PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
19225880-7	2009	CONCLUSION: Activation of calpains, cathepsin-b, caspase-3, and granzyme-b correlated with either apoptotic or necrotic cell deaths in cresyl violet staining.	cresyl violet	135-148	cathepsin B	Rattus norvegicus	36-47
19225880-7	2009	CONCLUSION: Activation of calpains, cathepsin-b, caspase-3, and granzyme-b correlated with either apoptotic or necrotic cell deaths in cresyl violet staining.	cresyl violet	135-148	caspase 3	Rattus norvegicus	49-58
19225880-7	2009	CONCLUSION: Activation of calpains, cathepsin-b, caspase-3, and granzyme-b correlated with either apoptotic or necrotic cell deaths in cresyl violet staining.	cresyl violet	135-148	granzyme B	Rattus norvegicus	64-74
18640243-2	2008	Neuronal cell counts and density measures from brain sections stained with Cresyl Violet revealed that exposure of P21 mice to 60 min of HI resulted in extensive damage to the ipsilateral cornu ammonis 1 (CA1) region of the hippocampus (40% cell loss) and striatum (30% cell loss) 7 days later.	cresyl violet	75-88	cyclin-dependent kinase inhibitor 1A (P21)	Mus musculus	115-118
18938164-2	2009	When histologically evaluated at 7 and 28 days after the forebrain ischemia (DAI) by staining with cresyl violet and Fluoro-Jade, the hippocampal CA1 region was most prominently damaged.	cresyl violet	99-112	carbonic anhydrase 1	Rattus norvegicus	146-149
18442442-7	2008	Our morphometric study of flat-mount preparations stained with cresyl violet or retrograde labeling with a neuro-tracer and Western blot analysis of NF showed that inhibition of ERK phosphorylation led to acceleration of ET-1-induced RGC death and optic nerve damage.	cresyl violet	63-76	mitogen-activated protein kinase 1	Homo sapiens	178-181
17919780-8	2008	In the histochemical study, the cresyl violet positive neurons in the Pep-1-SOD1-treated group were abundantly detected in the hippocampal CA1 region 5 days after ischemia/reperfusion.	cresyl violet	32-45	superoxide dismutase 1	Homo sapiens	76-80
17919780-8	2008	In the histochemical study, the cresyl violet positive neurons in the Pep-1-SOD1-treated group were abundantly detected in the hippocampal CA1 region 5 days after ischemia/reperfusion.	cresyl violet	32-45	carbonic anhydrase 1	Homo sapiens	139-142
18387598-7	2008	Cresyl violet stain demonstrated massive loss of neurons in the ipsilateral septal hippocampus in the CA3 and CA1 regions associated with atrophy.	cresyl violet	0-13	carbonic anhydrase 3	Mus musculus	102-105
18387598-7	2008	Cresyl violet stain demonstrated massive loss of neurons in the ipsilateral septal hippocampus in the CA3 and CA1 regions associated with atrophy.	cresyl violet	0-13	carbonic anhydrase 1	Mus musculus	110-113
17562385-2	2007	We found that intracerebroventricular administration of kainic acid shows significant neuronal damage on the hippocampal CA3 region in the brain slices stained with cresyl violet.	cresyl violet	165-178	carbonic anhydrase 3	Homo sapiens	121-124
17882016-6	2007	Histologic assessment with cresyl violet staining revealed that the number of surviving neurons in the injured spinal cord was significantly increased in G-CSF-treated mice.	cresyl violet	27-40	colony stimulating factor 3 (granulocyte)	Mus musculus	154-159
16867245-4	2006	In roasted licorice-treated animals, approximately 66%-71% of CA1 pyramidal cells in the ischemic hippocampus were stained with cresyl violet compared to the control group.	cresyl violet	128-141	carbonic anhydrase 1	Rattus norvegicus	62-65
17064355-9	2006	Photomicrographs of cresyl violet-stained brain sections showed that E2 reduced CA1 neuron loss after 5 days of reperfusion, which was abolished by ICI 182,780 and LY 294,002.	cresyl violet	20-33	carbonic anhydrase 1	Rattus norvegicus	80-83
