of diabetic bladder disease.	mir-128	0-7	RELA proto-oncogene, NF-kB subunit	Rattus norvegicus	18-23
32678046-11	2020	miR-128 regulates NF-KB/p-JNK through CB1, thus affecting the occurrence of diabetic bladder disease.	mir-128	0-7	mitogen-activated protein kinase 8	Rattus norvegicus	26-29
32678046-11	2020	miR-128 regulates NF-KB/p-JNK through CB1, thus affecting the occurrence of diabetic bladder disease.	mir-128	0-7	cannabinoid receptor 1	Rattus norvegicus	38-41
32292498-11	2020	Results: The miR-128 level was found to be positively correlated with the increase in Nfatc1 level in mouse/human bone specimens and mouse primary BMMs.	mir-128	13-20	nuclear factor of activated T cells, cytoplasmic, calcineurin dependent 1	Mus musculus	86-92
32292498-17	2020	Conclusions: Our findings reveal a critical mechanism for osteoclastogenesis that is mediated by the miR-128/SIRT1/NF-kappaB signaling axis, highlighting a possible avenue for the further exploration of diagnostic and therapeutic target molecules in PMOP.	mir-128	101-108	sirtuin 1	Mus musculus	109-114
32292498-17	2020	Conclusions: Our findings reveal a critical mechanism for osteoclastogenesis that is mediated by the miR-128/SIRT1/NF-kappaB signaling axis, highlighting a possible avenue for the further exploration of diagnostic and therapeutic target molecules in PMOP.	mir-128	101-108	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	115-124
32127947-8	2020	The next study indicated that lnc-SNHG16 knockdown markedly increased miR-128 level which is down-regulated in CC.	mir-128	70-77	small nucleolar RNA host gene 16	Homo sapiens	34-40
32127947-9	2020	Moreover, miR-128 overexpression significantly inhibited proliferation, EMT process and tumor growth by directly targeting GSPT1 and WNT3A.	mir-128	10-17	G1 to S phase transition 1	Homo sapiens	123-128
32127947-9	2020	Moreover, miR-128 overexpression significantly inhibited proliferation, EMT process and tumor growth by directly targeting GSPT1 and WNT3A.	mir-128	10-17	Wnt family member 3A	Homo sapiens	133-138
32127947-10	2020	Finally, lnc-SNHG16 activates but miR-128 inactivates the WNT/beta-catenin pathways in CC cells.	mir-128	34-41	catenin beta 1	Homo sapiens	62-74
31037985-1	2019	MiR-128, one of the most enriched miRNAs in the human brain, has been reported to protect MCAO mice via inhibiting P38alpha MAPK.	mir-128	0-7	mitogen-activated protein kinase 14	Mus musculus	115-123
31341054-3	2019	We have further determined that miR-128 represses the expression of all three TNPO proteins (transportins TNPO1, TNPO2, and TNPO3).	mir-128	32-39	transportin 1	Homo sapiens	106-111
31341054-3	2019	We have further determined that miR-128 represses the expression of all three TNPO proteins (transportins TNPO1, TNPO2, and TNPO3).	mir-128	32-39	transportin 2	Homo sapiens	113-118
31341054-3	2019	We have further determined that miR-128 represses the expression of all three TNPO proteins (transportins TNPO1, TNPO2, and TNPO3).	mir-128	32-39	transportin 3	Homo sapiens	124-129
31341054-5	2019	Here, we report that type I interferon (IFN)-inducible miR-128 directly targets two sites in the TNPO3 mRNA, significantly downregulating TNPO3 mRNA and protein expression levels.	mir-128	55-62	transportin 3	Homo sapiens	97-102
31341054-5	2019	Here, we report that type I interferon (IFN)-inducible miR-128 directly targets two sites in the TNPO3 mRNA, significantly downregulating TNPO3 mRNA and protein expression levels.	mir-128	55-62	transportin 3	Homo sapiens	138-143
31341054-7	2019	Manipulation of miR-128 levels in HIV-1 target cell lines and in primary CD4+ T-cells by overexpression or knockdown showed that reduction of TNPO3 levels by miR-128 significantly affects HIV-1 replication but not murine leukemia virus (MLV) infection and that miR-128 modulation of HIV-1 replication is reduced with TNPO3-independent HIV-1 virus, suggesting that miR-128-indued TNPO3 repression contributes to the inhibition of HIV-1 replication.	mir-128	16-23	transportin 3	Homo sapiens	142-147
31341054-7	2019	Manipulation of miR-128 levels in HIV-1 target cell lines and in primary CD4+ T-cells by overexpression or knockdown showed that reduction of TNPO3 levels by miR-128 significantly affects HIV-1 replication but not murine leukemia virus (MLV) infection and that miR-128 modulation of HIV-1 replication is reduced with TNPO3-independent HIV-1 virus, suggesting that miR-128-indued TNPO3 repression contributes to the inhibition of HIV-1 replication.	mir-128	158-165	transportin 3	Homo sapiens	142-147
31341054-7	2019	Manipulation of miR-128 levels in HIV-1 target cell lines and in primary CD4+ T-cells by overexpression or knockdown showed that reduction of TNPO3 levels by miR-128 significantly affects HIV-1 replication but not murine leukemia virus (MLV) infection and that miR-128 modulation of HIV-1 replication is reduced with TNPO3-independent HIV-1 virus, suggesting that miR-128-indued TNPO3 repression contributes to the inhibition of HIV-1 replication.	mir-128	158-165	transportin 3	Homo sapiens	142-147
31341054-7	2019	Manipulation of miR-128 levels in HIV-1 target cell lines and in primary CD4+ T-cells by overexpression or knockdown showed that reduction of TNPO3 levels by miR-128 significantly affects HIV-1 replication but not murine leukemia virus (MLV) infection and that miR-128 modulation of HIV-1 replication is reduced with TNPO3-independent HIV-1 virus, suggesting that miR-128-indued TNPO3 repression contributes to the inhibition of HIV-1 replication.	mir-128	158-165	transportin 3	Homo sapiens	142-147
31341054-9	2019	Thus, we have established a novel role of miR-128 in antiviral defense in human cells, namely inhibiting HIV-1 replication by altering the cellular milieu through targeting factors that include TNPO3.IMPORTANCE HIV-1 is the causative agent of AIDS.	mir-128	42-49	transportin 3	Homo sapiens	194-199
31477582-0	2019	MiR-128 inhibits the osteogenic differentiation in osteoporosis by down-regulating SIRT6 expression.	mir-128	0-7	sirtuin 6	Mus musculus	83-88
31477582-4	2019	In addition, the results showed that overexpression of miR-128 significantly inhibited the mRNA and protein expression levels of osteocalcin (OC), alkaline phosphatase (ALP) and collagen I type-alpha1 (COL1A1) in C2C12 cells, while miR-128 inhibitor could reverse this effect.	mir-128	55-62	bone gamma-carboxyglutamate protein 2	Mus musculus	129-140
31477582-4	2019	In addition, the results showed that overexpression of miR-128 significantly inhibited the mRNA and protein expression levels of osteocalcin (OC), alkaline phosphatase (ALP) and collagen I type-alpha1 (COL1A1) in C2C12 cells, while miR-128 inhibitor could reverse this effect.	mir-128	55-62	collagen, type I, alpha 1	Mus musculus	202-208
31477582-4	2019	In addition, the results showed that overexpression of miR-128 significantly inhibited the mRNA and protein expression levels of osteocalcin (OC), alkaline phosphatase (ALP) and collagen I type-alpha1 (COL1A1) in C2C12 cells, while miR-128 inhibitor could reverse this effect.	mir-128	232-239	bone gamma-carboxyglutamate protein 2	Mus musculus	129-140
31477582-6	2019	The qRT-PCR and Western Blot results found that miR-128 significantly down-regulated the mRNA and protein expressions of SIRT6.	mir-128	48-55	sirtuin 6	Mus musculus	121-126
31477582-7	2019	Furthermore, silencing SIRT6 significantly inhibited the promoting effect of the miR-128 inhibitor on the expression of osteoblast markers.Conclusion: The above results confirmed that miR-128 inhibited osteoblast differentiation in OP by down-regulating SIRT6 expression, thus accelerating the development of OP.	mir-128	81-88	sirtuin 6	Mus musculus	23-28
31477582-7	2019	Furthermore, silencing SIRT6 significantly inhibited the promoting effect of the miR-128 inhibitor on the expression of osteoblast markers.Conclusion: The above results confirmed that miR-128 inhibited osteoblast differentiation in OP by down-regulating SIRT6 expression, thus accelerating the development of OP.	mir-128	81-88	sirtuin 6	Mus musculus	254-259
31477582-7	2019	Furthermore, silencing SIRT6 significantly inhibited the promoting effect of the miR-128 inhibitor on the expression of osteoblast markers.Conclusion: The above results confirmed that miR-128 inhibited osteoblast differentiation in OP by down-regulating SIRT6 expression, thus accelerating the development of OP.	mir-128	184-191	sirtuin 6	Mus musculus	23-28
31477582-7	2019	Furthermore, silencing SIRT6 significantly inhibited the promoting effect of the miR-128 inhibitor on the expression of osteoblast markers.Conclusion: The above results confirmed that miR-128 inhibited osteoblast differentiation in OP by down-regulating SIRT6 expression, thus accelerating the development of OP.	mir-128	184-191	sirtuin 6	Mus musculus	254-259
30701536-2	2019	This study was performed to clarify the effect of microRNA-128 (miR-128) on the CMEC injury in coronary heart disease (CHD) by binding to insulin receptor substrate 1 (IRS1).	mir-128	64-71	insulin receptor substrate 1	Rattus norvegicus	138-166
30701536-2	2019	This study was performed to clarify the effect of microRNA-128 (miR-128) on the CMEC injury in coronary heart disease (CHD) by binding to insulin receptor substrate 1 (IRS1).	mir-128	64-71	insulin receptor substrate 1	Rattus norvegicus	168-172
30701536-9	2019	Notably, miR-128 targeted and negatively regulated IRS1.	mir-128	9-16	insulin receptor substrate 1	Rattus norvegicus	51-55
30701536-11	2019	The experimental results demonstrated that miR-128 mimics and IRS1 siRNA in rat CMECs promoted viability, migration ability, tube formation, VEGF expression, SOD activity, while repressing cell apoptosis, MDA and ROS levels.	mir-128	43-50	vascular endothelial growth factor A	Rattus norvegicus	141-145
31271703-6	2019	The association between miR-128 and HOXB8 was confirmed using dual-luciferase reporter assay.	mir-128	24-31	homeobox B8	Homo sapiens	36-41
31271703-9	2019	miR-128 directly targeted HOXB8 in ovarian cancer cell lines.	mir-128	0-7	homeobox B8	Homo sapiens	26-31
31271703-10	2019	Knockdown of HOXB8 abolished the effects of miR-128 inhibitor on ovarian cancer cell proliferation and paclitaxel sensitivity.	mir-128	44-51	homeobox B8	Homo sapiens	13-18
31271703-11	2019	Summarily, miR-128 displayed a tumour suppressor role in ovarian cancer via targeting HOXB8.	mir-128	11-18	homeobox B8	Homo sapiens	86-91
31352238-7	2019	Additionally, PCAT-1 functioned as a sponge of miR-128 in GC cells.	mir-128	47-54	prostate cancer associated transcript 1	Homo sapiens	14-20
31352238-8	2019	Moreover, inhibition of miR-128 reversed the inductive effect of PCAT-1 knockdown on DDP sensitivity of GC cells.	mir-128	24-31	prostate cancer associated transcript 1	Homo sapiens	65-71
31173166-0	2019	miR-128 is upregulated in epilepsy and promotes apoptosis through the SIRT1 cascade.	mir-128	0-7	sirtuin 1	Rattus norvegicus	70-75
31173166-3	2019	Subsequently, results from an in vitro epilepsy model demonstrated that overexpression of miR-128 promoted nerve cell apoptosis, increased the protein expression of tumor protein p53, BCL2 associated X (Bax) and Cytochrome c, and enhanced caspase-3/9 activity, whereas it suppressed the protein expression of sirtuin 1 (SIRT1).	mir-128	90-97	Wistar clone pR53P1 p53 pseudogene	Rattus norvegicus	179-182
31173166-3	2019	Subsequently, results from an in vitro epilepsy model demonstrated that overexpression of miR-128 promoted nerve cell apoptosis, increased the protein expression of tumor protein p53, BCL2 associated X (Bax) and Cytochrome c, and enhanced caspase-3/9 activity, whereas it suppressed the protein expression of sirtuin 1 (SIRT1).	mir-128	90-97	BCL2 associated X, apoptosis regulator	Rattus norvegicus	184-201
31173166-3	2019	Subsequently, results from an in vitro epilepsy model demonstrated that overexpression of miR-128 promoted nerve cell apoptosis, increased the protein expression of tumor protein p53, BCL2 associated X (Bax) and Cytochrome c, and enhanced caspase-3/9 activity, whereas it suppressed the protein expression of sirtuin 1 (SIRT1).	mir-128	90-97	BCL2 associated X, apoptosis regulator	Rattus norvegicus	203-206
31173166-3	2019	Subsequently, results from an in vitro epilepsy model demonstrated that overexpression of miR-128 promoted nerve cell apoptosis, increased the protein expression of tumor protein p53, BCL2 associated X (Bax) and Cytochrome c, and enhanced caspase-3/9 activity, whereas it suppressed the protein expression of sirtuin 1 (SIRT1).	mir-128	90-97	caspase 3	Rattus norvegicus	239-250
31173166-3	2019	Subsequently, results from an in vitro epilepsy model demonstrated that overexpression of miR-128 promoted nerve cell apoptosis, increased the protein expression of tumor protein p53, BCL2 associated X (Bax) and Cytochrome c, and enhanced caspase-3/9 activity, whereas it suppressed the protein expression of sirtuin 1 (SIRT1).	mir-128	90-97	sirtuin 1	Rattus norvegicus	309-318
31173166-3	2019	Subsequently, results from an in vitro epilepsy model demonstrated that overexpression of miR-128 promoted nerve cell apoptosis, increased the protein expression of tumor protein p53, BCL2 associated X (Bax) and Cytochrome c, and enhanced caspase-3/9 activity, whereas it suppressed the protein expression of sirtuin 1 (SIRT1).	mir-128	90-97	sirtuin 1	Rattus norvegicus	320-325
31173166-5	2019	Furthermore, treatment with CAY10602, a SIRT1 agonist, reduced the effects of miR-128 on nerve cells in vitro.	mir-128	78-85	sirtuin 1	Rattus norvegicus	40-45
31173166-6	2019	Treatment with pifithrin-beta hydrobromide, a p53 inhibitor, was additionally able to mitigate the effects of miR-128 in vitro.	mir-128	110-117	Wistar clone pR53P1 p53 pseudogene	Rattus norvegicus	46-49
31173166-7	2019	In conclusion, the present findings indicated that anti-miR-128 may exert neuroprotective effects in epilepsy, through the SIRT1/p53/Bax/Cytochrome c/caspase signaling pathway.	mir-128	56-63	sirtuin 1	Rattus norvegicus	123-128
31173166-7	2019	In conclusion, the present findings indicated that anti-miR-128 may exert neuroprotective effects in epilepsy, through the SIRT1/p53/Bax/Cytochrome c/caspase signaling pathway.	mir-128	56-63	Wistar clone pR53P1 p53 pseudogene	Rattus norvegicus	129-132
31173166-7	2019	In conclusion, the present findings indicated that anti-miR-128 may exert neuroprotective effects in epilepsy, through the SIRT1/p53/Bax/Cytochrome c/caspase signaling pathway.	mir-128	56-63	BCL2 associated X, apoptosis regulator	Rattus norvegicus	133-136
31173166-7	2019	In conclusion, the present findings indicated that anti-miR-128 may exert neuroprotective effects in epilepsy, through the SIRT1/p53/Bax/Cytochrome c/caspase signaling pathway.	mir-128	56-63	caspase 9	Rattus norvegicus	150-157
31037985-9	2019	Moreover, an in vitro study demonstrated that miR-128 antagomir aggravated primary neuronal damage and exacerbated cell cycle reactivation induced by OGD/R stimulation; the underlying mechanism involved increasing cyclin A2, PTEN, and ERK expression and promoting phosphorylation of PTEN and ERK.	mir-128	46-53	cyclin A2	Homo sapiens	214-223
31037985-9	2019	Moreover, an in vitro study demonstrated that miR-128 antagomir aggravated primary neuronal damage and exacerbated cell cycle reactivation induced by OGD/R stimulation; the underlying mechanism involved increasing cyclin A2, PTEN, and ERK expression and promoting phosphorylation of PTEN and ERK.	mir-128	46-53	phosphatase and tensin homolog	Homo sapiens	225-229
31037985-9	2019	Moreover, an in vitro study demonstrated that miR-128 antagomir aggravated primary neuronal damage and exacerbated cell cycle reactivation induced by OGD/R stimulation; the underlying mechanism involved increasing cyclin A2, PTEN, and ERK expression and promoting phosphorylation of PTEN and ERK.	mir-128	46-53	mitogen-activated protein kinase 1	Homo sapiens	235-238
31037985-9	2019	Moreover, an in vitro study demonstrated that miR-128 antagomir aggravated primary neuronal damage and exacerbated cell cycle reactivation induced by OGD/R stimulation; the underlying mechanism involved increasing cyclin A2, PTEN, and ERK expression and promoting phosphorylation of PTEN and ERK.	mir-128	46-53	phosphatase and tensin homolog	Homo sapiens	283-287
31037985-9	2019	Moreover, an in vitro study demonstrated that miR-128 antagomir aggravated primary neuronal damage and exacerbated cell cycle reactivation induced by OGD/R stimulation; the underlying mechanism involved increasing cyclin A2, PTEN, and ERK expression and promoting phosphorylation of PTEN and ERK.	mir-128	46-53	mitogen-activated protein kinase 1	Homo sapiens	292-295
