PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
3092188-5	1986	In a construct for producing beta-galactosidase, the effects on translational yields of the tri-nucleotide 5" to the initiation codon are dependent on the entire triplet.	tri-nucleotide	92-106	galactosidase beta 1	Homo sapiens	29-47
30271085-4	2018	Whole exome sequencing revealed a novel truncating variant (c.2158C>T/p.Gln720Stop) together with a novel tri-nucleotide insertion (c.893_894insTCG that caused the insertion of an arginine at amino acid position 299) in the MARS gene.	tri-nucleotide	109-123	methionyl-tRNA synthetase 1	Homo sapiens	227-231
32082115-2	2020	Structural variants within the genome can play a significant role in neurodegenerative disease risk, such as the repeat expansion in C9orf72 and the tri-nucleotide repeat in ATXN2, both of which are associated with familial and sporadic ALS.	tri-nucleotide	149-163	ataxin 2	Homo sapiens	174-179
32082115-2	2020	Structural variants within the genome can play a significant role in neurodegenerative disease risk, such as the repeat expansion in C9orf72 and the tri-nucleotide repeat in ATXN2, both of which are associated with familial and sporadic ALS.	tri-nucleotide	149-163	superoxide dismutase 1	Homo sapiens	237-240
33565318-3	2021	Cytosine-adenine repeat (IGF-I), rs3877899, G-2548A, GGC (eRF3a/GSPT1), IVS2nt-124A/G have shown an increased risk of breast cancers and a decreased risk has been observed in 4G/5G (PAI-1), rs6505162, Tri-nucleotide (GCG TGFBR1).	tri-nucleotide	201-215	insulin like growth factor 1	Homo sapiens	0-30
25358814-2	2014	In Huntington"s disease (HD), a neurodegenerative disease caused by a tri-nucleotide repeat expansion in the huntingtin gene, extensive transcriptional dysregulation has been reported.	tri-nucleotide	70-84	huntingtin	Homo sapiens	109-119
29556396-12	2018	Moreover, our results also confirmed an association between long tri-nucleotide repeats of androgen receptor, sex steroids, pituitary, and thyroid hormones in relation to  acquired premature ejaculatory dysfunction in diabetic patients.	tri-nucleotide	65-79	androgen receptor	Homo sapiens	91-108
28498846-0	2017	MeCP2 recognizes cytosine methylated tri-nucleotide and di-nucleotide sequences to tune transcription in the mammalian brain.	tri-nucleotide	37-51	methyl-CpG binding protein 2	Homo sapiens	0-5
28498846-3	2017	Here we show by in vitro and in vivo analyses that MeCP2 binding to non-CG methylated sites in brain is largely confined to the tri-nucleotide sequence mCAC.	tri-nucleotide	128-142	methyl-CpG binding protein 2	Homo sapiens	51-56
28498846-3	2017	Here we show by in vitro and in vivo analyses that MeCP2 binding to non-CG methylated sites in brain is largely confined to the tri-nucleotide sequence mCAC.	tri-nucleotide	128-142	recessive cataract	Mus musculus	152-156
28084402-4	2017	We fine-mapped the regulatory region located in SMIM1 intron 2 in Swedish blood donors, and observed a strong correlation between expression and rs1175550 as well as with a previously unreported tri-nucleotide insertion (rs143702418; C > CGCA).	tri-nucleotide	195-209	small integral membrane protein 1 (Vel blood group)	Homo sapiens	48-53
29111027-1	2017	Spinocerebellar ataxia type 8 (SCA8), an autosomal dominant neurodegenerative disorder showing slowly progressive cerebellar ataxia, is caused by a tri-nucleotide CTG repeat expansion (CTGexp) in the SCA8 gene.	tri-nucleotide	148-162	ataxin 8	Homo sapiens	0-29
29111027-1	2017	Spinocerebellar ataxia type 8 (SCA8), an autosomal dominant neurodegenerative disorder showing slowly progressive cerebellar ataxia, is caused by a tri-nucleotide CTG repeat expansion (CTGexp) in the SCA8 gene.	tri-nucleotide	148-162	ataxin 8	Homo sapiens	31-35
29111027-1	2017	Spinocerebellar ataxia type 8 (SCA8), an autosomal dominant neurodegenerative disorder showing slowly progressive cerebellar ataxia, is caused by a tri-nucleotide CTG repeat expansion (CTGexp) in the SCA8 gene.	tri-nucleotide	148-162	ataxin 8	Homo sapiens	200-204
27660999-0	2017	A Tri-Nucleotide Pattern in a 3" UTR Segment Affects The Activity of a Human Glucocorticoid Receptor Isoform.	tri-nucleotide	2-16	nuclear receptor subfamily 3 group C member 1	Homo sapiens	77-100
22718980-3	2012	The CCG repeats are associated with three tri-nucleotide repeat disorders: Huntington"s disease, myotonic dystrophy type 1 and chromosome X-linked mental retardation (FRAXE).	tri-nucleotide	42-56	fragile site, folic acid type, rare, fra(X)(q28) E	Homo sapiens	167-172
24503862-1	2014	Huntington"s disease (HD) is a devastating, genetic neurodegenerative disease caused by a tri-nucleotide expansion in exon 1 of the huntingtin gene.	tri-nucleotide	90-104	huntingtin	Homo sapiens	132-142