17048522-7	2006	Cystatin delivered into MCF-10A neoT cells by PLGA NPs effectively inhibited intracellular proteolytic activity of cathepsin B, as detected by specific fluorogenic substrate Z-Arg2 cresyl violet.	cresyl violet	181-194	cathepsin B	Homo sapiens	115-126
15094476-7	2004	In contrast to earlier studies, where t-PA exacerbated injury, t-PA treatment did not increase, but attenuated disseminate neuronal death in the striatum, as shown by cresyl violet, TUNEL and activated caspase-3 staining 1 and 3 days after the ischemic event (p < 0.05).	cresyl violet	167-180	plasminogen activator, tissue	Mus musculus	63-67
16329123-4	2006	VEGF increased neurite outgrowth, measured using a colorimetric assay for cresyl violet staining of neuronal processes, with half-maximal enhancement at 10 ng/mL and maximal, approximately 60% enhancement at 30-100 ng/mL.	cresyl violet	74-87	vascular endothelial growth factor A	Rattus norvegicus	0-4
15530858-7	2004	Cystatin, released from the NPs, effectively inhibited cathepsin B activity, as detected by degradation of specific Z-Arg-Arg cresyl violet substrate.	cresyl violet	126-139	cystatin C	Gallus gallus	0-8
15530858-7	2004	Cystatin, released from the NPs, effectively inhibited cathepsin B activity, as detected by degradation of specific Z-Arg-Arg cresyl violet substrate.	cresyl violet	126-139	cathepsin B	Homo sapiens	55-66
16315320-4	2006	Intracellular cathepsin B activity detected by degradation of Z-Arg-Arg cresyl violet substrate was co-localized with the products of DQ-collagen IV degradation in the perinuclear region and in the capillary-like tubular structures.	cresyl violet	72-85	cathepsin B	Homo sapiens	14-25
16595891-2	2006	In the vehicle-treated group, the percentage of cresyl violet positive pyramidal cells in the CA1 region was about 11.4% compared to the sham-operated group 4 d after ischemic insult.	cresyl violet	48-61	carbonic anhydrase 1	Homo sapiens	94-97
16595891-4	2006	In the BE-treated groups, about 60-75% of CA1 pyramidal cells were stained with cresyl violet 4 d after ischemic insult.	cresyl violet	80-93	carbonic anhydrase 1	Homo sapiens	42-45
15861457-4	2005	Molecular fingerprint comparison of cresyl violet-stained CA1 and CA3 pyramidal neurons microaspirated from the hippocampus of normal control subjects indicated significant differences in relative expression levels for approximately 16% (20 of 125) genes evaluated on the custom-designed cDNA array platform.	cresyl violet	36-49	carbonic anhydrase 1	Homo sapiens	58-61
14643774-12	2003	Instead, in Cresyl Violet-stained preparations, the highest density of neurons was found in untreated transgenic mice suggesting a favorable effect of trkB overexpression on the survival of neurons in the hippocampus.	cresyl violet	12-25	neurotrophic tyrosine kinase, receptor, type 2	Mus musculus	151-155
15501596-9	2004	The magnitude of cell death, using TdT-mediated dUTP nick end labeling and Cresyl Violet to quantify the incidence of apoptotic and necrotic cells, was region dependent (CA1>CA2/3>dentate gyrus) and persisted for at least 5 days following insult.	cresyl violet	75-88	carbonic anhydrase 1	Homo sapiens	170-173
15207322-7	2004	However, in the Org2766 (analog of ACTH)-treated group, 57.8% neurons compared with the sham-operated group were stained with Cresyl Violet 4 and 7 days after ischemic insult.	cresyl violet	126-139	proopiomelanocortin	Homo sapiens	35-39
14628683-1	2003	Using a bisubstituted caspase-3 target sequence: aspartate-glutamate-valine-aspartate, (z-DEVD)2 peptide derivative of the fluorophore, cresyl violet, we have obtained a cell permeant, fluorogenic, caspase substrate capable of detecting the site-specific presence of functionally active, caspase-3 and caspase-7 up-regulation within intact apoptotic cells.	cresyl violet	136-149	caspase 3	Homo sapiens	22-31