31173288-9	2019	Spearman"s correlation coefficient was adopted to evaluate the correlation between miR-128 and PVT1-214 levels.	mir-128	83-90	Pvt1 oncogene	Homo sapiens	95-99
31080058-5	2019	Moreover, miR-128, miR-9, and let-7 are functionally distinct; capable of specifying neurons for layer VI and layer V and layers IV, III, and II, respectively; and transiently altering their relative levels of expression can modulate stem-cell competence in a neurogenic-stage-specific manner to shift neuron production between earlier-born and later-born fates, partly by temporally regulating a neurogenesis program involving Hmga2.	mir-128	10-17	high mobility group AT-hook 2	Homo sapiens	428-433
30576236-5	2019	The relationship between MEG3/Girdin and miR-128 was determined and verified.	mir-128	41-48	maternally expressed 3	Mus musculus	25-29
30576236-5	2019	The relationship between MEG3/Girdin and miR-128 was determined and verified.	mir-128	41-48	coiled-coil domain containing 88A	Homo sapiens	30-36
30576236-8	2019	To further analyze the MEG3-mediated regulatory mechanism, miR-128 upregulation and inhibition were introduced into the HUVECs.	mir-128	59-66	maternally expressed 3	Mus musculus	23-27
30576236-10	2019	MEG3 downregulation was found to be capable of inhibiting Girdin but enhancing miR-128 expression.	mir-128	79-86	maternally expressed 3	Mus musculus	0-4
30576236-12	2019	These findings provide evidence supporting that MEG3 leads to miR-128 downregulation and Girdin upregulation, which promotes platelet phagocytosis, thus protecting VECs from senescence.	mir-128	62-69	maternally expressed 3	Mus musculus	48-52
30875657-9	2019	The potential relationship between miR-128 and WNT1 was analyzed.	mir-128	35-42	wingless-type MMTV integration site family, member 1	Mus musculus	47-51
30875657-11	2019	WNT1 was targeted and negatively regulated by miR-128.	mir-128	46-53	wingless-type MMTV integration site family, member 1	Mus musculus	0-4
30875657-13	2019	CONCLUSION: Collectively, miR-128 enhanced neuroprotective effect of DEX on HIBD neonatal mice by inhibiting WNT1.	mir-128	26-33	wingless-type MMTV integration site family, member 1	Mus musculus	109-113
31173288-13	2019	Additionally, PVT1-214 functions as a competing endogenous RNA (ceRNA) by binding to miR-128.	mir-128	85-92	Pvt1 oncogene	Homo sapiens	14-18
31173288-14	2019	Inhibition of miR-128 releases Tropomyosin receptor kinase C (TrkC) from the complementary binding complex, subsequently increasing the protein level of TrkC in GC cells.	mir-128	14-21	neurotrophic receptor tyrosine kinase 3	Homo sapiens	31-60
31173288-14	2019	Inhibition of miR-128 releases Tropomyosin receptor kinase C (TrkC) from the complementary binding complex, subsequently increasing the protein level of TrkC in GC cells.	mir-128	14-21	neurotrophic receptor tyrosine kinase 3	Homo sapiens	62-66
31173288-14	2019	Inhibition of miR-128 releases Tropomyosin receptor kinase C (TrkC) from the complementary binding complex, subsequently increasing the protein level of TrkC in GC cells.	mir-128	14-21	neurotrophic receptor tyrosine kinase 3	Homo sapiens	153-157
31173288-15	2019	CONCLUSIONS: PVT1-214-induced miR-128 repression regulates TrkC to further the progression of GC, indicating that this process will provide a promising therapeutic target in GC.	mir-128	30-37	Pvt1 oncogene	Homo sapiens	13-17
31173288-15	2019	CONCLUSIONS: PVT1-214-induced miR-128 repression regulates TrkC to further the progression of GC, indicating that this process will provide a promising therapeutic target in GC.	mir-128	30-37	neurotrophic receptor tyrosine kinase 3	Homo sapiens	59-63
30556928-9	2019	MAPK1 was highly expressed in ovarian cancer-resistant tissues and cells and could be regulated by linc00161 and miR-128.	mir-128	113-120	mitogen-activated protein kinase 1	Homo sapiens	0-5
30054858-9	2019	In contrast, reduction of miR-128, -15b, -195, -26b, -34b, -376b, and -381 by CSPGs was accompanied by increased EZR, KIF5A, DCX, GSK3B, and ROCK2 proteins.	mir-128	26-33	ezrin	Homo sapiens	113-116
30054858-9	2019	In contrast, reduction of miR-128, -15b, -195, -26b, -34b, -376b, and -381 by CSPGs was accompanied by increased EZR, KIF5A, DCX, GSK3B, and ROCK2 proteins.	mir-128	26-33	kinesin family member 5A	Homo sapiens	118-123
30867751-5	2019	In addition, it was identified that miR-128 could directly bind to the 3"-untranslated region of CIP2A.	mir-128	36-43	cellular inhibitor of PP2A	Homo sapiens	97-102
30265647-0	2019	MiR-128/SOX7 alleviates myocardial ischemia injury by regulating IL-33/sST2 in acute myocardial infarction.	mir-128	0-7	interleukin 33	Rattus norvegicus	65-70
30265647-6	2019	The relationship between SRY-related HMG-box (SOX7) and miR-128 was verified using luciferase reporter assay.	mir-128	56-63	SRY-box transcription factor 7	Rattus norvegicus	46-50
30265647-10	2019	Down-regulated miR-128 reversed the effects of IH on expression of SOX7, sST2 and cell apoptosis, while down-regulated sST2 abolished the effects of miR-128 inhibitor.	mir-128	15-22	SRY-box transcription factor 7	Rattus norvegicus	67-71
30265647-11	2019	In addition, overexpressed IL-33 abolished the effects of miR-128 inhibitor that induced by IH on the expression of SOX7 and cell apoptosis.	mir-128	58-65	interleukin 33	Rattus norvegicus	27-32
30265647-11	2019	In addition, overexpressed IL-33 abolished the effects of miR-128 inhibitor that induced by IH on the expression of SOX7 and cell apoptosis.	mir-128	58-65	SRY-box transcription factor 7	Rattus norvegicus	116-120
30265647-13	2019	The present study indicated that miR-128 modulated cell apoptosis by targeting SOX7, which was mediated by IL-33/sST2 signaling pathway.	mir-128	33-40	SRY-box transcription factor 7	Rattus norvegicus	79-83
30265647-13	2019	The present study indicated that miR-128 modulated cell apoptosis by targeting SOX7, which was mediated by IL-33/sST2 signaling pathway.	mir-128	33-40	interleukin 33	Rattus norvegicus	107-112
30132868-0	2019	LncRNA SNHG3 induces EMT and sorafenib resistance by modulating the miR-128/CD151 pathway in hepatocellular carcinoma.	mir-128	68-75	small nucleolar RNA host gene 3	Homo sapiens	7-12
30132868-0	2019	LncRNA SNHG3 induces EMT and sorafenib resistance by modulating the miR-128/CD151 pathway in hepatocellular carcinoma.	mir-128	68-75	CD151 molecule (Raph blood group)	Homo sapiens	76-81
30132868-5	2019	Moreover, SNHG3 overexpression induced HCC cells EMT via miR-128/CD151 cascade activation.	mir-128	57-64	small nucleolar RNA host gene 3	Homo sapiens	10-15
30551451-0	2019	MiR-128 suppresses the growth of thyroid carcinoma by negatively regulating SPHK1.	mir-128	0-7	sphingosine kinase 1	Homo sapiens	76-81
30535495-0	2019	Methylation-associated silencing of miR-128 promotes the development of esophageal cancer by targeting COX-2 in areas with a high incidence of esophageal cancer.	mir-128	36-43	prostaglandin-endoperoxide synthase 2	Mus musculus	103-108
30535495-7	2019	Furthermore, the expression of miR-128 was downregulated by methylation, and COX-2, a direct target of miR-128, was upregulated with the reduction in miR-128.	mir-128	103-110	prostaglandin-endoperoxide synthase 2	Mus musculus	77-82
30535495-7	2019	Furthermore, the expression of miR-128 was downregulated by methylation, and COX-2, a direct target of miR-128, was upregulated with the reduction in miR-128.	mir-128	103-110	prostaglandin-endoperoxide synthase 2	Mus musculus	77-82
30535495-8	2019	Upregulation of miR-128 inhibited the cell cycle, proliferation and metastasis, and the expression of COX-2, cyclin D1 and retinoblastoma protein (Rb).	mir-128	16-23	prostaglandin-endoperoxide synthase 2	Mus musculus	102-107
30535495-8	2019	Upregulation of miR-128 inhibited the cell cycle, proliferation and metastasis, and the expression of COX-2, cyclin D1 and retinoblastoma protein (Rb).	mir-128	16-23	cyclin D1	Mus musculus	109-118
30535495-9	2019	Furthermore, the relative expression level of miR-128 was negatively associated with COX-2 in ESCC tissues.	mir-128	46-53	prostaglandin-endoperoxide synthase 2	Mus musculus	85-90
30535495-10	2019	Collectively, these findings indicate that methylation-associated silencing of miR-128 promotes the development of esophageal cancer through upregulation of the expression of cyclin D1 and Rb by targeting COX-2 in ZD regions with a high incidence of esophageal cancer.	mir-128	79-86	cyclin D1	Mus musculus	175-184
30535495-10	2019	Collectively, these findings indicate that methylation-associated silencing of miR-128 promotes the development of esophageal cancer through upregulation of the expression of cyclin D1 and Rb by targeting COX-2 in ZD regions with a high incidence of esophageal cancer.	mir-128	79-86	prostaglandin-endoperoxide synthase 2	Mus musculus	205-210
30412727-10	2019	The interaction between miR-128 and PPARgamma were validated by bioinformatics analysis and luciferase reporter assay.	mir-128	24-31	peroxisome proliferator activated receptor gamma	Homo sapiens	36-45
30412727-13	2019	Moreover, PPARgamma was a target of miR-128.	mir-128	36-43	peroxisome proliferator activated receptor gamma	Mus musculus	10-19
30412727-14	2019	Additionally, miR-128 knockout or PPARgamma upregulation inhibited AD-like performances, amyloid plaque formation, Abeta generation, APP amyloidogenic processing and inflammatory responses in AD mice, while these effects of miR-128 knockout were abrogated by PPARgamma inhibitor.	mir-128	14-21	amyloid beta (A4) precursor protein	Mus musculus	115-120
30412727-14	2019	Additionally, miR-128 knockout or PPARgamma upregulation inhibited AD-like performances, amyloid plaque formation, Abeta generation, APP amyloidogenic processing and inflammatory responses in AD mice, while these effects of miR-128 knockout were abrogated by PPARgamma inhibitor.	mir-128	14-21	peroxisome proliferator activated receptor gamma	Mus musculus	259-268
30412727-15	2019	The results indicated MiR-128 knockout weakened AD-like performances, and reduced Abeta production and inflammatory responses by targeting PPARgamma in AD mice.	mir-128	22-29	amyloid beta (A4) precursor protein	Mus musculus	82-87
30412727-15	2019	The results indicated MiR-128 knockout weakened AD-like performances, and reduced Abeta production and inflammatory responses by targeting PPARgamma in AD mice.	mir-128	22-29	peroxisome proliferator activated receptor gamma	Mus musculus	139-148
30076714-0	2019	LncRNA PVT1 regulates VEGFC through inhibiting miR-128 in bladder cancer cells.	mir-128	47-54	Pvt1 oncogene	Homo sapiens	7-11
30076714-0	2019	LncRNA PVT1 regulates VEGFC through inhibiting miR-128 in bladder cancer cells.	mir-128	47-54	vascular endothelial growth factor C	Homo sapiens	22-27
30076714-7	2019	The results showed that the increased number of PVT1 transcripts interacted directly with miR-128 to decrease miR-128 binding to the VEGFC 3"-untranslated region.	mir-128	90-97	Pvt1 oncogene	Mus musculus	48-52
30076714-7	2019	The results showed that the increased number of PVT1 transcripts interacted directly with miR-128 to decrease miR-128 binding to the VEGFC 3"-untranslated region.	mir-128	90-97	vascular endothelial growth factor C	Homo sapiens	133-138
30076714-7	2019	The results showed that the increased number of PVT1 transcripts interacted directly with miR-128 to decrease miR-128 binding to the VEGFC 3"-untranslated region.	mir-128	110-117	Pvt1 oncogene	Mus musculus	48-52
30076714-7	2019	The results showed that the increased number of PVT1 transcripts interacted directly with miR-128 to decrease miR-128 binding to the VEGFC 3"-untranslated region.	mir-128	110-117	vascular endothelial growth factor C	Homo sapiens	133-138
30076714-8	2019	This effect suppressed VEGFC mRNA degradation by miR-128.	mir-128	49-56	vascular endothelial growth factor C	Homo sapiens	23-28
30076414-0	2019	Long noncoding RNA PVT1-214 promotes proliferation and invasion of colorectal cancer by stabilizing Lin28 and interacting with miR-128.	mir-128	127-134	Pvt1 oncogene	Homo sapiens	19-23
30551451-8	2019	Following, we characterized sphingosine kinase-1 (SPHK1) as a direct target of miR-128 that interacted with the 3"-untranslated region (UTR) of SPHK1, and the results were confirmed by using luciferase-reporter assay.	mir-128	79-86	sphingosine kinase 1	Homo sapiens	28-48
30551451-8	2019	Following, we characterized sphingosine kinase-1 (SPHK1) as a direct target of miR-128 that interacted with the 3"-untranslated region (UTR) of SPHK1, and the results were confirmed by using luciferase-reporter assay.	mir-128	79-86	sphingosine kinase 1	Homo sapiens	50-55
30551451-8	2019	Following, we characterized sphingosine kinase-1 (SPHK1) as a direct target of miR-128 that interacted with the 3"-untranslated region (UTR) of SPHK1, and the results were confirmed by using luciferase-reporter assay.	mir-128	79-86	sphingosine kinase 1	Homo sapiens	144-149
30551451-9	2019	We also observed that SPHK1 expression was decreased and negatively correlated with miR-128 expression in PTC and FTC tissues clinically.	mir-128	84-91	sphingosine kinase 1	Homo sapiens	22-27
30551451-10	2019	Importantly, ectopic expression of SPHK1 significantly abrogated the tumor-suppressive effect induced by miR-128, as supported by the reduced apoptosis, while the enhanced proliferation and metastasis.	mir-128	105-112	sphingosine kinase 1	Homo sapiens	35-40
30551451-11	2019	Finally, over-expressing miR-128 apparently reduced the tumor growth rate and tumor weight in vivo using xenograft tumor model, accompanied with a remarkable decrease of SPHK1.	mir-128	25-32	sphingosine kinase 1	Homo sapiens	170-175
30546426-2	2018	The mechanism of miR-128 on the regulation of Ribophorin-II (RPN2) in CRC cells was explored in the present study.	mir-128	17-24	ribophorin II	Homo sapiens	61-65
30407643-0	2018	NAIF1 suppresses osteosarcoma progression and is regulated by miR-128.	mir-128	62-69	nuclear apoptosis inducing factor 1	Homo sapiens	0-5
30407643-7	2018	The bioinformatic prediction and luciferase reporter assay revealed that miR-128 is a direct upstream regulator of NAIF1 and regulates NAIF1 expression by binding the 3"-UTR of NAIF1.	mir-128	73-80	nuclear apoptosis inducing factor 1	Homo sapiens	115-120
30407643-7	2018	The bioinformatic prediction and luciferase reporter assay revealed that miR-128 is a direct upstream regulator of NAIF1 and regulates NAIF1 expression by binding the 3"-UTR of NAIF1.	mir-128	73-80	nuclear apoptosis inducing factor 1	Homo sapiens	135-140
30407643-7	2018	The bioinformatic prediction and luciferase reporter assay revealed that miR-128 is a direct upstream regulator of NAIF1 and regulates NAIF1 expression by binding the 3"-UTR of NAIF1.	mir-128	73-80	nuclear apoptosis inducing factor 1	Homo sapiens	135-140
30407643-9	2018	Moreover, miR-128 expression levels were inversely correlated with that of NAIF1 in osteosarcoma tissues.	mir-128	10-17	nuclear apoptosis inducing factor 1	Homo sapiens	75-80
30407643-10	2018	Finally, functional assay showed that miR-128 significantly suppressed osteosarcoma progression partially mediated by inhibiting NAIF1 expression.	mir-128	38-45	nuclear apoptosis inducing factor 1	Homo sapiens	129-134
30407643-11	2018	These data indicate that the miR-128 and its target gene NAIF1 played important roles by regulating OS cell proliferation and migration phenotype.	mir-128	29-36	nuclear apoptosis inducing factor 1	Homo sapiens	57-62
30407643-15	2018	The data of this study indicated that the miR-128 and its target gene NAIF1 played important roles by regulating OS cell proliferation and migration phenotype.	mir-128	42-49	nuclear apoptosis inducing factor 1	Homo sapiens	70-75
30575919-0	2018	MiR-128 promotes the apoptosis of glioma cells via binding to NEK2.	mir-128	0-7	NIMA related kinase 2	Homo sapiens	62-66
30575919-1	2018	OBJECTIVE: The aim of this study was to explore whether miR-128 could promote the apoptosis of glioma cells by targeting NIMA related kinase-2 (NEK2), thus participating in the occurrence and progression of glioma.	mir-128	56-63	NIMA related kinase 2	Homo sapiens	121-142
30575919-1	2018	OBJECTIVE: The aim of this study was to explore whether miR-128 could promote the apoptosis of glioma cells by targeting NIMA related kinase-2 (NEK2), thus participating in the occurrence and progression of glioma.	mir-128	56-63	NIMA related kinase 2	Homo sapiens	144-148