22393909-1	2012	Spinocerebellar ataxia type 6 (SCA6) is an autosomal-dominant neurodegenerative disorder caused by a small expansion of tri-nucleotide (CAG) repeat encoding polyglutamine (polyQ) in the gene for alpha(1A) voltage-dependent calcium channel (Ca(v) 2.1).	tri-nucleotide	120-134	calcium voltage-gated channel subunit alpha1 A	Homo sapiens	0-29
22393909-1	2012	Spinocerebellar ataxia type 6 (SCA6) is an autosomal-dominant neurodegenerative disorder caused by a small expansion of tri-nucleotide (CAG) repeat encoding polyglutamine (polyQ) in the gene for alpha(1A) voltage-dependent calcium channel (Ca(v) 2.1).	tri-nucleotide	120-134	calcium voltage-gated channel subunit alpha1 A	Homo sapiens	31-35
22393909-1	2012	Spinocerebellar ataxia type 6 (SCA6) is an autosomal-dominant neurodegenerative disorder caused by a small expansion of tri-nucleotide (CAG) repeat encoding polyglutamine (polyQ) in the gene for alpha(1A) voltage-dependent calcium channel (Ca(v) 2.1).	tri-nucleotide	120-134	calcium voltage-gated channel subunit alpha1 A	Homo sapiens	195-238
22393909-1	2012	Spinocerebellar ataxia type 6 (SCA6) is an autosomal-dominant neurodegenerative disorder caused by a small expansion of tri-nucleotide (CAG) repeat encoding polyglutamine (polyQ) in the gene for alpha(1A) voltage-dependent calcium channel (Ca(v) 2.1).	tri-nucleotide	120-134	immunoglobulin lambda variable 3-1	Homo sapiens	240-249
21971961-3	2011	The disorder is caused by an expanded cystosine adenine guanine (CAG) tri-nucleotide repeat encoding polyglutamine (polyQ) in the first exon of the Huntingtin gene.	tri-nucleotide	70-84	huntingtin	Homo sapiens	148-158
17431729-0	2007	Association of tri-nucleotide (CAG and GGC) repeat polymorphism of androgen receptor gene in Taiwanese women with refractory or remission rheumatoid arthritis.	tri-nucleotide	15-29	androgen receptor	Homo sapiens	67-84
20979941-2	2010	We studied the effect of tri-nucleotide repeat (CTGs) polymorphisms in exon 2 of the CNDP1 gene, which codes for carnosinase and is responsible for the degradation of carnosine, on the clinical outcome of Chinese peritoneal dialysis (PD) patients.	tri-nucleotide	25-39	carnosine dipeptidase 1	Homo sapiens	85-90
17482958-0	2007	CYP19 TCT tri-nucleotide Del/Del genotype is a susceptibility marker for prostate cancer in a Taiwanese population.	tri-nucleotide	10-24	cytochrome P450 family 19 subfamily A member 1	Homo sapiens	0-5
16872533-11	2006	Using family-based tests of association the number of tri-nucleotide repeats in a nearby region of the MEF2A gene was not associated with IHD in our study group.	tri-nucleotide	54-68	myocyte enhancer factor 2A	Homo sapiens	103-108
16951497-5	2006	RESULTS: The tri-nucleotide (CAG) length polymorphism in the last coding exon of MEF2A in the Chinese was revealed and 4 of the 175 (2.3%) CAD samples containing 4 prolines were due to one proline deletion in MEF2A gene.	tri-nucleotide	13-27	myocyte enhancer factor 2A	Homo sapiens	81-86
34680707-8	2021	We detected 105 repeat motifs, of which AT/AT (50.19%), AC/GT (39.15%), CAA/TTG (32.46%), and ACA/TGT (10.86%) were the most common in di- and tri-nucleotide repeats.	tri-nucleotide	143-157	queuine tRNA-ribosyltransferase catalytic subunit	Bactrocera dorsalis	98-101
16644517-1	2006	Friedreich"s ataxia (FRDA), the most common subtype of early onset hereditary spinocerebellar ataxia (SCA), is an autosomal recessive neurodegenerative disorder caused by unstable GAA tri-nucleotide expansions in the first intron of FRDA gene located at 9q13-q21.1 position.	tri-nucleotide	184-198	alpha glucosidase	Homo sapiens	180-183
18404512-8	2005	Tri-nucleotide pretreatment also reduced the EGF induced Ca(2+) response.	tri-nucleotide	0-14	epidermal growth factor	Homo sapiens	45-48
10369863-1	1999	Spinocerebellar ataxia type 6 (SCA6) is one of the eight neurodegenerative diseases caused by a tri-nucleotide (CAG) repeat expansion coding polyglutamine (CAG repeat/polyglutamine diseases) and is characterized by late onset autosomal dominant cerebellar ataxia and predominant loss of cerebellar Purkinje cells.	tri-nucleotide	96-110	calcium voltage-gated channel subunit alpha1 A	Homo sapiens	0-29
10369863-1	1999	Spinocerebellar ataxia type 6 (SCA6) is one of the eight neurodegenerative diseases caused by a tri-nucleotide (CAG) repeat expansion coding polyglutamine (CAG repeat/polyglutamine diseases) and is characterized by late onset autosomal dominant cerebellar ataxia and predominant loss of cerebellar Purkinje cells.	tri-nucleotide	96-110	calcium voltage-gated channel subunit alpha1 A	Homo sapiens	31-35