14628683-1	2003	Using a bisubstituted caspase-3 target sequence: aspartate-glutamate-valine-aspartate, (z-DEVD)2 peptide derivative of the fluorophore, cresyl violet, we have obtained a cell permeant, fluorogenic, caspase substrate capable of detecting the site-specific presence of functionally active, caspase-3 and caspase-7 up-regulation within intact apoptotic cells.	cresyl violet	136-149	caspase 3	Homo sapiens	288-297
14628683-1	2003	Using a bisubstituted caspase-3 target sequence: aspartate-glutamate-valine-aspartate, (z-DEVD)2 peptide derivative of the fluorophore, cresyl violet, we have obtained a cell permeant, fluorogenic, caspase substrate capable of detecting the site-specific presence of functionally active, caspase-3 and caspase-7 up-regulation within intact apoptotic cells.	cresyl violet	136-149	caspase 7	Homo sapiens	302-311
14579742-6	2003	Fluorescent cresyl violet was generated by CD26/DPPIV-transfected Jurkat cells but not by wild-type Jurkat cells with a Km of 3.7 microM.	cresyl violet	12-25	dipeptidyl peptidase 4	Homo sapiens	43-47
14579742-6	2003	Fluorescent cresyl violet was generated by CD26/DPPIV-transfected Jurkat cells but not by wild-type Jurkat cells with a Km of 3.7 microM.	cresyl violet	12-25	dipeptidyl peptidase 4	Homo sapiens	48-53
12932851-2	2003	Seven days after transient global cerebral ischemia induced by four-vessel occlusion method, most of the pyramidal cells in the hippocampal CA1 subfield underwent disruption and pyknosis as detected by cresyl violet staining.	cresyl violet	202-215	carbonic anhydrase 1	Rattus norvegicus	140-143
11098108-2	2000	injection of kainate resulted in severe losses of neuronal cells stained by Cresyl Violet in the CA1 and CA3 pyramidal layers of the murine hippocampus within two weeks, without affecting those in the dentate granule layer up to six weeks.	cresyl violet	76-89	carbonic anhydrase 1	Mus musculus	97-100
12098641-4	2002	However, cresyl violet staining revealed approximately 20% neuronal loss following ethanol administration in CA1 and CA2 fields (P<0.01 and P<0.05, respectively), but not in DG.	cresyl violet	9-22	carbonic anhydrase 1	Mus musculus	109-112
12098641-4	2002	However, cresyl violet staining revealed approximately 20% neuronal loss following ethanol administration in CA1 and CA2 fields (P<0.01 and P<0.05, respectively), but not in DG.	cresyl violet	9-22	carbonic anhydrase 2	Mus musculus	117-120
12067235-10	2002	Finally, intraventricular administration of the caspase-2-like inhibitor (VDVAD-FMK) 30 min before induction of ischemia decreased the number of CA1 neurons staining positively for DNA damage (Klenow-labeling assay) and increased the number of healthy-appearing CA1 neurons (cresyl violet) compared with vehicle-treated controls.	cresyl violet	275-288	caspase 2	Rattus norvegicus	48-57
12067235-10	2002	Finally, intraventricular administration of the caspase-2-like inhibitor (VDVAD-FMK) 30 min before induction of ischemia decreased the number of CA1 neurons staining positively for DNA damage (Klenow-labeling assay) and increased the number of healthy-appearing CA1 neurons (cresyl violet) compared with vehicle-treated controls.	cresyl violet	275-288	carbonic anhydrase 1	Rattus norvegicus	145-148
11598495-7	2001	The activation of CREB phosphorylation in the OLGs was accompanied by expression of antiapoptotic protein bcl-2, normal staining with cresyl violet, and negative TUNEL (terminal deoxynucleotidyl transferase-mediated deoxyuridine triphosphate nick-end labeling) staining.	cresyl violet	134-147	cAMP responsive element binding protein 1	Rattus norvegicus	18-22
10686068-7	2000	Cresyl violet staining revealed CA1 pyramidal neurons to be maintained intact until 14 day of recirculation, at which time CREB phosphorylation has returned to the control level.	cresyl violet	0-13	carbonic anhydrase 1	Rattus norvegicus	32-35
11172745-2	2001	A direct injection of lipopolysaccharide (LPS) into the striatum of gerbils induced lectin-positive macrophage parenchymal invasion, minimal local microglial staining but extensive neurodegeneration (cresyl violet and silver staining) when evaluated 4 days later.	cresyl violet	200-213	toll-like receptor 4	Mus musculus	42-45