30575919-6	2018	Meanwhile, rescue experiments were conducted to determine whether miR-128 could promote the apoptosis of glioma cells by targeting NEK2.	mir-128	66-73	NIMA related kinase 2	Homo sapiens	131-135
30575919-10	2018	Meanwhile, overexpression of miR-128 promoted the apoptosis of U87 cells, upregulated protein levels of cleaved Caspase-3 and BCL2-associated X (Bax), and downregulated B-cell lymphoma-2 (Bcl-2).	mir-128	29-36	caspase 3	Homo sapiens	112-121
30575919-10	2018	Meanwhile, overexpression of miR-128 promoted the apoptosis of U87 cells, upregulated protein levels of cleaved Caspase-3 and BCL2-associated X (Bax), and downregulated B-cell lymphoma-2 (Bcl-2).	mir-128	29-36	BCL2 associated X, apoptosis regulator	Homo sapiens	126-143
30575919-10	2018	Meanwhile, overexpression of miR-128 promoted the apoptosis of U87 cells, upregulated protein levels of cleaved Caspase-3 and BCL2-associated X (Bax), and downregulated B-cell lymphoma-2 (Bcl-2).	mir-128	29-36	BCL2 associated X, apoptosis regulator	Homo sapiens	145-148
30575919-10	2018	Meanwhile, overexpression of miR-128 promoted the apoptosis of U87 cells, upregulated protein levels of cleaved Caspase-3 and BCL2-associated X (Bax), and downregulated B-cell lymphoma-2 (Bcl-2).	mir-128	29-36	BCL2 apoptosis regulator	Homo sapiens	169-186
30575919-10	2018	Meanwhile, overexpression of miR-128 promoted the apoptosis of U87 cells, upregulated protein levels of cleaved Caspase-3 and BCL2-associated X (Bax), and downregulated B-cell lymphoma-2 (Bcl-2).	mir-128	29-36	BCL2 apoptosis regulator	Homo sapiens	188-193
30575919-11	2018	Dual-luciferase reporter gene assay indicated that miR-128 directly bound to NEK2.	mir-128	51-58	NIMA related kinase 2	Homo sapiens	77-81
30575919-12	2018	Further rescue experiments suggested that NEK2 overexpression partially reversed the effect of miR-128 on the apoptosis of glioma cells.	mir-128	95-102	NIMA related kinase 2	Homo sapiens	42-46
30575919-13	2018	CONCLUSIONS: Downregulated miR-128 inhibited the apoptosis of glioma cells via targeting NEK2.	mir-128	27-34	NIMA related kinase 2	Homo sapiens	89-93
30546426-7	2018	In the present study, it was identified that aberrant decreased miR-128 was negatively correlated with RPN2 in CRC tissues.	mir-128	64-71	ribophorin II	Homo sapiens	103-107
30546426-11	2018	Markedly decreased Akt phosphorylation and cyclin D1 levels and increased p53 levels were detected when cells were transfected with miR-128 mimics.	mir-128	132-139	AKT serine/threonine kinase 1	Homo sapiens	19-22
30546426-11	2018	Markedly decreased Akt phosphorylation and cyclin D1 levels and increased p53 levels were detected when cells were transfected with miR-128 mimics.	mir-128	132-139	cyclin D1	Homo sapiens	43-52
30546426-11	2018	Markedly decreased Akt phosphorylation and cyclin D1 levels and increased p53 levels were detected when cells were transfected with miR-128 mimics.	mir-128	132-139	tumor protein p53	Homo sapiens	74-77
30546426-13	2018	Subsequent to screening with miRNA target prediction databases, the specificity of miR-128-targeted RPN2 was validated by a luciferase reporter assay.	mir-128	83-90	ribophorin II	Homo sapiens	100-104
30546426-14	2018	In conclusion, the results suggested that miR-128 was a specific negative regulator of RPN2, which regulated colorectal cancer cell proliferation and migration by affecting the Akt-p53-cyclin pathway.	mir-128	42-49	ribophorin II	Homo sapiens	87-91
30546426-14	2018	In conclusion, the results suggested that miR-128 was a specific negative regulator of RPN2, which regulated colorectal cancer cell proliferation and migration by affecting the Akt-p53-cyclin pathway.	mir-128	42-49	AKT serine/threonine kinase 1	Homo sapiens	177-180
30546426-14	2018	In conclusion, the results suggested that miR-128 was a specific negative regulator of RPN2, which regulated colorectal cancer cell proliferation and migration by affecting the Akt-p53-cyclin pathway.	mir-128	42-49	tumor protein p53	Homo sapiens	181-184
30546426-14	2018	In conclusion, the results suggested that miR-128 was a specific negative regulator of RPN2, which regulated colorectal cancer cell proliferation and migration by affecting the Akt-p53-cyclin pathway.	mir-128	42-49	proliferating cell nuclear antigen	Homo sapiens	185-191
30177387-3	2018	In this study, we found that miR-128 is a critical tumor suppressor that is downregulated in GC patients and GC cells and that GAREM is a direct downstream target of miR-128.	mir-128	166-173	GRB2 associated regulator of MAPK1 subtype 1	Homo sapiens	127-132
30328325-0	2018	Inhibition of miR-128 Abates Abeta-Mediated Cytotoxicity by Targeting PPAR-gamma via NF-kappaB Inactivation in Primary Mouse Cortical Neurons and Neuro2a Cells.	mir-128	14-21	amyloid beta (A4) precursor protein	Mus musculus	29-34
30328325-0	2018	Inhibition of miR-128 Abates Abeta-Mediated Cytotoxicity by Targeting PPAR-gamma via NF-kappaB Inactivation in Primary Mouse Cortical Neurons and Neuro2a Cells.	mir-128	14-21	peroxisome proliferator activated receptor gamma	Mus musculus	70-80
30328325-0	2018	Inhibition of miR-128 Abates Abeta-Mediated Cytotoxicity by Targeting PPAR-gamma via NF-kappaB Inactivation in Primary Mouse Cortical Neurons and Neuro2a Cells.	mir-128	14-21	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	85-94
30328325-11	2018	RESULTS: MiR-128 expression was upregulated and PPAR-gamma expression was downregulated in plasma from AD patients and amyloid-beta (Abeta)-treated primary mouse cortical neurons (MCN) and Neuro2a (N2a) cells.	mir-128	9-16	amyloid beta precursor protein	Homo sapiens	119-131
30328325-11	2018	RESULTS: MiR-128 expression was upregulated and PPAR-gamma expression was downregulated in plasma from AD patients and amyloid-beta (Abeta)-treated primary mouse cortical neurons (MCN) and Neuro2a (N2a) cells.	mir-128	9-16	amyloid beta precursor protein	Homo sapiens	133-138
30328325-12	2018	Inhibition of miR-128 decreased Abeta-mediated cytotoxicity through inactivation of NF-kappaB in MCN and N2a cells.	mir-128	14-21	amyloid beta (A4) precursor protein	Mus musculus	32-37
30328325-12	2018	Inhibition of miR-128 decreased Abeta-mediated cytotoxicity through inactivation of NF-kappaB in MCN and N2a cells.	mir-128	14-21	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	84-93
30328325-13	2018	Moreover, PPAR-gamma was a target of miR-128.	mir-128	37-44	peroxisome proliferator activated receptor gamma	Mus musculus	10-20
30328325-15	2018	Furthermore, PPAR-gamma downregulation was able to abolish the effect of anti-miR-128 on cytotoxicity and NF-kappaB activity in MCN and N2a cells.	mir-128	78-85	peroxisome proliferator activated receptor gamma	Mus musculus	13-23
30328325-15	2018	Furthermore, PPAR-gamma downregulation was able to abolish the effect of anti-miR-128 on cytotoxicity and NF-kappaB activity in MCN and N2a cells.	mir-128	78-85	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	106-115
30328325-16	2018	CONCLUSION: MiR-128 inhibitor decreased Abeta-mediated cytotoxicity by upregulating PPAR-gamma via inactivation of NF-kappaB in MCN and N2a cells, providing a new potential target in AD treatment.	mir-128	12-19	amyloid beta (A4) precursor protein	Mus musculus	40-45
30328325-16	2018	CONCLUSION: MiR-128 inhibitor decreased Abeta-mediated cytotoxicity by upregulating PPAR-gamma via inactivation of NF-kappaB in MCN and N2a cells, providing a new potential target in AD treatment.	mir-128	12-19	peroxisome proliferator activated receptor gamma	Mus musculus	84-94
30328325-16	2018	CONCLUSION: MiR-128 inhibitor decreased Abeta-mediated cytotoxicity by upregulating PPAR-gamma via inactivation of NF-kappaB in MCN and N2a cells, providing a new potential target in AD treatment.	mir-128	12-19	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	115-124
30206206-7	2018	In vitro, miR-128 signaling hindered Atg12 expression, LC3-II conversion, and autophagic puncta formation through targeting the 3"-untranslated region of Atg12.	mir-128	10-17	autophagy related 12	Homo sapiens	37-42
30206206-7	2018	In vitro, miR-128 signaling hindered Atg12 expression, LC3-II conversion, and autophagic puncta formation through targeting the 3"-untranslated region of Atg12.	mir-128	10-17	autophagy related 12	Homo sapiens	154-159
30203508-0	2018	NEAT1 contributes to ox-LDL-induced inflammation and oxidative stress in macrophages through inhibiting miR-128.	mir-128	104-111	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	0-5
30203508-12	2018	Interestingly, miR-128 mimics was able to reverse AS-correlated events induced by overexpression of NEAT1.	mir-128	15-22	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	100-105
30203508-13	2018	By using bioinformatics analysis, miR-128 was predicted as a target of NEAT1 and the correlation between them was validated in our study.	mir-128	34-41	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	71-76
30203508-14	2018	Taken these together, it was implied that NEAT1 participated in ox-LDL-induced inflammation and oxidative stress in AS development through sponging miR-128.	mir-128	148-155	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	42-47
29777777-6	2018	As verified using Immunoblotting assays, miR-128 significantly regulated the protein levels of VEGFC/VEGFR3, whereas miR-3916 only slightly modulated VEGFC and VEGFR3 proteins.	mir-128	41-48	vascular endothelial growth factor C	Homo sapiens	95-100
29777777-6	2018	As verified using Immunoblotting assays, miR-128 significantly regulated the protein levels of VEGFC/VEGFR3, whereas miR-3916 only slightly modulated VEGFC and VEGFR3 proteins.	mir-128	41-48	fms related receptor tyrosine kinase 4	Homo sapiens	101-107
29777777-7	2018	Contrary to VEGFC, miR-128 overexpression remarkably suppressed LEC proliferation, Ca2+ release and ERK1/2-Akt signaling; moreover, the effect of VEGFC could be partially attenuated by miR-128.	mir-128	19-26	mitogen-activated protein kinase 3	Homo sapiens	100-106
29777777-7	2018	Contrary to VEGFC, miR-128 overexpression remarkably suppressed LEC proliferation, Ca2+ release and ERK1/2-Akt signaling; moreover, the effect of VEGFC could be partially attenuated by miR-128.	mir-128	19-26	AKT serine/threonine kinase 1	Homo sapiens	107-110
29777777-7	2018	Contrary to VEGFC, miR-128 overexpression remarkably suppressed LEC proliferation, Ca2+ release and ERK1/2-Akt signaling; moreover, the effect of VEGFC could be partially attenuated by miR-128.	mir-128	19-26	vascular endothelial growth factor C	Homo sapiens	146-151
29777777-7	2018	Contrary to VEGFC, miR-128 overexpression remarkably suppressed LEC proliferation, Ca2+ release and ERK1/2-Akt signaling; moreover, the effect of VEGFC could be partially attenuated by miR-128.	mir-128	185-192	vascular endothelial growth factor C	Homo sapiens	146-151
29777777-8	2018	In summary, miR-128 interacts with the 3"-UTR of VEGFC and VEGFR3 to inhibit their expression, thus suppressing LEC proliferation through Ca2+ and ERK1/2-Akt signaling.	mir-128	12-19	vascular endothelial growth factor C	Homo sapiens	49-54
29777777-8	2018	In summary, miR-128 interacts with the 3"-UTR of VEGFC and VEGFR3 to inhibit their expression, thus suppressing LEC proliferation through Ca2+ and ERK1/2-Akt signaling.	mir-128	12-19	fms related receptor tyrosine kinase 4	Homo sapiens	59-65
29777777-8	2018	In summary, miR-128 interacts with the 3"-UTR of VEGFC and VEGFR3 to inhibit their expression, thus suppressing LEC proliferation through Ca2+ and ERK1/2-Akt signaling.	mir-128	12-19	mitogen-activated protein kinase 3	Homo sapiens	147-153
29777777-8	2018	In summary, miR-128 interacts with the 3"-UTR of VEGFC and VEGFR3 to inhibit their expression, thus suppressing LEC proliferation through Ca2+ and ERK1/2-Akt signaling.	mir-128	12-19	AKT serine/threonine kinase 1	Homo sapiens	154-157
30025750-0	2018	miR-128 targets the CC chemokine ligand 18 gene (CCL18) in cutaneous malignant melanoma progression.	mir-128	0-7	C-C motif chemokine ligand 18	Homo sapiens	20-42
30025750-0	2018	miR-128 targets the CC chemokine ligand 18 gene (CCL18) in cutaneous malignant melanoma progression.	mir-128	0-7	C-C motif chemokine ligand 18	Homo sapiens	49-54
30025750-6	2018	The changes in expression of CCL18 after miR-128 mimic transfection of A375 melanoma cells were determined by both qRT-PCR and Western-bloting.	mir-128	41-48	C-C motif chemokine ligand 18	Homo sapiens	29-34
30025750-12	2018	RESULTS: The expression of miR-128 had negative relevance with CCL18 in CMM.	mir-128	27-34	C-C motif chemokine ligand 18	Homo sapiens	63-68
30025750-14	2018	Transfected miR-128 mimic significantly reduced CCL18 expression and this impairment of CCL18 gene promoted apoptosis, inhibited migration and colony formation of A375 melanoma cells.	mir-128	12-19	C-C motif chemokine ligand 18	Homo sapiens	48-53
30025750-14	2018	Transfected miR-128 mimic significantly reduced CCL18 expression and this impairment of CCL18 gene promoted apoptosis, inhibited migration and colony formation of A375 melanoma cells.	mir-128	12-19	C-C motif chemokine ligand 18	Homo sapiens	88-93
30025750-17	2018	Overexpression of miR-128 inhibits the oncogenic effect of CCL18.	mir-128	18-25	C-C motif chemokine ligand 18	Homo sapiens	59-64
30177387-4	2018	Overexpression of miR-128 in HGC-27 and MKN-45 cells resulted in suppressed cell growth and promoted cell apoptosis through a GAREM-dependent mechanism.	mir-128	18-25	GRB2 associated regulator of MAPK1 subtype 1	Homo sapiens	126-131
30177387-5	2018	Moreover, the precise mechanisms underlying the antitumor effect of miR-128 in GC are at least partially due to suppressing activation of the MAPK signaling pathway, induced by suppressing GAREM expression.	mir-128	68-75	GRB2 associated regulator of MAPK1 subtype 1	Homo sapiens	189-194
30121007-9	2018	Furthermore, we found that tumor suppressor miR-128 negatively regulated PAICS expression by binding to its 3"-untranslated region.	mir-128	44-51	phosphoribosylaminoimidazole carboxylase and phosphoribosylaminoimidazolesuccinocarboxamide synthase	Gallus gallus	73-78
30223934-6	2018	Besides, the association between miR-128-3p and ERK and PI3K/AKT pathways was further explored.	mir-128	33-40	mitogen-activated protein kinase 1	Homo sapiens	48-51
30223934-6	2018	Besides, the association between miR-128-3p and ERK and PI3K/AKT pathways was further explored.	mir-128	33-40	AKT serine/threonine kinase 1	Homo sapiens	61-64
29524580-4	2018	However, the inhibitory effects of miR-128 mimics on the invasion and proliferation of glioma cells were reversed by overexpression of cyclooxygenase-2 (COX-2).	mir-128	35-42	prostaglandin-endoperoxide synthase 2	Homo sapiens	135-151
29524580-4	2018	However, the inhibitory effects of miR-128 mimics on the invasion and proliferation of glioma cells were reversed by overexpression of cyclooxygenase-2 (COX-2).	mir-128	35-42	prostaglandin-endoperoxide synthase 2	Homo sapiens	153-158
29524580-5	2018	Our data showed that COX-2 was a candidate target of miR-128.	mir-128	53-60	prostaglandin-endoperoxide synthase 2	Homo sapiens	21-26
29524580-6	2018	Luciferase activity of 3"-UTR of COX-2 was reduced in the presence of miR-128.	mir-128	70-77	prostaglandin-endoperoxide synthase 2	Homo sapiens	33-38
29524580-7	2018	Additionally, miR-128 obviously decreased COX-2 mRNA stability determined by real time PCR.	mir-128	14-21	prostaglandin-endoperoxide synthase 2	Homo sapiens	42-47
29524580-8	2018	Contrarily, we found that miR-128 inhibitor significantly increased the COX-2 mRNA expression, and elevated the protein expression of MMP9 and ki67, and promoted the proliferation of glioma cells.	mir-128	26-33	prostaglandin-endoperoxide synthase 2	Homo sapiens	72-77
29524580-8	2018	Contrarily, we found that miR-128 inhibitor significantly increased the COX-2 mRNA expression, and elevated the protein expression of MMP9 and ki67, and promoted the proliferation of glioma cells.	mir-128	26-33	matrix metallopeptidase 9	Homo sapiens	134-138
29524580-10	2018	All of these results supported that miR-128 was a direct regulator of COX-2.	mir-128	36-43	prostaglandin-endoperoxide synthase 2	Homo sapiens	70-75
29524580-11	2018	Further studies proved that COX-2 was elevated in glioma tissues and its expression was negatively correlated with the levels of miR-128.	mir-128	129-136	prostaglandin-endoperoxide synthase 2	Homo sapiens	28-33
