PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
27009865-8	2016	Moreover, silencing of IRE1alpha dramatically abrogated IMB-6G-induced pro-apoptotic ASK1-JNK signaling.	IMB-6G	56-62	endoplasmic reticulum to nucleus signaling 1	Homo sapiens	23-32
27009865-8	2016	Moreover, silencing of IRE1alpha dramatically abrogated IMB-6G-induced pro-apoptotic ASK1-JNK signaling.	IMB-6G	56-62	mitogen-activated protein kinase kinase kinase 5	Homo sapiens	85-89
27009865-9	2016	Importantly, interruption of CHOP rendered HCC cells sensitive to IMB-6G-induced apoptosis via inactivation of Bim, PUMA and Bax.	IMB-6G	66-72	DNA damage inducible transcript 3	Homo sapiens	29-33
27009865-10	2016	Thus, the IRE1alpha-ASK1 and PERK-CHOP pathways may be a novel molecular mechanism of IMB-6G-induced apoptosis.	IMB-6G	86-92	endoplasmic reticulum to nucleus signaling 1	Homo sapiens	10-19
27009865-10	2016	Thus, the IRE1alpha-ASK1 and PERK-CHOP pathways may be a novel molecular mechanism of IMB-6G-induced apoptosis.	IMB-6G	86-92	mitogen-activated protein kinase kinase kinase 5	Homo sapiens	20-24
27009865-10	2016	Thus, the IRE1alpha-ASK1 and PERK-CHOP pathways may be a novel molecular mechanism of IMB-6G-induced apoptosis.	IMB-6G	86-92	eukaryotic translation initiation factor 2 alpha kinase 3	Homo sapiens	29-33
27009865-10	2016	Thus, the IRE1alpha-ASK1 and PERK-CHOP pathways may be a novel molecular mechanism of IMB-6G-induced apoptosis.	IMB-6G	86-92	DNA damage inducible transcript 3	Homo sapiens	34-38