11063989-7	2000	Neuronal cell loss in the hippocampal CA1 was evaluated by cresyl-violet staining of sections 7 days later.	cresyl violet	59-72	carbonic anhydrase 1	Rattus norvegicus	38-41
9763473-6	1998	Neuronal damage, as assessed by cresyl violet staining and DNA fragmentation analysis, was significantly reduced in the hippocampal CA1 region, cortex, striatum, and thalamus in SOD1 Tg rats at 3 d, as compared with the non-Tg littermates.	cresyl violet	32-45	carbonic anhydrase 1	Rattus norvegicus	132-135
10082855-5	1999	On day 7, CA1 neuronal damage was examined in the hippocampal slices stained with cresyl violet.	cresyl violet	82-95	carbonic anhydrase 1	Homo sapiens	10-13
9763473-6	1998	Neuronal damage, as assessed by cresyl violet staining and DNA fragmentation analysis, was significantly reduced in the hippocampal CA1 region, cortex, striatum, and thalamus in SOD1 Tg rats at 3 d, as compared with the non-Tg littermates.	cresyl violet	32-45	superoxide dismutase 1	Rattus norvegicus	178-182
9784284-3	1998	Cresyl violet staining of hippocampal sections revealed that as early as 3 h after injection of 300 ng OKA a notable neurodegeneration occurred in the CA1 subfield, the dentate gyrus, and the hilus, particularly in the former.	cresyl violet	0-13	carbonic anhydrase 1	Rattus norvegicus	151-154
9225739-2	1997	Cell counts of CR-immunolabeled interneurons indicated that these cells are spared by the neurotoxicant-induced degeneration, characterized by a generalized neuronal loss, as shown by quantitative analysis after cresyl violet staining.	cresyl violet	212-225	calbindin 2	Rattus norvegicus	15-17
8772431-12	1996	Sections that were emulsion-dipped and counterstained with cresyl violet revealed EAAC1 labeling localized exclusively over neuronal cell bodies, including some nonglutamatergic neurons such as spinal cord ventral horn cells.	cresyl violet	59-72	solute carrier family 1 member 1	Rattus norvegicus	82-87
18966619-1	1996	The use of Cresyl Violet and sodium dodecyl sulphate for the kinetic fluorimetric determination of lysozyme from dynamic fluorescence measurements at a long wavelength in the range near 600 nm was explored.	cresyl violet	11-24	lysozyme	Homo sapiens	99-107
8654529-4	1996	administration with severe neuronal loss, which was significant in CAI and CA3, as revealed by cell counts after cresyl violet staining.	cresyl violet	113-126	carbonic anhydrase 3	Rattus norvegicus	75-78
8731433-4	1996	The type of cells expressing IL-1 beta mRNA was likely glia because their nuclei were densely stained by Cresyl violet and were relatively small.	cresyl violet	105-118	interleukin 1 beta	Rattus norvegicus	29-38
8742133-8	1995	Cresyl violet staining shows no hippocampal cell death after central fluid percussion injury or on the side contralateral to lateral fluid percussion injury but on the ipsilateral side cell death was identified in hippocampal area CA3.	cresyl violet	0-13	carbonic anhydrase 3	Rattus norvegicus	231-234
21551913-5	1996	Histologic analysis of cresyl violet-stained paraffin sections was performed at 72 h. bcl-2 protein was expressed in CA1 neurons, a region that is injured, yet survives after seizures.	cresyl violet	23-36	BCL2, apoptosis regulator	Rattus norvegicus	86-91
21551913-5	1996	Histologic analysis of cresyl violet-stained paraffin sections was performed at 72 h. bcl-2 protein was expressed in CA1 neurons, a region that is injured, yet survives after seizures.	cresyl violet	23-36	carbonic anhydrase 1	Rattus norvegicus	117-120
8255177-6	1993	In these regions, IL-1 beta mRNA was expressed mainly in the glial cells, which were densely stained by Cresyl violet and did not contain glial fibrillary acidic protein.	cresyl violet	104-117	interleukin 1 beta	Rattus norvegicus	18-27