29497903-11	2018	The levels of both cleaved caspase-3 and cleaved caspase-9 were decreased in hippocampus exposed to the miR-128 mimic, whereas they were markedly increased in miR-128 inhibitor-treated hippocampus.	mir-128	104-111	caspase 3	Rattus norvegicus	27-36
29805606-5	2018	First, the expressions levels of miR-128 and its target gene, SAM and SH3 domain-containing 1 (SASH1), were measured in tissues from patients with osteosarcoma, and their correlation with osteosarcoma in terms of the pathological level were examined.	mir-128	33-40	SAM and SH3 domain containing 1	Homo sapiens	62-93
29805606-5	2018	First, the expressions levels of miR-128 and its target gene, SAM and SH3 domain-containing 1 (SASH1), were measured in tissues from patients with osteosarcoma, and their correlation with osteosarcoma in terms of the pathological level were examined.	mir-128	33-40	SAM and SH3 domain containing 1	Homo sapiens	95-100
29805606-7	2018	Subsequently, the association between SASH1 and miR-128 was evaluated using a dual luciferase gene reporter assay.	mir-128	48-55	SAM and SH3 domain containing 1	Homo sapiens	38-43
29805606-12	2018	Additionally, the expression of B-cell lymphoma 2 (Bcl-2) was downregulated in the miR-128-inhibited group, compared with that in the control group, whereas the expression levels of SASH1, Bcl-2-associated X protein and caspase-3 were upregulated in the group with miR-128 inhibition (P&lt;0.05).	mir-128	83-90	BCL2 apoptosis regulator	Homo sapiens	32-49
29805606-12	2018	Additionally, the expression of B-cell lymphoma 2 (Bcl-2) was downregulated in the miR-128-inhibited group, compared with that in the control group, whereas the expression levels of SASH1, Bcl-2-associated X protein and caspase-3 were upregulated in the group with miR-128 inhibition (P&lt;0.05).	mir-128	83-90	BCL2 apoptosis regulator	Homo sapiens	51-56
29805606-12	2018	Additionally, the expression of B-cell lymphoma 2 (Bcl-2) was downregulated in the miR-128-inhibited group, compared with that in the control group, whereas the expression levels of SASH1, Bcl-2-associated X protein and caspase-3 were upregulated in the group with miR-128 inhibition (P&lt;0.05).	mir-128	83-90	SAM and SH3 domain containing 1	Homo sapiens	182-187
29091297-5	2018	JAG1 is targeted and negatively regulated by miR-128.	mir-128	45-52	jagged canonical Notch ligand 1	Homo sapiens	0-4
29091297-7	2018	Compared with the resistance group, the anti-miR-128 group showed decreasedBax expression along with a lowered cell inhibition rate and apoptosis rate, but increased JAG1 and Bcl-2 expression with reduced cells arrested in the S phase; while the miR-128 mimic group showed an opposite trend; the si-JAG1 group showed decreased Bcl-2 expression and reduced cells in the S phase.	mir-128	45-52	jagged canonical Notch ligand 1	Homo sapiens	166-170
29091297-7	2018	Compared with the resistance group, the anti-miR-128 group showed decreasedBax expression along with a lowered cell inhibition rate and apoptosis rate, but increased JAG1 and Bcl-2 expression with reduced cells arrested in the S phase; while the miR-128 mimic group showed an opposite trend; the si-JAG1 group showed decreased Bcl-2 expression and reduced cells in the S phase.	mir-128	45-52	BCL2 apoptosis regulator	Homo sapiens	175-180
29091297-7	2018	Compared with the resistance group, the anti-miR-128 group showed decreasedBax expression along with a lowered cell inhibition rate and apoptosis rate, but increased JAG1 and Bcl-2 expression with reduced cells arrested in the S phase; while the miR-128 mimic group showed an opposite trend; the si-JAG1 group showed decreased Bcl-2 expression and reduced cells in the S phase.	mir-128	45-52	jagged canonical Notch ligand 1	Homo sapiens	299-303
29091297-7	2018	Compared with the resistance group, the anti-miR-128 group showed decreasedBax expression along with a lowered cell inhibition rate and apoptosis rate, but increased JAG1 and Bcl-2 expression with reduced cells arrested in the S phase; while the miR-128 mimic group showed an opposite trend; the si-JAG1 group showed decreased Bcl-2 expression and reduced cells in the S phase.	mir-128	45-52	BCL2 apoptosis regulator	Homo sapiens	327-332
29600803-0	2018	Publisher Correction: Simultaneous overactivation of Wnt/beta-catenin and TGFbeta signalling by miR-128-3p confers chemoresistance-associated metastasis in NSCLC.	mir-128	96-103	transforming growth factor beta 1	Homo sapiens	74-81
29805525-0	2018	miR-128 induces pancreas cancer cell apoptosis by targeting MDM4.	mir-128	0-7	MDM4 regulator of p53	Homo sapiens	60-64
29805606-12	2018	Additionally, the expression of B-cell lymphoma 2 (Bcl-2) was downregulated in the miR-128-inhibited group, compared with that in the control group, whereas the expression levels of SASH1, Bcl-2-associated X protein and caspase-3 were upregulated in the group with miR-128 inhibition (P&lt;0.05).	mir-128	83-90	BCL2 apoptosis regulator	Homo sapiens	189-194
29805606-12	2018	Additionally, the expression of B-cell lymphoma 2 (Bcl-2) was downregulated in the miR-128-inhibited group, compared with that in the control group, whereas the expression levels of SASH1, Bcl-2-associated X protein and caspase-3 were upregulated in the group with miR-128 inhibition (P&lt;0.05).	mir-128	83-90	caspase 3	Homo sapiens	220-229
29805606-12	2018	Additionally, the expression of B-cell lymphoma 2 (Bcl-2) was downregulated in the miR-128-inhibited group, compared with that in the control group, whereas the expression levels of SASH1, Bcl-2-associated X protein and caspase-3 were upregulated in the group with miR-128 inhibition (P&lt;0.05).	mir-128	265-272	BCL2 apoptosis regulator	Homo sapiens	32-49
29805606-12	2018	Additionally, the expression of B-cell lymphoma 2 (Bcl-2) was downregulated in the miR-128-inhibited group, compared with that in the control group, whereas the expression levels of SASH1, Bcl-2-associated X protein and caspase-3 were upregulated in the group with miR-128 inhibition (P&lt;0.05).	mir-128	265-272	BCL2 apoptosis regulator	Homo sapiens	51-56
29805606-13	2018	SASH1 was confirmed as a direct target of miR-128 using a dual luciferase gene reporter assay.	mir-128	42-49	SAM and SH3 domain containing 1	Homo sapiens	0-5
29805606-15	2018	The results of the present study suggested that miR-128 may regulate the tumorigenesis and evolution of osteosarcoma through targeting SASH1.	mir-128	48-55	SAM and SH3 domain containing 1	Homo sapiens	135-140
29600803-0	2018	Publisher Correction: Simultaneous overactivation of Wnt/beta-catenin and TGFbeta signalling by miR-128-3p confers chemoresistance-associated metastasis in NSCLC.	mir-128	96-103	catenin beta 1	Homo sapiens	57-69
29581509-5	2018	ARPP21 antagonizes miR-128 activity by co-regulating a subset of miR-128 target mRNAs enriched for neurodevelopmental functions.	mir-128	19-26	cAMP regulated phosphoprotein 21	Homo sapiens	0-6
29497903-11	2018	The levels of both cleaved caspase-3 and cleaved caspase-9 were decreased in hippocampus exposed to the miR-128 mimic, whereas they were markedly increased in miR-128 inhibitor-treated hippocampus.	mir-128	104-111	caspase 9	Rattus norvegicus	49-58
30537735-12	2018	CONCLUSION: Evidence from experimental models revealed that miR-128 might reduce apoptosis of DA neurons while increasing the expression of EAAT4 which might be related to the downregulation of AXIN1.	mir-128	60-67	solute carrier family 1 (high affinity aspartate/glutamate transporter), member 6	Mus musculus	140-145
30257253-0	2018	miR-128 Targets the SIRT1/ROS/DR5 Pathway to Sensitize Colorectal Cancer to TRAIL-Induced Apoptosis.	mir-128	0-7	sirtuin 1	Homo sapiens	20-25
30257253-0	2018	miR-128 Targets the SIRT1/ROS/DR5 Pathway to Sensitize Colorectal Cancer to TRAIL-Induced Apoptosis.	mir-128	0-7	TNF receptor superfamily member 10b	Homo sapiens	30-33
30257253-0	2018	miR-128 Targets the SIRT1/ROS/DR5 Pathway to Sensitize Colorectal Cancer to TRAIL-Induced Apoptosis.	mir-128	0-7	TNF superfamily member 10	Homo sapiens	76-81
30257253-5	2018	MTT assays were used to evaluate the effect of miR-128 on TRAIL-induced cytotoxicity against CRC cell lines.	mir-128	47-54	TNF superfamily member 10	Homo sapiens	58-63
30257253-7	2018	Western blot, flow cytometry, and luciferase reporter assays were performed to evaluate the potential mechanism and pathway of miR-128-promoted apoptosis in TRAIL-treated CRC cells.	mir-128	127-134	TNF superfamily member 10	Homo sapiens	157-162
30257253-9	2018	The enforced expression of miR-128 sensitized CRC cells to TRAIL-induced cytotoxicity by inducing apoptosis.	mir-128	27-34	TNF superfamily member 10	Homo sapiens	59-64
30257253-10	2018	Mechanistically, bioinformatics, western blot analysis, and luciferase reporter assays showed that miR-128 directly targeted sirtuin 1 (SIRT1) in CRC cells.	mir-128	99-106	sirtuin 1	Homo sapiens	125-134
30257253-10	2018	Mechanistically, bioinformatics, western blot analysis, and luciferase reporter assays showed that miR-128 directly targeted sirtuin 1 (SIRT1) in CRC cells.	mir-128	99-106	sirtuin 1	Homo sapiens	136-141
30257253-11	2018	miR-128 overexpression suppressed SIRT1 expression, which promoted the production of ROS in TRAIL-treated CRC cells.	mir-128	0-7	sirtuin 1	Homo sapiens	34-39
30257253-11	2018	miR-128 overexpression suppressed SIRT1 expression, which promoted the production of ROS in TRAIL-treated CRC cells.	mir-128	0-7	TNF superfamily member 10	Homo sapiens	92-97
30537735-4	2018	The targeting relationship between miR-128 and AXIN1 was verified via a bioinformatics prediction and dual-luciferase reporter gene assay.	mir-128	35-42	axin 1	Mus musculus	47-52
30537735-5	2018	After separation, DA neurons were subjected to a series of inhibitors, activators and shRNAs to validate the mechanisms of miR-128 in controlling of AXIN1 in PD.	mir-128	123-130	axin 1	Mus musculus	149-154
30537735-10	2018	AXIN1 was targeted and negatively mediated by miR-128.	mir-128	46-53	axin 1	Mus musculus	0-5
30537735-11	2018	In the DA neurons, upregulated miR-128 expression or sh-AXIN1 increased the positive expression rate of EAAT4 together with mRNA and protein levels, but decreased the mRNA and protein levels of AXIN1, apoptosis rate along with the positive expression rate of AXIN1; however, the opposite trend was found in response to transfection with miR-128 inhibitors.	mir-128	31-38	solute carrier family 1 (high affinity aspartate/glutamate transporter), member 6	Mus musculus	104-109
30537735-11	2018	In the DA neurons, upregulated miR-128 expression or sh-AXIN1 increased the positive expression rate of EAAT4 together with mRNA and protein levels, but decreased the mRNA and protein levels of AXIN1, apoptosis rate along with the positive expression rate of AXIN1; however, the opposite trend was found in response to transfection with miR-128 inhibitors.	mir-128	31-38	axin 1	Mus musculus	194-199
30537735-11	2018	In the DA neurons, upregulated miR-128 expression or sh-AXIN1 increased the positive expression rate of EAAT4 together with mRNA and protein levels, but decreased the mRNA and protein levels of AXIN1, apoptosis rate along with the positive expression rate of AXIN1; however, the opposite trend was found in response to transfection with miR-128 inhibitors.	mir-128	31-38	axin 1	Mus musculus	194-199
29552332-0	2018	Correction: Posttranscriptional regulation of Galectin-3 by miR-128 contributes to colorectal cancer progression.	mir-128	60-67	galectin 3	Homo sapiens	46-56
30537735-12	2018	CONCLUSION: Evidence from experimental models revealed that miR-128 might reduce apoptosis of DA neurons while increasing the expression of EAAT4 which might be related to the downregulation of AXIN1.	mir-128	60-67	axin 1	Mus musculus	194-199
28928797-5	2017	A luciferase assay was applied to assess the binding of miR-128 to c-Met mRNA.	mir-128	56-63	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	67-72
28974576-0	2017	MicroRNA miR-128 represses LINE-1 (L1) retrotransposition by down-regulating the nuclear import factor TNPO1.	mir-128	9-16	L1 cell adhesion molecule	Homo sapiens	27-37
28974576-0	2017	MicroRNA miR-128 represses LINE-1 (L1) retrotransposition by down-regulating the nuclear import factor TNPO1.	mir-128	9-16	transportin 1	Homo sapiens	103-108
28974576-7	2017	We found that miR-128 targets the 3" UTR of nuclear import factor transportin 1 (TNPO1) mRNA.	mir-128	14-21	transportin 1	Homo sapiens	66-79
28974576-7	2017	We found that miR-128 targets the 3" UTR of nuclear import factor transportin 1 (TNPO1) mRNA.	mir-128	14-21	transportin 1	Homo sapiens	81-86
28974576-8	2017	Manipulation of miR-128 and TNPO1 levels demonstrated that induction or depletion of TNPO1 affects L1 retrotransposition and nuclear import of an L1-ribonucleoprotein complex (using L1-encoded ORF1p as a proxy for L1-ribonucleoprotein complexes).	mir-128	16-23	transportin 1	Homo sapiens	85-90
28974576-9	2017	Moreover, TNPO1 overexpression partially reversed the repressive effect of miR-128 on L1 retrotransposition.	mir-128	75-82	transportin 1	Homo sapiens	10-15
28928797-8	2017	Similarly, miR-128 expression in primary cardiomyocytes cultured under deprivation of oxygen and glucose increased with the culture time and reached a peak at 12 h. c-Met expression decreased significantly (P&lt;0.05) and the ratio of apoptotic cells increased significantly (P&lt;0.05), following transfection of miR-128 mimics.	mir-128	314-321	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	165-170
28928797-10	2017	The dual luciferase assay indicated that fluorescence intensity decreased significantly in miR-128 mimics and wild type c-Met group (P&lt;0.05), indicating that miR-128 can directly target c-Met.	mir-128	91-98	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	189-194
28928797-10	2017	The dual luciferase assay indicated that fluorescence intensity decreased significantly in miR-128 mimics and wild type c-Met group (P&lt;0.05), indicating that miR-128 can directly target c-Met.	mir-128	161-168	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	120-125
28928797-10	2017	The dual luciferase assay indicated that fluorescence intensity decreased significantly in miR-128 mimics and wild type c-Met group (P&lt;0.05), indicating that miR-128 can directly target c-Met.	mir-128	161-168	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	189-194
28928797-11	2017	Therefore, the results of the current study suggest that miR-128 may promote myocardial cell injury by regulating c-Met expression.	mir-128	57-64	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	114-119
28497421-10	2017	The decrease in miR-128 was sufficient to induce the loss of nephrin and the impairment of filtration barrier function, while the increase of miR-21 exacerbated the process.	mir-128	16-23	NPHS1 adhesion molecule, nephrin	Homo sapiens	61-68
28497421-11	2017	Snail and phosphatase and tensin homolog (PTEN) were identified as the targets of miR-128 and miR-21.	mir-128	82-89	snail family transcriptional repressor 1	Homo sapiens	0-5
28497421-11	2017	Snail and phosphatase and tensin homolog (PTEN) were identified as the targets of miR-128 and miR-21.	mir-128	82-89	phosphatase and tensin homolog	Homo sapiens	42-46
28497421-12	2017	Decreased miR-128 induced Snail expression, and the increased miR-21 stabilized Snail by regulating the PTEN/Akt/GSK3beta pathway.	mir-128	10-17	snail family transcriptional repressor 1	Homo sapiens	26-31
28497421-13	2017	Supplementation of miR-128 and inhibition of miR-21 suppressed Snail expression and prevented the podocyte injury induced by LPS.	mir-128	19-26	snail family transcriptional repressor 1	Homo sapiens	63-68
29067466-0	2017	miR-128 enhances dendritic cell-mediated anti-tumor immunity via targeting of p38.	mir-128	0-7	mitogen-activated protein kinase 14	Mus musculus	78-81
29067466-8	2017	Additionally, miR-128 inhibited the protein expression of p38 in DCs in a dose-dependent manner, however no significant effect on the p38 mRNA level was observed.	mir-128	14-21	mitogen-activated protein kinase 14	Mus musculus	58-61
29067466-9	2017	Furthermore, miR-128 mimic or p38 inhibitor decreased the mRNA expression and secretion of interleukin (IL)-6 and IL-10 cytokines and increased the level of IL-12 in DCs, whereas an miR-128 inhibitor exhibited the opposite effects.	mir-128	13-20	interleukin 6	Mus musculus	91-109
29067466-9	2017	Furthermore, miR-128 mimic or p38 inhibitor decreased the mRNA expression and secretion of interleukin (IL)-6 and IL-10 cytokines and increased the level of IL-12 in DCs, whereas an miR-128 inhibitor exhibited the opposite effects.	mir-128	13-20	interleukin 10	Mus musculus	114-119