1407719-4	1992	Counterstaining with Cresyl violet, a general stain of cells, revealed that the increase of CREB positive glial cells was congruent with the increase of the number of glial cells.	cresyl violet	21-34	cAMP responsive element binding protein 1	Rattus norvegicus	92-96
1977414-3	1990	The analysis of sections stained with glucocorticoid receptor antibody and cresyl violet showed that around 90% of the entire striatal neuronal population contained glucocorticoid receptor immunoreactivity.	cresyl violet	75-88	nuclear receptor subfamily 3, group C, member 1	Rattus norvegicus	165-188
1563457-2	1992	In this study, the Ch4 of the nbM was examined using the indirect immunoperoxidase method with a monoclonal antibody to acetylcholinesterase (AChE) counterstained with cresyl violet.	cresyl violet	168-181	acetylcholinesterase (Cartwright blood group)	Homo sapiens	120-140
1563457-2	1992	In this study, the Ch4 of the nbM was examined using the indirect immunoperoxidase method with a monoclonal antibody to acetylcholinesterase (AChE) counterstained with cresyl violet.	cresyl violet	168-181	acetylcholinesterase (Cartwright blood group)	Homo sapiens	142-146
1713525-9	1991	The analysis of adjacent sections stained with Cresyl violet showed a pattern of age-related changes (decrease of neuronal profiles in CA1 field pyramidal layer and dentate gyrus granular layer, and no change in the central amygdaloid nucleus of aged rats) which paralleled the observed changes in GR-IR in the same areas.	cresyl violet	47-60	carbonic anhydrase 1	Rattus norvegicus	135-138
33686675-8	2021	Cresyl violet (CV) staining showed that crocin increased the number of cresyl violet-positive cells in the CA1 region of the hippocampus compared with the Abeta group.	cresyl violet	71-84	carbonic anhydrase 1	Rattus norvegicus	107-110
35142986-9	2022	Moreover, FGF21 overexpressed rats treated with Rapamycin revamped the neuronal density as confirmed by histochemical, cresyl violet and immunofluorescence analysis.	cresyl violet	119-132	fibroblast growth factor 21	Rattus norvegicus	10-15
32006563-6	2020	Cresyl violet staining was used to show the survival of hippocampal CA1 pyramidal neurons.	cresyl violet	0-13	carbonic anhydrase 1	Homo sapiens	68-71
33607171-7	2021	RESULTS: Experimental results are presented for automatic counts using deep learning-based and hand-crafted algorithms for sections immunostained for neurons (Neu-N) or microglial cells (Iba-1) with cresyl violet counterstain.	cresyl violet	199-212	allograft inflammatory factor 1	Homo sapiens	187-192
28911222-3	2017	By conjugating pantothenic acid with cresyl violet, herein we develop a new fluorescence probe CV-PA for the assay of pantetheinase.	cresyl violet	37-50	vanin 1	Homo sapiens	118-131
31908577-7	2020	Cresyl violet staining showed that pilocarpine-induced SE resulted in cell death in the CA1 and CA3 subfields of the hippocampus from 3 day after SE.	cresyl violet	0-13	carbonic anhydrase 1	Mus musculus	88-91
31908577-7	2020	Cresyl violet staining showed that pilocarpine-induced SE resulted in cell death in the CA1 and CA3 subfields of the hippocampus from 3 day after SE.	cresyl violet	0-13	carbonic anhydrase 3	Mus musculus	96-99
31539160-5	2019	The survival of hippocampal CA1 pyramidal cells after administration of the three drugs was examined by cresyl violet staining.	cresyl violet	104-117	carbonic anhydrase 1	Rattus norvegicus	28-31
29657393-4	2018	Furthermore, cresyl violet staining was performed to analyze hippocampus formation mainly in CA1 and CA3 area.	cresyl violet	13-26	carbonic anhydrase 1	Rattus norvegicus	93-96
27923634-7	2017	Cresyl violet staining was used to examine the survival of hippocampal CA1 pyramidal neurons.	cresyl violet	0-13	carbonic anhydrase 1	Rattus norvegicus	71-74
28377303-5	2017	Similar to MB, these analogues also are specific MAO-A inhibitors with cresyl violet (IC50=0.0037muM), Nile blue (IC50=0.0077muM) and 1,9-dimethyl methylene blue (IC50=0.018muM) exhibiting higher potency inhibition compared to MB (IC50=0.07muM).	cresyl violet	71-84	monoamine oxidase A	Homo sapiens	49-54
28377303-8	2017	Benzophenoxazines such as cresyl violet and Nile blue are, similar to phenothiazines (e.g. MB), representative of high potency MAO-A inhibitors with a potential risk of ST.	cresyl violet	26-39	monoamine oxidase A	Homo sapiens	127-132
27488855-7	2017	Cresyl violet staining was carried out to examine the neuronal death of hippocampal CA1 region.	cresyl violet	0-13	carbonic anhydrase 1	Rattus norvegicus	84-87
28017679-5	2017	Cresyl Violet staining was used to examine the survival of hippocampal CA1 pyramidal neurons.	cresyl violet	0-13	carbonic anhydrase 1	Rattus norvegicus	71-74
28487986-7	2017	Administration of Tat-SUMO-1 significantly improved Tarlov scores 24 h after ischemia/reperfusion and the number of cresyl violet positive neurons was significantly increased in the ventral horn of the spinal cord compared with the vehicle-treated group.	cresyl violet	116-129	small ubiquitin-related modifier 1	Oryctolagus cuniculus	22-28
28487994-3	2017	Transient cerebral ischemia was performed for 5 min in gerbils, and neuronal death in the CA1 region following transient cerebral ischemia was confirmed using cresyl violet staining, neuronal nuclear antigen immunohistochemistry and Fluoro-Jade B histofluorescent staining.	cresyl violet	159-172	carbonic anhydrase 1	Homo sapiens	90-93
28491492-9	2017	There were subfield specific associations between hippocampal radial distance and tau in CA2, and cresyl violet neuronal counts in CA1 and subiculum.	cresyl violet	98-111	carbonic anhydrase 1	Homo sapiens	131-134
27420046-6	2016	Cresyl violet staining validated that these compounds exerted neuroprotective effects against the cerebral I/R-induced damage to hippocampal CA1 neurons.	cresyl violet	0-13	carbonic anhydrase 1	Homo sapiens	141-144
26434561-8	2016	Histopathological examination of hippocampal CA1 region was performed using cresyl violet staining.	cresyl violet	76-89	carbonic anhydrase 1	Mus musculus	45-48
26433378-6	2016	We first found that MB significantly improved ischemic neuronal survival in the hippocampal CA1 region as measured by cresyl violet staining as well as NeuN staining.	cresyl violet	118-131	carbonic anhydrase 1	Rattus norvegicus	92-95
27213933-6	2016	In addition, Cresyl violet staining indicated a significantly elevated number of cells with the degenerative morphological features in all hippocampal subregions (CA1-CA3, hilus, and DG).	cresyl violet	13-26	carbonic anhydrase 1	Rattus norvegicus	163-166
27213933-6	2016	In addition, Cresyl violet staining indicated a significantly elevated number of cells with the degenerative morphological features in all hippocampal subregions (CA1-CA3, hilus, and DG).	cresyl violet	13-26	carbonic anhydrase 3	Rattus norvegicus	167-170
25687432-4	2016	Cresyl violet staining was used to examine the survival of hippocampal CA1 pyramidal neurons.	cresyl violet	0-13	carbonic anhydrase 1	Rattus norvegicus	71-74
26344664-8	2016	Histological examination of the hippocampus CA1 segment, the most vulnerable segment of the cerebrum, demonstrated extensive damage in the cresyl violet staining, as well as in the Fluoro-Jade B staining and in the Iba-1 staining, indicating recruitment of microglia after the hypoxic-ischaemic event.	cresyl violet	139-152	carbonic anhydrase 1	Rattus norvegicus	44-47
26794251-5	2016	Cresyl violet staining was used to examine the survival of hippocampal CA1 pyramidal neurons.	cresyl violet	0-13	carbonic anhydrase 1	Rattus norvegicus	71-74
26176215-1	2015	A new sensitive fluorescent probe with long analytical wavelengths for gamma-glutamyl transpeptidase (GGT) assay has been developed by incorporating the gamma-glutamyl group as a recognition unit into the fluorophore of cresyl violet (CV).	cresyl violet	220-233	inactive glutathione hydrolase 2	Homo sapiens	71-100