29067466-9	2017	Furthermore, miR-128 mimic or p38 inhibitor decreased the mRNA expression and secretion of interleukin (IL)-6 and IL-10 cytokines and increased the level of IL-12 in DCs, whereas an miR-128 inhibitor exhibited the opposite effects.	mir-128	182-189	mitogen-activated protein kinase 14	Mus musculus	30-33
28514100-3	2017	The miR-128/NEK2 pathway has been reported to predict prognosis in colorectal cancer; however, the determination of a relationship between miR-128 and NEK2 in lung cancer has remained elusive.	mir-128	139-146	NIMA related kinase 2	Homo sapiens	151-155
29067466-10	2017	These findings suggested that miR-128 regulated the immune response of DCs via p38-downstream cytokines.	mir-128	30-37	mitogen-activated protein kinase 14	Mus musculus	79-82
29067466-12	2017	The results therefore suggest that miR-128 enhances the anti-tumor immunity response of DCs via targeting of the p38 mitogen activated protein kinase signaling pathway.	mir-128	35-42	mitogen-activated protein kinase 14	Mus musculus	113-116
28514100-4	2017	We explored the association between miR-128 and NEK2 in lung cancer.	mir-128	36-43	NIMA related kinase 2	Homo sapiens	48-52
28514100-11	2017	Dual luciferase assay further confirmed that NEK2 was a direct target of miR-128 in lung cancer, and transfection with miR-128 mimic could decrease the NEK2 protein level while the miR-128 inhibitor increased NEK2 expression.	mir-128	73-80	NIMA related kinase 2	Homo sapiens	45-49
28514100-11	2017	Dual luciferase assay further confirmed that NEK2 was a direct target of miR-128 in lung cancer, and transfection with miR-128 mimic could decrease the NEK2 protein level while the miR-128 inhibitor increased NEK2 expression.	mir-128	73-80	NIMA related kinase 2	Homo sapiens	152-156
28514100-11	2017	Dual luciferase assay further confirmed that NEK2 was a direct target of miR-128 in lung cancer, and transfection with miR-128 mimic could decrease the NEK2 protein level while the miR-128 inhibitor increased NEK2 expression.	mir-128	73-80	NIMA related kinase 2	Homo sapiens	152-156
28514100-11	2017	Dual luciferase assay further confirmed that NEK2 was a direct target of miR-128 in lung cancer, and transfection with miR-128 mimic could decrease the NEK2 protein level while the miR-128 inhibitor increased NEK2 expression.	mir-128	119-126	NIMA related kinase 2	Homo sapiens	45-49
28514100-11	2017	Dual luciferase assay further confirmed that NEK2 was a direct target of miR-128 in lung cancer, and transfection with miR-128 mimic could decrease the NEK2 protein level while the miR-128 inhibitor increased NEK2 expression.	mir-128	119-126	NIMA related kinase 2	Homo sapiens	152-156
28514100-11	2017	Dual luciferase assay further confirmed that NEK2 was a direct target of miR-128 in lung cancer, and transfection with miR-128 mimic could decrease the NEK2 protein level while the miR-128 inhibitor increased NEK2 expression.	mir-128	119-126	NIMA related kinase 2	Homo sapiens	152-156
28514100-12	2017	Finally, the apoptotic effect of lung cancer cells induced by miR-128 mimic could be reversed by NEK2 overexpression.	mir-128	62-69	NIMA related kinase 2	Homo sapiens	97-101
28514100-13	2017	CONCLUSIONS: NEK2 was regulated by miR-128 in lung cancer and miR-128 induced lung cancer cell apoptosis by mediating NEK2 expression.	mir-128	35-42	NIMA related kinase 2	Homo sapiens	13-17
28514100-13	2017	CONCLUSIONS: NEK2 was regulated by miR-128 in lung cancer and miR-128 induced lung cancer cell apoptosis by mediating NEK2 expression.	mir-128	35-42	NIMA related kinase 2	Homo sapiens	118-122
28514100-13	2017	CONCLUSIONS: NEK2 was regulated by miR-128 in lung cancer and miR-128 induced lung cancer cell apoptosis by mediating NEK2 expression.	mir-128	62-69	NIMA related kinase 2	Homo sapiens	13-17
28514100-13	2017	CONCLUSIONS: NEK2 was regulated by miR-128 in lung cancer and miR-128 induced lung cancer cell apoptosis by mediating NEK2 expression.	mir-128	62-69	NIMA related kinase 2	Homo sapiens	118-122
28424413-4	2017	Using prediction tools, western blotting, and luciferase reporter assays, we found that Bmi-1 was the direct target of miR-128.	mir-128	119-126	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	88-93
28424413-5	2017	Additionally, overexpression of miR-128 inhibited gastric cancer cell migration, invasion, and proliferation by targeting Bmi-1 in vitro and in vivo.	mir-128	32-39	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	122-127
28424413-8	2017	In fact, we found that miR-128 could reverse epithelial-to-mesenchymal transition induced by Bmi-1 via the PI3K/AKT pathway.	mir-128	23-30	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	93-98
28424413-8	2017	In fact, we found that miR-128 could reverse epithelial-to-mesenchymal transition induced by Bmi-1 via the PI3K/AKT pathway.	mir-128	23-30	AKT serine/threonine kinase 1	Homo sapiens	112-115
27485180-0	2017	miR-128 promoted adipogenic differentiation and inhibited osteogenic differentiation of human mesenchymal stem cells by suppression of VEGF pathway.	mir-128	0-7	vascular endothelial growth factor A	Homo sapiens	135-139
27485180-8	2017	Furthermore, over-expression of miR-128 down-regulated VEGF expression in adipogenically and osteogenically differentiated cells.	mir-128	32-39	vascular endothelial growth factor A	Homo sapiens	55-59
27485180-9	2017	We further identified VEGF as a key regulator in miR-128-induced adipogenic and osteogenic differentiation.	mir-128	49-56	vascular endothelial growth factor A	Homo sapiens	22-26
27485180-11	2017	CONCLUSION: It was indicated that miR-128 could regulate adipogenic and osteogenic differentiation of hMSCs significantly through the suppression of VEGF pathway.	mir-128	34-41	vascular endothelial growth factor A	Homo sapiens	149-153
28146425-0	2017	Posttranscriptional regulation of Galectin-3 by miR-128 contributes to colorectal cancer progression.	mir-128	48-55	galectin 3	Homo sapiens	34-44
28179359-8	2017	SRC knockdown was rescued by antagomir to miR-128.	mir-128	42-49	SRC proto-oncogene, non-receptor tyrosine kinase	Homo sapiens	0-3
28146425-4	2017	To explore the mechanism for Galectin-3 dysregulation, we found that miR-128 level was frequently down-regulated in CRC and negatively correlated with Galectin-3 level.	mir-128	69-76	galectin 3	Homo sapiens	29-39
28146425-4	2017	To explore the mechanism for Galectin-3 dysregulation, we found that miR-128 level was frequently down-regulated in CRC and negatively correlated with Galectin-3 level.	mir-128	69-76	galectin 3	Homo sapiens	151-161
28146425-5	2017	Using bioinformatics analysis and experimental validation, we showed that miR-128 could directly target Galectin-3 to repress its protein level.	mir-128	74-81	galectin 3	Homo sapiens	104-114
28146425-8	2017	Galectin-3 expression impaired the cancer suppressive effects of miR-128.	mir-128	65-72	galectin 3	Homo sapiens	0-10
28114268-10	2017	In addition, miR-128 overexpression significantly decreased the expression levels of microglial M1 phenotypic markers CD86 and CD32, and increased the expression levels of M2 phenotypic markers Arg1 and CD206.	mir-128	13-20	CD86 antigen	Mus musculus	118-122
27905005-6	2017	To reveal the mechanism for posttranscription of p38alpha protein, we tested the effect of miR-128-3p.	mir-128	91-98	mitogen-activated protein kinase 14	Mus musculus	49-57
28114268-10	2017	In addition, miR-128 overexpression significantly decreased the expression levels of microglial M1 phenotypic markers CD86 and CD32, and increased the expression levels of M2 phenotypic markers Arg1 and CD206.	mir-128	13-20	mannose receptor, C type 1	Mus musculus	203-208
28114268-11	2017	Furthermore, miR-128 overexpression obviously decreased the concentration of TNF-alpha, IL-1beta, and IL-6.	mir-128	13-20	tumor necrosis factor	Mus musculus	77-86
28114268-11	2017	Furthermore, miR-128 overexpression obviously decreased the concentration of TNF-alpha, IL-1beta, and IL-6.	mir-128	13-20	interleukin 1 beta	Mus musculus	88-96
28114268-11	2017	Furthermore, miR-128 overexpression obviously decreased the concentration of TNF-alpha, IL-1beta, and IL-6.	mir-128	13-20	interleukin 6	Mus musculus	102-106
28114268-12	2017	We found that miR-128 overexpression significantly downregulated the expression levels of P38 andP-P38.	mir-128	14-21	mitogen-activated protein kinase 14	Mus musculus	90-93
28114268-12	2017	We found that miR-128 overexpression significantly downregulated the expression levels of P38 andP-P38.	mir-128	14-21	mitogen-activated protein kinase 14	Mus musculus	99-102
28114268-13	2017	CONCLUSIONS Our findings indicate that down-regulation of miR-128 in murine microglial cells may contribute to the development of NPP following SCI via activation of P38.	mir-128	58-65	mitogen-activated protein kinase 14	Mus musculus	166-169
28114268-10	2017	In addition, miR-128 overexpression significantly decreased the expression levels of microglial M1 phenotypic markers CD86 and CD32, and increased the expression levels of M2 phenotypic markers Arg1 and CD206.	mir-128	13-20	Fc receptor, IgG, low affinity IIb	Mus musculus	127-131
28114268-10	2017	In addition, miR-128 overexpression significantly decreased the expression levels of microglial M1 phenotypic markers CD86 and CD32, and increased the expression levels of M2 phenotypic markers Arg1 and CD206.	mir-128	13-20	arginase, liver	Mus musculus	194-198
27695040-3	2016	In mouse and human ALS spinal cord, the reduction of miR-128 that destabilizes TrkC.T1 mRNA results in up-regulated TrkC.T1 and TNF-alpha in astrocytes.	mir-128	53-60	neurotrophic receptor tyrosine kinase 3	Homo sapiens	79-83
27690301-0	2016	MiR-128 reverses the gefitinib resistance of the lung cancer stem cells by inhibiting the c-met/PI3K/AKT pathway.	mir-128	0-7	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	90-95
27690301-0	2016	MiR-128 reverses the gefitinib resistance of the lung cancer stem cells by inhibiting the c-met/PI3K/AKT pathway.	mir-128	0-7	AKT serine/threonine kinase 1	Homo sapiens	101-104
27690301-5	2016	Mechanically, we showed significant down-regulation of miR-128 in the PC9-CSCs compared with the non-CSCs.	mir-128	55-62	proprotein convertase subtilisin/kexin type 9	Homo sapiens	70-73
27690301-6	2016	Overexpression of miR-128 significantly increased the sensitivity of PC9-CSCs to gefitinib-induced apoptosis.	mir-128	18-25	proprotein convertase subtilisin/kexin type 9	Homo sapiens	69-72
27690301-7	2016	In addition, the gene of c-met was proved to be directly inhibited by miR-128.	mir-128	70-77	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	25-30
27690301-8	2016	Enforced expression of c-met could "rescue" the miR-128 promoted apoptosis and cleavage of caspases in PC9-CSCs treated with gefitinib.	mir-128	48-55	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	23-28
27690301-8	2016	Enforced expression of c-met could "rescue" the miR-128 promoted apoptosis and cleavage of caspases in PC9-CSCs treated with gefitinib.	mir-128	48-55	proprotein convertase subtilisin/kexin type 9	Homo sapiens	103-106
27695040-3	2016	In mouse and human ALS spinal cord, the reduction of miR-128 that destabilizes TrkC.T1 mRNA results in up-regulated TrkC.T1 and TNF-alpha in astrocytes.	mir-128	53-60	neurotrophic receptor tyrosine kinase 3	Homo sapiens	116-120
27695040-3	2016	In mouse and human ALS spinal cord, the reduction of miR-128 that destabilizes TrkC.T1 mRNA results in up-regulated TrkC.T1 and TNF-alpha in astrocytes.	mir-128	53-60	tumor necrosis factor	Homo sapiens	128-137
26700675-8	2016	Importantly, we identified that the 3"-untranslated region (3"-UTR) of ITGA2 was a direct target of miR-128.	mir-128	100-107	integrin subunit alpha 2	Homo sapiens	71-76
26307612-9	2016	We also identified that TROVE2 is a novel target of miR-128 by the luciferase reporter system.	mir-128	52-59	Ro60, Y RNA binding protein	Homo sapiens	24-30
26307612-11	2016	Finally, the levels of TROVE2 are negatively correlated with miR-128 in astrocytoma tissues.	mir-128	61-68	Ro60, Y RNA binding protein	Homo sapiens	23-29
27222923-1	2016	MicroRNA-128-3p (miR-128) is a brain-enriched microRNA reported to target Doublecortin (Dcx), a key transcriptional factor during adult neurogenesis.	mir-128	17-24	doublecortin	Mus musculus	74-86
27322220-6	2016	Knockdown of the most abundant miRNAs in exosomes and the MCF-7 proliferation assay showed that miR-128 in exosomes negatively regulates the level of Bax in MCF-7 recipient cells and inhibits cell proliferation.	mir-128	96-103	BCL2 associated X, apoptosis regulator	Homo sapiens	150-153
27893811-10	2016	Furthermore, the overexpression of miR-128 alone enhanced apoptotic death of glioma cells through caspase-3/9 activation, poly(ADP ribose) polymerase degradation, reactive oxygen species generation, mitochondrial membrane potential loss, and non-protective autophagy formation.	mir-128	35-42	caspase 3	Homo sapiens	98-107
27893811-10	2016	Furthermore, the overexpression of miR-128 alone enhanced apoptotic death of glioma cells through caspase-3/9 activation, poly(ADP ribose) polymerase degradation, reactive oxygen species generation, mitochondrial membrane potential loss, and non-protective autophagy formation.	mir-128	35-42	poly(ADP-ribose) polymerase 1	Homo sapiens	122-149
27893811-12	2016	TMZ inhibited mTOR signaling through miR-128 regulation.	mir-128	37-44	mechanistic target of rapamycin kinase	Homo sapiens	14-18
27893811-13	2016	These results indicate that miR-128-inhibited mTOR signaling is involved in TMZ-mediated cytotoxicity.	mir-128	28-35	mechanistic target of rapamycin kinase	Homo sapiens	46-50
27222923-1	2016	MicroRNA-128-3p (miR-128) is a brain-enriched microRNA reported to target Doublecortin (Dcx), a key transcriptional factor during adult neurogenesis.	mir-128	17-24	doublecortin	Mus musculus	88-91
27222923-2	2016	However, the downstream physiological effects of this miR-128-DCX axis remain unclear.	mir-128	54-61	doublecortin	Mus musculus	62-65
27222923-4	2016	During differentiation of neural stem cells, over-expressing miR-128 with a lentivirus system inhibited the up-regulation of Dcx on Day 5, subsequently decreasing the percentage of TuJ+ cells on Day 16.	mir-128	61-68	doublecortin	Mus musculus	125-128
27222923-5	2016	Administration of the lentivirus encoding miR-128 into mouse hippocampi significantly impaired water maze learning after 14days, which could be attenuated when the Dcx-encoding virus was delivered simultaneously.	mir-128	42-49	doublecortin	Mus musculus	164-167
27222923-6	2016	In addition, similar changes including miR-128 up-regulation, Dcx down-regulation and learning defects were observed after a 14-day infusion of Abeta-42, which were also partially reversed by over-expressing Dcx.	mir-128	39-46	doublecortin	Mus musculus	208-211
27150726-2	2016	In vitro, the expression of PPARG was detected by reverse transcription-quantitative polymerase chain reaction and western blotting in neonatal rat ventricular myocytes (NRVMs) and HEK293 cells transfected with the mimics or inhibitors of miR-128 or control RNA.	mir-128	239-246	peroxisome proliferator-activated receptor gamma	Rattus norvegicus	28-33
27150726-3	2016	Luciferase reporter assays were used to identify whether PPARG is a direct target of miR-128.	mir-128	85-92	peroxisome proliferator activated receptor gamma	Homo sapiens	57-62
27150726-7	2016	PPARG mRNA and protein were downregulated in NRVMs transfected with miR-128 mimics, but upregulated by antagomir-128 compared with control.	mir-128	68-75	peroxisome proliferator activated receptor gamma	Homo sapiens	0-5
27150726-8	2016	This indicates that PPARG is a direct miR-128 target.	mir-128	38-45	peroxisome proliferator activated receptor gamma	Homo sapiens	20-25
27150726-9	2016	Activation of Akt (p-Akt), Mcl-1 and PPARG expression in the myocardium were increased by miR-128 inhibition.	mir-128	90-97	AKT serine/threonine kinase 1	Homo sapiens	14-17
27150726-9	2016	Activation of Akt (p-Akt), Mcl-1 and PPARG expression in the myocardium were increased by miR-128 inhibition.	mir-128	90-97	AKT serine/threonine kinase 1	Homo sapiens	19-24
27150726-9	2016	Activation of Akt (p-Akt), Mcl-1 and PPARG expression in the myocardium were increased by miR-128 inhibition.	mir-128	90-97	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	27-32
27150726-9	2016	Activation of Akt (p-Akt), Mcl-1 and PPARG expression in the myocardium were increased by miR-128 inhibition.	mir-128	90-97	peroxisome proliferator activated receptor gamma	Homo sapiens	37-42
27150726-10	2016	Furthermore, miR-128 antagomirs significantly reduced apoptosis in hearts subjected to I/R injury, which was blocked by the PPARG inhibitor GW9662.	mir-128	13-20	peroxisome proliferator activated receptor gamma	Homo sapiens	124-129
27150726-11	2016	In conclusion, miR-128 inhibition attenuated I/R injury-induced cardiomyocyte apoptosis by the targeted activation of PPARG signaling.	mir-128	15-22	peroxisome proliferator activated receptor gamma	Homo sapiens	118-123
27341528-0	2016	AurkA controls self-renewal of breast cancer-initiating cells promoting wnt3a stabilization through suppression of miR-128.	mir-128	115-122	aurora kinase A	Homo sapiens	0-5
27341528-7	2016	Interestingly, we found that AurkA suppressed the expression of miR-128, inhibitor of wnt3a mRNA stabilization.	mir-128	64-71	aurora kinase A	Homo sapiens	29-34
27341528-7	2016	Interestingly, we found that AurkA suppressed the expression of miR-128, inhibitor of wnt3a mRNA stabilization.	mir-128	64-71	Wnt family member 3A	Homo sapiens	86-91
27341528-8	2016	Namely, miR-128 suppression realized after AurkA binding to Snail.	mir-128	8-15	aurora kinase A	Homo sapiens	43-48
27341528-8	2016	Namely, miR-128 suppression realized after AurkA binding to Snail.	mir-128	8-15	snail family transcriptional repressor 1	Homo sapiens	60-65
27341528-11	2016	In addition, it suggests a new therapeutic strategy taking advantage of miR-128 to suppress AurkA-Wnt3a signaling.	mir-128	72-79	aurora kinase A	Homo sapiens	92-97
27341528-11	2016	In addition, it suggests a new therapeutic strategy taking advantage of miR-128 to suppress AurkA-Wnt3a signaling.	mir-128	72-79	Wnt family member 3A	Homo sapiens	98-103
27133073-6	2016	In addition, mini-hF9 transcripts were accumulated after transfection with miR-128 or miR-125 mimics in E7a and E7b.	mir-128	75-82	coagulation factor IX	Homo sapiens	18-21
27087048-0	2016	miR-128 regulates differentiation of hair follicle mesenchymal stem cells into smooth muscle cells by targeting SMAD2.	mir-128	0-7	SMAD family member 2	Homo sapiens	112-117
27087048-3	2016	In present study, we found that miR-128 was remarkably decreased during the differentiation of hHFMSCs into SMCs induced by transforming growth factor-beta1 (TGF-beta1).	mir-128	32-39	transforming growth factor beta 1	Homo sapiens	124-156
27087048-3	2016	In present study, we found that miR-128 was remarkably decreased during the differentiation of hHFMSCs into SMCs induced by transforming growth factor-beta1 (TGF-beta1).	mir-128	32-39	transforming growth factor beta 1	Homo sapiens	158-167
27087048-4	2016	Moreover, overexpression of miR-128 led to decreased expression of SMC cellular marker proteins, such as smooth muscle actin (SMA) and calponin, in TGF-beta1-induced SMC differentiation.	mir-128	28-35	transforming growth factor beta 1	Homo sapiens	148-157
27087048-5	2016	Further, we identified that miR-128 targeted the 3"-UTR of SMAD2 transcript for translational inhibition of SMAD2 protein, and knockdown of SMAD2 abrogated the promotional effect of antagomir-128 (miR-128 neutralizer) on SMC differentiation.	mir-128	28-35	SMAD family member 2	Homo sapiens	59-64
27087048-5	2016	Further, we identified that miR-128 targeted the 3"-UTR of SMAD2 transcript for translational inhibition of SMAD2 protein, and knockdown of SMAD2 abrogated the promotional effect of antagomir-128 (miR-128 neutralizer) on SMC differentiation.	mir-128	28-35	SMAD family member 2	Homo sapiens	108-113
27087048-5	2016	Further, we identified that miR-128 targeted the 3"-UTR of SMAD2 transcript for translational inhibition of SMAD2 protein, and knockdown of SMAD2 abrogated the promotional effect of antagomir-128 (miR-128 neutralizer) on SMC differentiation.	mir-128	28-35	SMAD family member 2	Homo sapiens	108-113
27087048-6	2016	These results suggest that miR-128 regulates the differentiation of hHFMSCs into SMCs via targeting SMAD2, a main transcription regulator in TGF-beta signaling pathway involving SMC differentiation.	mir-128	27-34	SMAD family member 2	Homo sapiens	100-105
27087048-6	2016	These results suggest that miR-128 regulates the differentiation of hHFMSCs into SMCs via targeting SMAD2, a main transcription regulator in TGF-beta signaling pathway involving SMC differentiation.	mir-128	27-34	transforming growth factor beta 1	Homo sapiens	141-149
26700675-9	2016	Luciferase reporter assays confirmed that miR-128 binding to the 3"-UTR regions of ITGA2 inhibited the expression of ITGA2 in MG-63 cells.	mir-128	42-49	integrin subunit alpha 2	Homo sapiens	83-88
26700675-9	2016	Luciferase reporter assays confirmed that miR-128 binding to the 3"-UTR regions of ITGA2 inhibited the expression of ITGA2 in MG-63 cells.	mir-128	42-49	integrin subunit alpha 2	Homo sapiens	117-122
26700675-10	2016	At the same time, overexpressed ITGA2 also reversed EMT inhibited by miR-128.	mir-128	69-76	integrin subunit alpha 2	Homo sapiens	32-37
26700675-11	2016	In conclusion, this study suggested that high miR-128 expression suppressed osteosarcoma cell migration, invasion, and EMT development through targeting ITGA2, which may be recommended as a therapeutic target for osteosarcoma.	mir-128	46-53	integrin subunit alpha 2	Homo sapiens	153-158
26833195-9	2016	Changes in miR-128 and Sp1 expression levels also affected the protein levels of MyoD and CDKN1A.	mir-128	11-18	myogenic differentiation 1	Bos taurus	81-85
27186316-0	2016	miR-128 modulates hepatocellular carcinoma by inhibition of ITGA2 and ITGA5 expression [Retraction].	mir-128	0-7	integrin subunit alpha 2	Homo sapiens	60-65
27186316-0	2016	miR-128 modulates hepatocellular carcinoma by inhibition of ITGA2 and ITGA5 expression [Retraction].	mir-128	0-7	integrin subunit alpha 5	Homo sapiens	70-75
26460960-3	2015	Suppression of VEGF-C by miR-27b, miR-101 and miR-128 was investigated by luciferase assays, Western blot and ELISA.	mir-128	46-53	vascular endothelial growth factor C	Homo sapiens	15-21
26883496-5	2016	Overexpression of miR-128 suppressed the translation of PCM1, and knockdown of endogenous PCM1 phenocopied the observed effects of miR-128 overexpression.	mir-128	18-25	pericentriolar material 1	Homo sapiens	56-60
26883496-5	2016	Overexpression of miR-128 suppressed the translation of PCM1, and knockdown of endogenous PCM1 phenocopied the observed effects of miR-128 overexpression.	mir-128	131-138	pericentriolar material 1	Homo sapiens	90-94
26883496-6	2016	Furthermore, concomitant overexpression of PCM1 and miR-128 in NPCs rescued the phenotype associated with miR-128 overexpression, enhancing neurogenesis but inhibiting proliferation, both in vitro and in utero.	mir-128	106-113	pericentriolar material 1	Homo sapiens	43-47
27186417-3	2016	Here, we show that miR-128 directly targets PFK liver type (PFKL) in lung cancer cells and regulates endogenous expression of PFKL at both the mRNA and protein levels.	mir-128	19-26	phosphofructokinase, liver type	Homo sapiens	60-64
27186417-3	2016	Here, we show that miR-128 directly targets PFK liver type (PFKL) in lung cancer cells and regulates endogenous expression of PFKL at both the mRNA and protein levels.	mir-128	19-26	phosphofructokinase, liver type	Homo sapiens	126-130
27186417-6	2016	Moreover, we observed that miR-128 expression inversely correlated with PFKL mRNA levels in clinic lung cancer samples and that increased PFKL expression predicted poor overall survival in lung cancer patients.	mir-128	27-34	phosphofructokinase, liver type	Homo sapiens	72-76
27186417-7	2016	Mechanistically, we showed that miR-128 regulates PFKL via a feedback loop that involves inhibition of the AKT signaling pathway.	mir-128	32-39	phosphofructokinase, liver type	Homo sapiens	50-54
27186417-7	2016	Mechanistically, we showed that miR-128 regulates PFKL via a feedback loop that involves inhibition of the AKT signaling pathway.	mir-128	32-39	AKT serine/threonine kinase 1	Homo sapiens	107-110
26460960-9	2015	Overexpression of miR-27b, miR-101, or miR-128 suppressed migration, proliferation activity, and tube formation in HUVECs by repressing VEGF-C secretion in gastric cancer cells.	mir-128	39-46	vascular endothelial growth factor C	Homo sapiens	136-142
26460960-10	2015	We conclude that miR-27b, miR-101 and miR-128 inhibit angiogenesis by down-regulating VEGF-C expression in gastric cancers.	mir-128	38-45	vascular endothelial growth factor C	Homo sapiens	86-92
25958325-9	2015	Taken together, these results reveal that there is a novel pathway in skeletal muscle development in which miR-128 regulates myostatin at CDS region to inhibit proliferation but promote differentiation of myoblast cells.	mir-128	107-114	myostatin	Mus musculus	125-134
26722309-0	2015	miR-128 downregulation promotes growth and metastasis of bladder cancer cells and involves VEGF-C upregulation.	mir-128	0-7	vascular endothelial growth factor C	Homo sapiens	91-97
26722309-3	2015	To investigate the regulation of miR-128 on VEGF-C expression and their effects on proliferation and metastasis of bladder cancer, T24 and 5637 BC cells were transfected with pre-miR-128, anti-miR-128 and their respective negative control.	mir-128	33-40	vascular endothelial growth factor C	Homo sapiens	44-50
26722309-5	2015	The present results indicated that miR-128 negatively regulated VEGF-C expression in BC T24 and 5637 BC cells.	mir-128	35-42	vascular endothelial growth factor C	Homo sapiens	64-70
26722309-6	2015	VEGF-C is a direct target of miR-128 in BC cells.	mir-128	29-36	vascular endothelial growth factor C	Homo sapiens	0-6
26352220-7	2015	In addition, insulin receptor substrate 1 (IRS1), a key mediator in oncogenic insulin-like growth factor (IGF) signaling, was confirmed as a direct target of miR-128 by a luciferase reporter assay.	mir-128	158-165	insulin receptor substrate 1	Homo sapiens	13-41
26352220-7	2015	In addition, insulin receptor substrate 1 (IRS1), a key mediator in oncogenic insulin-like growth factor (IGF) signaling, was confirmed as a direct target of miR-128 by a luciferase reporter assay.	mir-128	158-165	insulin receptor substrate 1	Homo sapiens	43-47
26352220-8	2015	Western blot analysis indicated that the overexpression of miR-128 significantly downregulated IRS1 expression and its downstream Akt signaling in CRC cells.	mir-128	59-66	insulin receptor substrate 1	Homo sapiens	95-99
26352220-9	2015	Moreover, miR-128 was negatively associated with IRS1 in CRC tissues compared to adjacent non-tumor tissues.	mir-128	10-17	insulin receptor substrate 1	Homo sapiens	49-53
26352220-10	2015	Taken together, these data suggested that miR-128 serves as a tumor suppressor and blocks CRC growth and metastasis by targeting IRS1.	mir-128	42-49	insulin receptor substrate 1	Homo sapiens	129-133
26550456-0	2015	miR-128 modulates hepatocellular carcinoma by inhibition of ITGA2 and ITGA5 expression.	mir-128	0-7	integrin subunit alpha 2	Homo sapiens	60-65
26550456-0	2015	miR-128 modulates hepatocellular carcinoma by inhibition of ITGA2 and ITGA5 expression.	mir-128	0-7	integrin subunit alpha 5	Homo sapiens	70-75
26550456-6	2015	ITGA2 and ITGA5 were inversely correlated with the expression of miR-128 in HCC cells.	mir-128	65-72	integrin subunit alpha 2	Homo sapiens	0-5
26550456-6	2015	ITGA2 and ITGA5 were inversely correlated with the expression of miR-128 in HCC cells.	mir-128	65-72	integrin subunit alpha 5	Homo sapiens	10-15
26550456-7	2015	Importantly, we demonstrate that the overexpression of miR-128 significantly inhibits HCC cell metastasis and stem-cell like properties via ITGA2 and ITGA5.	mir-128	55-62	integrin subunit alpha 2	Homo sapiens	140-145
26550456-7	2015	Importantly, we demonstrate that the overexpression of miR-128 significantly inhibits HCC cell metastasis and stem-cell like properties via ITGA2 and ITGA5.	mir-128	55-62	integrin subunit alpha 5	Homo sapiens	150-155
26553132-5	2015	It promoted the association of p53 to the promoter region of miR-128, and enhanced the transcriptional activation of p53 on miR-128 expression.	mir-128	61-68	tumor protein p53	Homo sapiens	31-34
26553132-6	2015	miR-128 can downregulate prohibitin expression, and subsequently promote apoptosis.	mir-128	0-7	prohibitin 1	Homo sapiens	25-35
25781272-1	2015	Brain-enriched miR-128 is repressed in glioma cells, and could inhibit the proliferation of gliomas by targeting genes such as E2F3a and BMI1.	mir-128	15-22	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	137-141
25781272-7	2015	Moreover, we observed that the expression of CORO1C, TROVE2, and HnRNPF were higher in glioma cell lines compared to normal brain tissues and presented a tendency toward downregulation after overexpression of miR-128 in T98G cells.	mir-128	209-216	coronin 1C	Homo sapiens	45-51
25781272-7	2015	Moreover, we observed that the expression of CORO1C, TROVE2, and HnRNPF were higher in glioma cell lines compared to normal brain tissues and presented a tendency toward downregulation after overexpression of miR-128 in T98G cells.	mir-128	209-216	Ro60, Y RNA binding protein	Homo sapiens	53-59
25781272-7	2015	Moreover, we observed that the expression of CORO1C, TROVE2, and HnRNPF were higher in glioma cell lines compared to normal brain tissues and presented a tendency toward downregulation after overexpression of miR-128 in T98G cells.	mir-128	209-216	heterogeneous nuclear ribonucleoprotein F	Homo sapiens	65-71
25742789-5	2015	Furthermore, we demonstrated that three murine microRNAs, namely miR-128, -134, and -330, can target the three Mmps Mmp3, Mmp10, and Mmp13, respectively.	mir-128	65-72	matrix metallopeptidase 13	Mus musculus	111-115
25921099-0	2015	miR-128 modulates chemosensitivity and invasion of prostate cancer cells through targeting ZEB1.	mir-128	0-7	zinc finger E-box binding homeobox 1	Homo sapiens	91-95
25742789-5	2015	Furthermore, we demonstrated that three murine microRNAs, namely miR-128, -134, and -330, can target the three Mmps Mmp3, Mmp10, and Mmp13, respectively.	mir-128	65-72	matrix metallopeptidase 3	Mus musculus	116-120
25742789-5	2015	Furthermore, we demonstrated that three murine microRNAs, namely miR-128, -134, and -330, can target the three Mmps Mmp3, Mmp10, and Mmp13, respectively.	mir-128	65-72	matrix metallopeptidase 10	Mus musculus	122-127
25742789-5	2015	Furthermore, we demonstrated that three murine microRNAs, namely miR-128, -134, and -330, can target the three Mmps Mmp3, Mmp10, and Mmp13, respectively.	mir-128	65-72	matrix metallopeptidase 13	Mus musculus	133-138
25246803-2	2014	Especially, it has been demonstrated that miR-128 may play an important role in the proliferation of human osteosarcoma cells in vitro by directly inhibiting PTEN, which functions as a tumor suppressor in this malignancy.	mir-128	42-49	phosphatase and tensin homolog	Homo sapiens	158-162
25556700-0	2015	miR-128 regulates neuronal migration, outgrowth and intrinsic excitability via the intellectual disability gene Phf6.	mir-128	0-7	PHD finger protein 6	Mus musculus	112-116
25556700-5	2015	We show that Phf6, a gene mutated in the cognitive disorder Borjeson-Forssman-Lehmann syndrome, is an important regulatory target for miR-128.	mir-128	134-141	PHD finger protein 6	Mus musculus	13-17
25556700-6	2015	Restoring PHF6 expression counteracts the deleterious effect of miR-128 on neuronal migration, outgrowth and intrinsic physiological properties.	mir-128	64-71	PHD finger protein 6	Mus musculus	10-14
25556700-7	2015	Our results place miR-128 upstream of PHF6 in a pathway vital for cortical lamination as well as for the development of neuronal morphology and intrinsic excitability.	mir-128	18-25	PHD finger protein 6	Mus musculus	38-42
25248111-6	2014	Overexpression of miR-128 resensitized SKOV3/CP cells to cisplatin and reduced the expression of cisplatin-resistant-related proteins ABCC5 and Bmi-1, whereas miR-128 inhibitors increased cisplatin resistance in SKOV3 cells.	mir-128	18-25	ATP binding cassette subfamily C member 5	Homo sapiens	134-139
25248111-6	2014	Overexpression of miR-128 resensitized SKOV3/CP cells to cisplatin and reduced the expression of cisplatin-resistant-related proteins ABCC5 and Bmi-1, whereas miR-128 inhibitors increased cisplatin resistance in SKOV3 cells.	mir-128	18-25	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	144-149