27051437-5	2016	At P28 and P56, cresyl violet-positive cells were observed in the granule cell layer, as well as in the polymorphic layer.	cresyl violet	16-29	glutathione S-transferase omega 1	Mus musculus	3-6
27051437-5	2016	At P28 and P56, cresyl violet-positive cells were observed in the granule cell layer, as well as in the polymorphic layer.	cresyl violet	16-29	interferon-induced protein with tetratricopeptide repeats 1	Mus musculus	11-14
26047734-5	2015	To investigate the changes caused by the inhibition of IRS-1-mediated insulin signaling in the brain, we performed Cresyl Violet staining and immunochemical analysis.	cresyl violet	115-128	insulin receptor substrate 1	Mus musculus	55-60
26176215-1	2015	A new sensitive fluorescent probe with long analytical wavelengths for gamma-glutamyl transpeptidase (GGT) assay has been developed by incorporating the gamma-glutamyl group as a recognition unit into the fluorophore of cresyl violet (CV).	cresyl violet	220-233	inactive glutathione hydrolase 2	Homo sapiens	102-105
23625556-7	2013	RESULTS: Mutant ataxin-3 caused an evolving neuronal dysfunction (loss of DARPP-32 staining) leading to neurodegeneration (cresyl violet and neuronal nuclei staining) associated with increased number of mutant ataxin-3 inclusions in the basal ganglia.	cresyl violet	123-136	ataxin 3	Mus musculus	16-24
25440530-8	2014	once daily) significantly reduced the densities of cresyl violet positive neurons in the sub-areas CA1, CA3 and the dentate gyrus, but not in the CA2 area.	cresyl violet	51-64	carbonic anhydrase 1	Rattus norvegicus	99-102
25440530-8	2014	once daily) significantly reduced the densities of cresyl violet positive neurons in the sub-areas CA1, CA3 and the dentate gyrus, but not in the CA2 area.	cresyl violet	51-64	carbonic anhydrase 3	Rattus norvegicus	104-107
24464837-3	2014	The cresyl violet staining results indicated that exogenous TK protected the rat hippocampal CA1 neurons against cerebral ischemia/reperfusion (I/R) injury.	cresyl violet	4-17	kallikrein 1-related peptidase C12	Rattus norvegicus	60-62
25655275-11	2015	GH decreased cresyl violet positive motoneuronal loss with strong Bcl-2 and less GFAP immunoreactivity.	cresyl violet	13-26	growth hormone	Mus musculus	0-2
21576037-7	2011	Cresyl-violet brain staining confirmed the locations of transections in the CA3 region.	cresyl violet	0-13	carbonic anhydrase 3	Rattus norvegicus	76-79
22907804-5	2012	Cresyl violet staining showed that hypoxia-ischemia induced progressive cell death in the hippocampi of bis(+/+) mice.	cresyl violet	0-13	BCL2-associated athanogene 3	Mus musculus	104-107
22354094-3	2012	The nano-patterned SERS substrate, with excellent control and reproducibility of plasmon-polaritons generation, shows very efficient enhanced Raman signal in the presence of intrinsically fluorescent molecules such as cresyl violet, rhodamine, and green fluorescent protein.	cresyl violet	218-231	seryl-tRNA synthetase 2, mitochondrial	Homo sapiens	19-23
21849914-4	2011	We also observed an increased number of surviving CA3 neurons in 0.1 and 1 microM concentrations of CoQ10-treated groups using cresyl violet staining.	cresyl violet	127-140	carbonic anhydrase 3	Rattus norvegicus	50-53
21786801-6	2011	All the coumarin-, cresyl violet- and rhodamine-based fluorogenic probes turned out to be good substrates for most GSTs, especially for GSTA(1-1), in terms of strong fluorescence increases (71-1200-fold), high k(cat)/K(m) values (10(4)-10(7) M(-1) s(-1)) and significant rate enhancements (10(6)-10(9)-fold).	cresyl violet	19-32	hematopoietic prostaglandin D synthase	Homo sapiens	115-119
21786801-6	2011	All the coumarin-, cresyl violet- and rhodamine-based fluorogenic probes turned out to be good substrates for most GSTs, especially for GSTA(1-1), in terms of strong fluorescence increases (71-1200-fold), high k(cat)/K(m) values (10(4)-10(7) M(-1) s(-1)) and significant rate enhancements (10(6)-10(9)-fold).	cresyl violet	19-32	glutathione S-transferase alpha 1	Homo sapiens	136-144