25246803-9	2014	CONCLUSION: These findings indicate for the first time that the deregulation of miR-128 and its target gene PTEN may be involved in the aggressive progression of human osteosarcoma.	mir-128	80-87	phosphatase and tensin homolog	Homo sapiens	108-112
25246803-10	2014	Notably, the upregulation of miR-128 cooperating with the downregulation of PTEN may confer an unfavorable prognosis in patients with this malignancy.	mir-128	29-36	phosphatase and tensin homolog	Homo sapiens	76-80
24959930-11	2014	The CGGA data showed that the Pearson correlation index between SNAI1 and miR-128 was negatively correlated.	mir-128	74-81	snail family transcriptional repressor 1	Homo sapiens	64-69
24859886-0	2014	Loss of SNAIL inhibits cellular growth and metabolism through the miR-128-mediated RPS6KB1/HIF-1alpha/PKM2 signaling pathway in prostate cancer cells.	mir-128	66-73	snail family transcriptional repressor 1	Homo sapiens	8-13
24859886-0	2014	Loss of SNAIL inhibits cellular growth and metabolism through the miR-128-mediated RPS6KB1/HIF-1alpha/PKM2 signaling pathway in prostate cancer cells.	mir-128	66-73	ribosomal protein S6 kinase B1	Homo sapiens	83-90
24859886-0	2014	Loss of SNAIL inhibits cellular growth and metabolism through the miR-128-mediated RPS6KB1/HIF-1alpha/PKM2 signaling pathway in prostate cancer cells.	mir-128	66-73	hypoxia inducible factor 1 subunit alpha	Homo sapiens	91-101
24859886-0	2014	Loss of SNAIL inhibits cellular growth and metabolism through the miR-128-mediated RPS6KB1/HIF-1alpha/PKM2 signaling pathway in prostate cancer cells.	mir-128	66-73	pyruvate kinase M1/2	Homo sapiens	102-106
24859886-9	2014	Furthermore, down-expression of miR-128 partially restored the effect of si-SNAIL on the suppression of cellular growth, metabolism, and RPS6KB1/HIF-1alpha/PKM2 signaling pathway.	mir-128	32-39	snail family transcriptional repressor 1	Homo sapiens	76-81
24859886-9	2014	Furthermore, down-expression of miR-128 partially restored the effect of si-SNAIL on the suppression of cellular growth, metabolism, and RPS6KB1/HIF-1alpha/PKM2 signaling pathway.	mir-128	32-39	ribosomal protein S6 kinase B1	Homo sapiens	137-144
24859886-9	2014	Furthermore, down-expression of miR-128 partially restored the effect of si-SNAIL on the suppression of cellular growth, metabolism, and RPS6KB1/HIF-1alpha/PKM2 signaling pathway.	mir-128	32-39	hypoxia inducible factor 1 subunit alpha	Homo sapiens	145-155
24859886-9	2014	Furthermore, down-expression of miR-128 partially restored the effect of si-SNAIL on the suppression of cellular growth, metabolism, and RPS6KB1/HIF-1alpha/PKM2 signaling pathway.	mir-128	32-39	pyruvate kinase M1/2	Homo sapiens	156-160
24859886-10	2014	To our knowledge, it is the first time to demonstrate that SNAIL/miR-128/RPS6KB1 pathway plays a critical role in the progression of PCa.	mir-128	65-72	snail family transcriptional repressor 1	Homo sapiens	59-64
24859886-10	2014	To our knowledge, it is the first time to demonstrate that SNAIL/miR-128/RPS6KB1 pathway plays a critical role in the progression of PCa.	mir-128	65-72	ribosomal protein S6 kinase B1	Homo sapiens	73-80
25001183-5	2014	Interestingly, ectopic miR-128 overexpression could significantly inhibit vascular endothelial growth factor (VEGF)-C expression and reduce the activity of a luciferase reporter containing the VEGF-C 3"-untranslated region.	mir-128	23-30	vascular endothelial growth factor A	Homo sapiens	74-108
25001183-5	2014	Interestingly, ectopic miR-128 overexpression could significantly inhibit vascular endothelial growth factor (VEGF)-C expression and reduce the activity of a luciferase reporter containing the VEGF-C 3"-untranslated region.	mir-128	23-30	vascular endothelial growth factor A	Homo sapiens	110-114
25001183-5	2014	Interestingly, ectopic miR-128 overexpression could significantly inhibit vascular endothelial growth factor (VEGF)-C expression and reduce the activity of a luciferase reporter containing the VEGF-C 3"-untranslated region.	mir-128	23-30	vascular endothelial growth factor C	Homo sapiens	193-199
25001183-6	2014	In addition, overexpression of miR-128 in NSCLC cells and human umbilical vein endothelial cells (HUVECs) cells led to decreased expression of VEGF-A, vascular endothelial growth factor receptor (VEGFR)-2 and VEGFR-3, critical factors responsible for cancer angiogenesis and lymphangiogenesis, and subsequently decreased phosphorylation of extracellular signal-regulated kinase (ERK), phosphatidylinositol 3-kinase (AKT) and p38 signalling pathways.	mir-128	31-38	vascular endothelial growth factor A	Homo sapiens	143-149
25001183-6	2014	In addition, overexpression of miR-128 in NSCLC cells and human umbilical vein endothelial cells (HUVECs) cells led to decreased expression of VEGF-A, vascular endothelial growth factor receptor (VEGFR)-2 and VEGFR-3, critical factors responsible for cancer angiogenesis and lymphangiogenesis, and subsequently decreased phosphorylation of extracellular signal-regulated kinase (ERK), phosphatidylinositol 3-kinase (AKT) and p38 signalling pathways.	mir-128	31-38	vascular endothelial growth factor A	Homo sapiens	151-185
25001183-6	2014	In addition, overexpression of miR-128 in NSCLC cells and human umbilical vein endothelial cells (HUVECs) cells led to decreased expression of VEGF-A, vascular endothelial growth factor receptor (VEGFR)-2 and VEGFR-3, critical factors responsible for cancer angiogenesis and lymphangiogenesis, and subsequently decreased phosphorylation of extracellular signal-regulated kinase (ERK), phosphatidylinositol 3-kinase (AKT) and p38 signalling pathways.	mir-128	31-38	kinase insert domain receptor	Homo sapiens	196-204
25001183-6	2014	In addition, overexpression of miR-128 in NSCLC cells and human umbilical vein endothelial cells (HUVECs) cells led to decreased expression of VEGF-A, vascular endothelial growth factor receptor (VEGFR)-2 and VEGFR-3, critical factors responsible for cancer angiogenesis and lymphangiogenesis, and subsequently decreased phosphorylation of extracellular signal-regulated kinase (ERK), phosphatidylinositol 3-kinase (AKT) and p38 signalling pathways.	mir-128	31-38	fms related receptor tyrosine kinase 4	Homo sapiens	209-216
25001183-6	2014	In addition, overexpression of miR-128 in NSCLC cells and human umbilical vein endothelial cells (HUVECs) cells led to decreased expression of VEGF-A, vascular endothelial growth factor receptor (VEGFR)-2 and VEGFR-3, critical factors responsible for cancer angiogenesis and lymphangiogenesis, and subsequently decreased phosphorylation of extracellular signal-regulated kinase (ERK), phosphatidylinositol 3-kinase (AKT) and p38 signalling pathways.	mir-128	31-38	mitogen-activated protein kinase 1	Homo sapiens	340-377
25001183-6	2014	In addition, overexpression of miR-128 in NSCLC cells and human umbilical vein endothelial cells (HUVECs) cells led to decreased expression of VEGF-A, vascular endothelial growth factor receptor (VEGFR)-2 and VEGFR-3, critical factors responsible for cancer angiogenesis and lymphangiogenesis, and subsequently decreased phosphorylation of extracellular signal-regulated kinase (ERK), phosphatidylinositol 3-kinase (AKT) and p38 signalling pathways.	mir-128	31-38	mitogen-activated protein kinase 1	Homo sapiens	379-382
25001183-6	2014	In addition, overexpression of miR-128 in NSCLC cells and human umbilical vein endothelial cells (HUVECs) cells led to decreased expression of VEGF-A, vascular endothelial growth factor receptor (VEGFR)-2 and VEGFR-3, critical factors responsible for cancer angiogenesis and lymphangiogenesis, and subsequently decreased phosphorylation of extracellular signal-regulated kinase (ERK), phosphatidylinositol 3-kinase (AKT) and p38 signalling pathways.	mir-128	31-38	AKT serine/threonine kinase 1	Homo sapiens	416-419
25001183-6	2014	In addition, overexpression of miR-128 in NSCLC cells and human umbilical vein endothelial cells (HUVECs) cells led to decreased expression of VEGF-A, vascular endothelial growth factor receptor (VEGFR)-2 and VEGFR-3, critical factors responsible for cancer angiogenesis and lymphangiogenesis, and subsequently decreased phosphorylation of extracellular signal-regulated kinase (ERK), phosphatidylinositol 3-kinase (AKT) and p38 signalling pathways.	mir-128	31-38	mitogen-activated protein kinase 1	Homo sapiens	425-428
25001183-8	2014	These findings suggest that miR-128 could play a role in NSCLC tumourigenesis at least in part by modulation of angiogenesis and lymphangiogenesis through targeting VEGF-C, and could simultaneously block ERK, AKT and p38 signalling pathways.	mir-128	28-35	vascular endothelial growth factor C	Homo sapiens	165-171
25001183-8	2014	These findings suggest that miR-128 could play a role in NSCLC tumourigenesis at least in part by modulation of angiogenesis and lymphangiogenesis through targeting VEGF-C, and could simultaneously block ERK, AKT and p38 signalling pathways.	mir-128	28-35	mitogen-activated protein kinase 1	Homo sapiens	204-207
25001183-8	2014	These findings suggest that miR-128 could play a role in NSCLC tumourigenesis at least in part by modulation of angiogenesis and lymphangiogenesis through targeting VEGF-C, and could simultaneously block ERK, AKT and p38 signalling pathways.	mir-128	28-35	AKT serine/threonine kinase 1	Homo sapiens	209-212
25001183-8	2014	These findings suggest that miR-128 could play a role in NSCLC tumourigenesis at least in part by modulation of angiogenesis and lymphangiogenesis through targeting VEGF-C, and could simultaneously block ERK, AKT and p38 signalling pathways.	mir-128	28-35	mitogen-activated protein kinase 1	Homo sapiens	217-220
25017996-0	2014	miR-128 and miR-149 enhance the chemosensitivity of temozolomide by Rap1B-mediated cytoskeletal remodeling in glioblastoma.	mir-128	0-7	RAP1B, member of RAS oncogene family	Homo sapiens	68-73
24959930-13	2014	Moreover, introduction of miR-128 anti-sense oligonucleotide alleviated the cell cycle retardation, proliferation and invasion inhibition induced by SNAI1 shRNA.	mir-128	26-33	snail family transcriptional repressor 1	Homo sapiens	149-154
24415783-7	2014	Macrophage colony-stimulating factor (M-CSF) was identified as a target of miR-128, and increased miR-128 levels in epithelial cells due to infection with strain SE2472 significantly decreased the level of cell-secreted M-CSF, leading to impaired M-CSF-mediated macrophage recruitment.	mir-128	75-82	colony stimulating factor 1 (macrophage)	Mus musculus	38-43
24415783-7	2014	Macrophage colony-stimulating factor (M-CSF) was identified as a target of miR-128, and increased miR-128 levels in epithelial cells due to infection with strain SE2472 significantly decreased the level of cell-secreted M-CSF, leading to impaired M-CSF-mediated macrophage recruitment.	mir-128	75-82	colony stimulating factor 1 (macrophage)	Mus musculus	220-225
24415783-7	2014	Macrophage colony-stimulating factor (M-CSF) was identified as a target of miR-128, and increased miR-128 levels in epithelial cells due to infection with strain SE2472 significantly decreased the level of cell-secreted M-CSF, leading to impaired M-CSF-mediated macrophage recruitment.	mir-128	75-82	colony stimulating factor 1 (macrophage)	Mus musculus	220-225
24415783-7	2014	Macrophage colony-stimulating factor (M-CSF) was identified as a target of miR-128, and increased miR-128 levels in epithelial cells due to infection with strain SE2472 significantly decreased the level of cell-secreted M-CSF, leading to impaired M-CSF-mediated macrophage recruitment.	mir-128	98-105	colony stimulating factor 1 (macrophage)	Mus musculus	38-43
24415783-7	2014	Macrophage colony-stimulating factor (M-CSF) was identified as a target of miR-128, and increased miR-128 levels in epithelial cells due to infection with strain SE2472 significantly decreased the level of cell-secreted M-CSF, leading to impaired M-CSF-mediated macrophage recruitment.	mir-128	98-105	colony stimulating factor 1 (macrophage)	Mus musculus	220-225
24415783-7	2014	Macrophage colony-stimulating factor (M-CSF) was identified as a target of miR-128, and increased miR-128 levels in epithelial cells due to infection with strain SE2472 significantly decreased the level of cell-secreted M-CSF, leading to impaired M-CSF-mediated macrophage recruitment.	mir-128	98-105	colony stimulating factor 1 (macrophage)	Mus musculus	220-225
24415783-9	2014	Moreover, intragastric delivery of anti-miR-128 antagomir into mice significantly increased M-CSF-mediated macrophage recruitment and suppressed Salmonella infection.	mir-128	40-47	colony stimulating factor 1 (macrophage)	Mus musculus	92-97
24311694-3	2013	miR-128 governs motor activity by suppressing the expression of various ion channels and signaling components of the extracellular signal-regulated kinase ERK2 network that regulate neuronal excitability.	mir-128	0-7	mitogen-activated protein kinase 1	Mus musculus	155-159
24132591-5	2014	At the molecular level, our results demonstrated that miR-128 overexpression could repress expression of PTEN by directly targeting PTEN 3"-untranslated region.	mir-128	54-61	phosphatase and tensin homolog	Homo sapiens	105-109
24132591-5	2014	At the molecular level, our results demonstrated that miR-128 overexpression could repress expression of PTEN by directly targeting PTEN 3"-untranslated region.	mir-128	54-61	phosphatase and tensin homolog	Homo sapiens	132-136
24132591-7	2014	Therefore, our results suggest that miR-128 plays an important role in the proliferation of human osteosarcoma cells by directly regulation of PTEN/AKT signaling.	mir-128	36-43	phosphatase and tensin homolog	Homo sapiens	143-147
24448358-10	2014	Expression of miR-128, -10b, -502-3p and -192 differed between SCC and AC, and miR-128 and -192 - between NLP and NSCLC.	mir-128	14-21	serpin family B member 3	Homo sapiens	63-66
24046120-3	2014	In particular, we examined miR-128 expression, which is thought to target NEK2.	mir-128	27-34	NIMA related kinase 2	Homo sapiens	74-78
24046120-6	2014	RESULTS: MiR-128 inhibited NEK2 expression and cancer cell proliferation via cell cycle arrest.	mir-128	9-16	NIMA related kinase 2	Homo sapiens	27-31
24046120-12	2014	CONCLUSIONS: NEK2 may be an independent prognostic factor for CRC and was regulated by miR-128, a microRNA that was subjected to epigenetic regulation.	mir-128	87-94	NIMA related kinase 2	Homo sapiens	13-17
23835497-5	2013	Target site prediction algorithms indicated that miR-128 binds the 3"-untranslated regions of erythropoietin-producing hepatocellular receptor (Eph)B1 and EphB2 mRNAs.	mir-128	49-56	EPH receptor B1	Homo sapiens	94-150
24055866-0	2013	miR-128 regulates non-myocyte hyperplasia, deposition of extracellular matrix and Islet1 expression during newt cardiac regeneration.	mir-128	0-7	ISL LIM homeobox 1	Homo sapiens	82-88
24055866-9	2013	From these studies we conclude that miR-128 regulates both cardiac hyperplasia and Islet1 expression during newt heart regeneration and that this information could be translated into future mammalian cardiac studies.	mir-128	36-43	ISL LIM homeobox 1	Homo sapiens	83-89
24141048-7	2013	Co-transfection of HspB1 with specific inhibitors of miR-20a or miR-128 prevented the decrease in PDZ-RhoGEF and blocked the neurite growth promoting effects of HspB1.	mir-128	64-71	heat shock protein family B (small) member 1	Rattus norvegicus	19-24
24141048-7	2013	Co-transfection of HspB1 with specific inhibitors of miR-20a or miR-128 prevented the decrease in PDZ-RhoGEF and blocked the neurite growth promoting effects of HspB1.	mir-128	64-71	heat shock protein family B (small) member 1	Rattus norvegicus	161-166
23835497-5	2013	Target site prediction algorithms indicated that miR-128 binds the 3"-untranslated regions of erythropoietin-producing hepatocellular receptor (Eph)B1 and EphB2 mRNAs.	mir-128	49-56	EPH receptor B2	Homo sapiens	155-160
23835497-6	2013	Luciferase reporter assays confirmed that miR-128 binds and regulates EphB1 and EphB2 mRNAs.	mir-128	42-49	EPH receptor B1	Homo sapiens	70-75
23835497-6	2013	Luciferase reporter assays confirmed that miR-128 binds and regulates EphB1 and EphB2 mRNAs.	mir-128	42-49	EPH receptor B2	Homo sapiens	80-85
23835497-7	2013	Overexpression of EphB2 reduced the ability of miR-128 to promote cell-cell adhesion.	mir-128	47-54	EPH receptor B2	Homo sapiens	18-23
23835497-8	2013	The wound-healing assay indicated that miR-128 significantly inhibited cell migration via EphB2.	mir-128	39-46	EPH receptor B2	Homo sapiens	90-95
23835497-9	2013	This study revealed the novel functions of miR-128 in cell-cell adhesion and cell migration in glioma cells through the regulation of EphB2, and identified EphB1 and EphB2 as novel miR-128 targets.	mir-128	43-50	EPH receptor B2	Homo sapiens	134-139
23835497-9	2013	This study revealed the novel functions of miR-128 in cell-cell adhesion and cell migration in glioma cells through the regulation of EphB2, and identified EphB1 and EphB2 as novel miR-128 targets.	mir-128	181-188	EPH receptor B1	Homo sapiens	156-161
23835497-9	2013	This study revealed the novel functions of miR-128 in cell-cell adhesion and cell migration in glioma cells through the regulation of EphB2, and identified EphB1 and EphB2 as novel miR-128 targets.	mir-128	181-188	EPH receptor B2	Homo sapiens	166-171
23482671-13	2013	The inhibition of Bmi1 by NPV-LDE-225 was regulated by induction of miR-128.	mir-128	68-75	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	18-22
23526655-2	2013	We have confirmed that Bax is the target of miR-128 by negative post-transcriptional regulation.	mir-128	44-51	BCL2 associated X, apoptosis regulator	Homo sapiens	23-26
23526655-3	2013	miR-128 and Bax were detected in the breast cancer cell line, MDA-MB-231, which was then transfected with miR-128 MIMIC (precursor of miR-128) or AMO (antisense-miR-128 oligonucleotides).	mir-128	106-113	BCL2 associated X, apoptosis regulator	Homo sapiens	12-15
23526655-3	2013	miR-128 and Bax were detected in the breast cancer cell line, MDA-MB-231, which was then transfected with miR-128 MIMIC (precursor of miR-128) or AMO (antisense-miR-128 oligonucleotides).	mir-128	106-113	BCL2 associated X, apoptosis regulator	Homo sapiens	12-15
22909061-0	2012	Regulation of colony stimulating factor-1 expression and ovarian cancer cell behavior in vitro by miR-128 and miR-152.	mir-128	98-105	colony stimulating factor 1	Homo sapiens	14-41
23492773-10	2013	Pretreatment with PUMA and Bak siRNAs abolished miR-128-induced apoptosis in HCT116 p53+/+ and HCT116 p53-/- cells.	mir-128	48-55	tumor protein p53	Homo sapiens	84-87
23492773-10	2013	Pretreatment with PUMA and Bak siRNAs abolished miR-128-induced apoptosis in HCT116 p53+/+ and HCT116 p53-/- cells.	mir-128	48-55	tumor protein p53	Homo sapiens	102-105
23492773-11	2013	Taken together, we present the first evidence of miR-128 to be a new component joining the p53 network.	mir-128	49-56	tumor protein p53	Homo sapiens	91-94
23492773-12	2013	This study emphasizes that miR-128 is a novel mitochondria-targeted miRNA that can be further evaluated as a chemotherapeutic agent for human cancers as it induces apoptosis irrespective of p53 status.	mir-128	27-34	tumor protein p53	Homo sapiens	190-193
23041308-9	2013	In particular, in HSC overexpressing miR-128, many members of the chemokine family were bound to the Ago2 repression complex.	mir-128	37-44	argonaute RISC catalytic component 2	Homo sapiens	101-105
23567619-12	2013	The inhibition of Bmi-1 by NVP-LDE-225 was regulated by upregulation of miR-128.	mir-128	72-79	Bmi1 polycomb ring finger oncogene	Mus musculus	18-23
22614013-6	2013	Furthermore, we identified that miR-26b and miR-128 affected pituitary tumor cell behavior through regulation of their direct targets, PTEN and BMI1, respectively.	mir-128	44-51	phosphatase and tensin homolog	Homo sapiens	135-139
22614013-6	2013	Furthermore, we identified that miR-26b and miR-128 affected pituitary tumor cell behavior through regulation of their direct targets, PTEN and BMI1, respectively.	mir-128	44-51	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	144-148
22614013-7	2013	In addition, we found that miR-128 through BMI1 direct binding on the PTEN promoter affected PTEN expression levels and AKT activity in the pituitary tumor cells.	mir-128	27-34	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	43-47
22614013-7	2013	In addition, we found that miR-128 through BMI1 direct binding on the PTEN promoter affected PTEN expression levels and AKT activity in the pituitary tumor cells.	mir-128	27-34	phosphatase and tensin homolog	Homo sapiens	70-74
22614013-7	2013	In addition, we found that miR-128 through BMI1 direct binding on the PTEN promoter affected PTEN expression levels and AKT activity in the pituitary tumor cells.	mir-128	27-34	phosphatase and tensin homolog	Homo sapiens	93-97
22614013-7	2013	In addition, we found that miR-128 through BMI1 direct binding on the PTEN promoter affected PTEN expression levels and AKT activity in the pituitary tumor cells.	mir-128	27-34	AKT serine/threonine kinase 1	Homo sapiens	120-123
22614013-11	2013	MiR-26b suppression and miR-128 upregulation could have therapeutic potential in GH-producing pituitary tumor patients.	mir-128	24-31	growth hormone 1	Homo sapiens	81-83
23492773-0	2013	miR-128 exerts pro-apoptotic effect in a p53 transcription-dependent and -independent manner via PUMA-Bak axis.	mir-128	0-7	tumor protein p53	Homo sapiens	41-44
23492773-4	2013	We herein demonstrate that miR-128 positively regulates p53 activity.	mir-128	27-34	tumor protein p53	Homo sapiens	56-59
23492773-6	2013	miR-128 inhibition of SIRT1 led to an increase in acetylated p53 and its transcriptional targets.	mir-128	0-7	tumor protein p53	Homo sapiens	61-64
23492773-8	2013	We further demonstrated that miR-128 augments the antitumor effect of compounds that target the p53 pathway.	mir-128	29-36	tumor protein p53	Homo sapiens	96-99
23492773-9	2013	Furthermore, miR-128 induces apoptosis in wild (WT) p53 as well as in mutant p53-expressing cells in a p53-dependent and -independent manner via induction of PUMA.	mir-128	13-20	tumor protein p53	Homo sapiens	52-55
23492773-9	2013	Furthermore, miR-128 induces apoptosis in wild (WT) p53 as well as in mutant p53-expressing cells in a p53-dependent and -independent manner via induction of PUMA.	mir-128	13-20	tumor protein p53	Homo sapiens	77-80
23492773-9	2013	Furthermore, miR-128 induces apoptosis in wild (WT) p53 as well as in mutant p53-expressing cells in a p53-dependent and -independent manner via induction of PUMA.	mir-128	13-20	tumor protein p53	Homo sapiens	77-80
22922228-3	2012	Here, we demonstrate for the first time that platelet-derived growth factor-B (PDGF-B), a potent angiogenic growth factor involved in GBM development and progression, promotes downregulation of pro-oncogenic (miR-21) and anti-oncogenic (miR-128) miRNAs, as well as upregulation/downregulation of several miRNAs involved in GBM pathology.	mir-128	237-244	platelet derived growth factor subunit B	Homo sapiens	45-77
22922228-3	2012	Here, we demonstrate for the first time that platelet-derived growth factor-B (PDGF-B), a potent angiogenic growth factor involved in GBM development and progression, promotes downregulation of pro-oncogenic (miR-21) and anti-oncogenic (miR-128) miRNAs, as well as upregulation/downregulation of several miRNAs involved in GBM pathology.	mir-128	237-244	platelet derived growth factor subunit B	Homo sapiens	79-85
21953503-0	2011	Reduced miR-128 in breast tumor-initiating cells induces chemotherapeutic resistance via Bmi-1 and ABCC5.	mir-128	8-15	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	89-94
23056445-9	2012	Overexpression of miR-128 alone or with miR-103 and miR-137 significantly reduced endogenous neurofibromin 1 protein levels, while antisense inhibition of these microRNAs enhanced translation of endogenous neurofibromin 1 and reporter in primary cultures of hippocampal neurons.	mir-128	18-25	neurofibromin 1	Homo sapiens	93-108
23056445-9	2012	Overexpression of miR-128 alone or with miR-103 and miR-137 significantly reduced endogenous neurofibromin 1 protein levels, while antisense inhibition of these microRNAs enhanced translation of endogenous neurofibromin 1 and reporter in primary cultures of hippocampal neurons.	mir-128	18-25	neurofibromin 1	Homo sapiens	206-221
22442669-3	2012	Overexpression of miR-128 suppressed p70S6K1 and its downstream signaling molecules such as HIF-1 and VEGF expression, and attenuated cell proliferation, tumor growth and angiogenesis.	mir-128	18-25	vascular endothelial growth factor A	Homo sapiens	102-106
21953503-0	2011	Reduced miR-128 in breast tumor-initiating cells induces chemotherapeutic resistance via Bmi-1 and ABCC5.	mir-128	8-15	ATP binding cassette subfamily C member 5	Homo sapiens	99-104
21953503-2	2011	EXPERIMENTAL DESIGN: Lentivirus-mediated miR-128 transduction was done in BT-ICs, enriched by mammosphere cultures or CD44(+)CD24(-) fluorescence-activated cell sorting.	mir-128	41-48	CD44 molecule (Indian blood group)	Homo sapiens	118-122
21953503-2	2011	EXPERIMENTAL DESIGN: Lentivirus-mediated miR-128 transduction was done in BT-ICs, enriched by mammosphere cultures or CD44(+)CD24(-) fluorescence-activated cell sorting.	mir-128	41-48	CD24 molecule	Homo sapiens	125-129
21953503-5	2011	RESULTS: MiR-128 was significantly reduced in chemoresistant BT-ICs enriched from breast cancer cell lines and primary breast tumors (P &lt; 0.01), accompanied by an overexpression of Bmi-1 and ABCC5, which were identified as targets of miR-128.	mir-128	9-16	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	184-189
21953503-5	2011	RESULTS: MiR-128 was significantly reduced in chemoresistant BT-ICs enriched from breast cancer cell lines and primary breast tumors (P &lt; 0.01), accompanied by an overexpression of Bmi-1 and ABCC5, which were identified as targets of miR-128.	mir-128	9-16	ATP binding cassette subfamily C member 5	Homo sapiens	194-199
21953503-6	2011	Ectopic expression of miR-128 reduced the protein levels of Bmi-1 and ABCC5 in BT-ICs, along with decreased cell viability (P &lt; 0.001) and increased apoptosis (P &lt; 0.001) and DNA damage (P &lt; 0.001) in the presence of doxorubicin.	mir-128	22-29	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	60-65
21953503-6	2011	Ectopic expression of miR-128 reduced the protein levels of Bmi-1 and ABCC5 in BT-ICs, along with decreased cell viability (P &lt; 0.001) and increased apoptosis (P &lt; 0.001) and DNA damage (P &lt; 0.001) in the presence of doxorubicin.	mir-128	22-29	ATP binding cassette subfamily C member 5	Homo sapiens	70-75
21953503-8	2011	CONCLUSIONS: Reduction in miR-128 leading to Bmi-1 and ABCC5 overexpression is a stem cell-like feature of BT-ICs, which contributes to chemotherapeutic resistance in breast cancers.	mir-128	26-33	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	45-50
21953503-8	2011	CONCLUSIONS: Reduction in miR-128 leading to Bmi-1 and ABCC5 overexpression is a stem cell-like feature of BT-ICs, which contributes to chemotherapeutic resistance in breast cancers.	mir-128	26-33	ATP binding cassette subfamily C member 5	Homo sapiens	55-60
21596314-4	2011	miR-128 represses NMD by targeting the RNA helicase UPF1 and the exon-junction complex core component MLN51.	mir-128	0-7	UPF1 RNA helicase and ATPase	Homo sapiens	52-56
19010882-5	2008	miR-128 caused a striking decrease in expression of the Bmi-1 oncogene, by direct regulation of the Bmi-1 mRNA 3"-untranslated region, through a single miR-128 binding site.	mir-128	0-7	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	56-61
19010882-5	2008	miR-128 caused a striking decrease in expression of the Bmi-1 oncogene, by direct regulation of the Bmi-1 mRNA 3"-untranslated region, through a single miR-128 binding site.	mir-128	0-7	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	100-105
19010882-5	2008	miR-128 caused a striking decrease in expression of the Bmi-1 oncogene, by direct regulation of the Bmi-1 mRNA 3"-untranslated region, through a single miR-128 binding site.	mir-128	152-159	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	56-61
19010882-5	2008	miR-128 caused a striking decrease in expression of the Bmi-1 oncogene, by direct regulation of the Bmi-1 mRNA 3"-untranslated region, through a single miR-128 binding site.	mir-128	152-159	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	100-105
19010882-8	2008	We found that miR-128 expression caused a decrease in histone methylation (H3K27me(3)) and Akt phosphorylation, and up-regulation of p21(CIP1) levels, consistent with Bmi-1 down-regulation.	mir-128	14-21	H3 histone pseudogene 16	Homo sapiens	133-136
19010882-8	2008	We found that miR-128 expression caused a decrease in histone methylation (H3K27me(3)) and Akt phosphorylation, and up-regulation of p21(CIP1) levels, consistent with Bmi-1 down-regulation.	mir-128	14-21	cyclin dependent kinase inhibitor 1A	Homo sapiens	137-141
19010882-8	2008	We found that miR-128 expression caused a decrease in histone methylation (H3K27me(3)) and Akt phosphorylation, and up-regulation of p21(CIP1) levels, consistent with Bmi-1 down-regulation.	mir-128	14-21	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	167-172
19010882-10	2008	This showed that miR-128 specifically blocked glioma self-renewal consistent with Bmi-1 down-regulation.	mir-128	17-24	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	82-87
21596314-4	2011	miR-128 represses NMD by targeting the RNA helicase UPF1 and the exon-junction complex core component MLN51.	mir-128	0-7	CASC3 exon junction complex subunit	Homo sapiens	102-107
21372826-10	2011	The up-regulation of miR-128 by Rh2 was further verified in human U251, T98MG and A172 cells using quantitative real-time PCR.	mir-128	21-28	Rh associated glycoprotein	Homo sapiens	32-35
21372826-11	2011	In U251 cells, transfection of a miR-128 inhibitor (50 nmol/L) prevented the overexpression of miR-128 by Rh2, and significantly blunted Rh2-induced cytotoxicity, apoptosis, caspase 3 activation, transcriptional activation of E2F3a, a miR-128 target gene, as well as E2F3a protein expression.	mir-128	33-40	Rh associated glycoprotein	Homo sapiens	106-109
21372826-11	2011	In U251 cells, transfection of a miR-128 inhibitor (50 nmol/L) prevented the overexpression of miR-128 by Rh2, and significantly blunted Rh2-induced cytotoxicity, apoptosis, caspase 3 activation, transcriptional activation of E2F3a, a miR-128 target gene, as well as E2F3a protein expression.	mir-128	33-40	Rh associated glycoprotein	Homo sapiens	137-140
21372826-11	2011	In U251 cells, transfection of a miR-128 inhibitor (50 nmol/L) prevented the overexpression of miR-128 by Rh2, and significantly blunted Rh2-induced cytotoxicity, apoptosis, caspase 3 activation, transcriptional activation of E2F3a, a miR-128 target gene, as well as E2F3a protein expression.	mir-128	33-40	caspase 3	Homo sapiens	174-183
21372826-11	2011	In U251 cells, transfection of a miR-128 inhibitor (50 nmol/L) prevented the overexpression of miR-128 by Rh2, and significantly blunted Rh2-induced cytotoxicity, apoptosis, caspase 3 activation, transcriptional activation of E2F3a, a miR-128 target gene, as well as E2F3a protein expression.	mir-128	95-102	Rh associated glycoprotein	Homo sapiens	106-109
19713529-0	2009	MiR-128 up-regulation inhibits Reelin and DCX expression and reduces neuroblastoma cell motility and invasiveness.	mir-128	0-7	reelin	Homo sapiens	31-37
19713529-0	2009	MiR-128 up-regulation inhibits Reelin and DCX expression and reduces neuroblastoma cell motility and invasiveness.	mir-128	0-7	doublecortin	Homo sapiens	42-45
19713529-3	2009	We studied miR-128, a brain-enriched microRNA, in retinoic acid-differentiated neuroblastoma cells, and we found that this microRNA is up-regulated in treated cells, where it down-modulates the expression of two proteins involved in the migratory potential of neural cells: Reelin and DCX.	mir-128	11-18	reelin	Homo sapiens	274-280
19713529-3	2009	We studied miR-128, a brain-enriched microRNA, in retinoic acid-differentiated neuroblastoma cells, and we found that this microRNA is up-regulated in treated cells, where it down-modulates the expression of two proteins involved in the migratory potential of neural cells: Reelin and DCX.	mir-128	11-18	doublecortin	Homo sapiens	285-288
19713529-4	2009	Consistently, miR-128 ectopic overexpression suppressed Reelin and DCX, whereas the LNA antisense-mediated miR-128 knockdown caused the two proteins to increase.	mir-128	14-21	reelin	Homo sapiens	56-62
19713529-4	2009	Consistently, miR-128 ectopic overexpression suppressed Reelin and DCX, whereas the LNA antisense-mediated miR-128 knockdown caused the two proteins to increase.	mir-128	14-21	doublecortin	Homo sapiens	67-70
18381601-6	2008	In this study, we show that Tat deregulates expression levels of selected microRNAs, including the neuronal mir-128, in primary cortical neurons.	mir-128	108-115	tyrosine aminotransferase	Homo sapiens	28-31