23178948-8	2013	In addition, immunohistochemical data collected using a tyrosine hydroxylase antibody and cresyl violet staining demonstrated that PEP-1-rpS3 markedly protected DA cells in the SN against MPTP-induced oxidative stress.	cresyl violet	90-103	CNDP dipeptidase 2 (metallopeptidase M20 family)	Mus musculus	131-136
23178948-8	2013	In addition, immunohistochemical data collected using a tyrosine hydroxylase antibody and cresyl violet staining demonstrated that PEP-1-rpS3 markedly protected DA cells in the SN against MPTP-induced oxidative stress.	cresyl violet	90-103	ribosomal protein S3	Homo sapiens	137-141
22285595-5	2012	The pyramidal cells within the CA1 field were stained with cresyl violet.	cresyl violet	59-72	carbonic anhydrase 1	Rattus norvegicus	31-34
22088537-6	2011	The levels of protein expression and phospho-JNK1/2 were detected by Western blotting and the survival hippocampus neurons in CA1 zone were observed by cresyl violet staining.	cresyl violet	152-165	carbonic anhydrase 1	Rattus norvegicus	126-129
21756906-9	2011	Administration of IGF-I decreased seizure severity, hippocampal neurogenesis, and protected against neurodegeneration at the cellular level as assessed by FluoroJade B and Cresyl violet staining, as well as the number of GFAP and CD11b immunopositive cells.	cresyl violet	172-185	insulin like growth factor 1	Homo sapiens	18-23
21207139-7	2011	Cresyl violet positive (CV(+)) cells were significantly decreased in the stratum pyramidale (SP) of the hippocampal CA1 region (CA1) of the 4 days post-ischemia-group, while 79.4% of CV(+) cells were detected in the CA1 of the stressed-ischemia-group.	cresyl violet	0-13	carbonic anhydrase 1	Homo sapiens	116-119
21207139-7	2011	Cresyl violet positive (CV(+)) cells were significantly decreased in the stratum pyramidale (SP) of the hippocampal CA1 region (CA1) of the 4 days post-ischemia-group, while 79.4% of CV(+) cells were detected in the CA1 of the stressed-ischemia-group.	cresyl violet	0-13	carbonic anhydrase 1	Homo sapiens	128-131
21207139-7	2011	Cresyl violet positive (CV(+)) cells were significantly decreased in the stratum pyramidale (SP) of the hippocampal CA1 region (CA1) of the 4 days post-ischemia-group, while 79.4% of CV(+) cells were detected in the CA1 of the stressed-ischemia-group.	cresyl violet	0-13	carbonic anhydrase 1	Homo sapiens	128-131
20510297-10	2010	The dentate gyrus and CA1 exhibited significant neuronal death indicated by both cresyl violet and GFAP staining.	cresyl violet	81-94	carbonic anhydrase 1	Mus musculus	22-25
20571522-6	2010	CA1 neural injury was determined by cresyl violet staining.	cresyl violet	36-49	carbonic anhydrase 1	Mus musculus	0-3
19405149-5	2009	Here I analyze H2AX phosphorylation in immunostained testis sections comparing PAS/cresyl violet counterstained, noncounterstained, and immuno-fluorescence preparations and show several waves of H2AX phosphorylation/dephosphorylation coupled to various developmental phases of spermatogonia and spermatocytes as well as to spermatid differentiation.	cresyl violet	83-96	H2A.X variant histone	Homo sapiens	15-19
20226231-5	2010	Using cresyl violet staining, we also observed that an increased number of CA3 neurons survived in the 0.1 microM FK506 group compared to the KA only group.	cresyl violet	6-19	carbonic anhydrase 3	Homo sapiens	75-78
19777341-3	2010	Increased cell death--as detected by FluoroJade B staining--and extensive cell loss--as determined by cresyl violet staining--were observed mainly in the CA3 and CA4 areas of the ipsilateral and contralateral hippocampus, 7 days following intrahippocampal administration of kainic acid.	cresyl violet	102-115	carbonic anhydrase 4	Rattus norvegicus	162-165