PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
1499645-8	1992	24-h preincubation of human adipocytes with 10(-2), 10, 10(4) IU ml-1 interferon increased maximally-insulin stimulated 14C-glucose transport clearance (P less than 0.05) and glucose transport responsiveness to insulin was enhanced by 24% (P less than 0.05) in cells exposed to 10(-2) IU ml-1 interferon.	14c-glucose	120-131	insulin	Homo sapiens	101-108
3076052-3	1988	GIP also stimulated the conversion of 14C-glucose into extractable lipids by up to 81% at 10(-7) mol/l.	14c-glucose	38-49	gastric inhibitory polypeptide	Rattus norvegicus	0-3
2112110-4	1990	Maximally effective concentrations of insulin and HPro caused similar stimulations of 14C-glucose incorporation into glycogen and glycogen synthase activity.	14c-glucose	86-97	insulin	Homo sapiens	38-45
7323032-1	1981	The ability of somatotropin seival and its fragment 77-107, retaining a marked growth-stimulationg activity, to change the intensity of 14C-glucose consumption from the medium and 14C-glucose inclusion into glycogen by in vitro incubated semidiaphragm of hypophysectomized rats was studied.	14c-glucose	136-147	growth hormone 1	Rattus norvegicus	15-27
3932573-4	1985	We constructed an artificial hexose monophosphate pathway from these enzymes, providing as substrate 14C-labeled glucose-6-phosphate either directly or by continual generation from 14C-glucose by yeast hexokinase and adenosine triphosphate.	14c-glucose	181-192	hexokinase	Saccharomyces cerevisiae S288C	202-212
6374254-5	1984	The insulin stimulation of 14C-glucose (0.2 mM) incorporation to 14CO2 and total lipids was significantly reduced in the adipocytes pretreated with sera from 19 of the 29 uremic patients.	14c-glucose	27-38	insulin	Homo sapiens	4-11
3967842-3	1985	The biological actions of GH determined were the stimulation of 14C-glucose oxidation to 14CO2 and conversion to 14C-lipid.	14c-glucose	64-75	gonadotropin releasing hormone receptor	Rattus norvegicus	26-28
6788631-3	1981	The response of total cell glucose metabolism to insulin was assessed by measurement of 14C-glucose uptake.	14c-glucose	88-99	insulin	Homo sapiens	49-56
6253506-2	1980	Addition of insulin to cells cultured in 0.5% serum stimulated incorporation of 5mM 14C-glucose into glycogen and increased cellular glycogen content, without inducing proliferation.	14c-glucose	84-95	insulin	Mesocricetus auratus	12-19
6997676-4	1980	When insulin was infused at 1 mU/kg/min the net splanchnic production of 14C-glucose was suppressed by 80% but glucagon infused at the end of the study resulted in substantial release of 14C-glucose from the liver suggesting marked accumulation of labeled glucose in glycogen.	14c-glucose	73-84	insulin	Homo sapiens	5-12
6997676-4	1980	When insulin was infused at 1 mU/kg/min the net splanchnic production of 14C-glucose was suppressed by 80% but glucagon infused at the end of the study resulted in substantial release of 14C-glucose from the liver suggesting marked accumulation of labeled glucose in glycogen.	14c-glucose	187-198	insulin	Homo sapiens	5-12
6997676-5	1980	When insulin was infused at 5 mU/kg/min the splanchnic release of 14C-glucose was also markedly suppressed but in contrast to the lower insulin dose very little labeled glucose accumulated in glycogen.	14c-glucose	66-77	insulin	Homo sapiens	5-12
7000584-7	1980	The KE for a half-maximal response of hormone-stimulated incorporationof 14C-glucose into glycogen was 10(-11) M insulin, corresponding to less than 1% receptor occupancy.	14c-glucose	73-84	insulin	Homo sapiens	113-120
582621-2	1979	When 14C-glucose was used as substrate, incorporation into both phosphatidylcholine and saturated phosphatidylcholine was reduced by insulin.	14c-glucose	5-16	insulin	Oryctolagus cuniculus	133-140
6995010-3	1980	In addition, PTH increased the conversion of 14C-alanine to 14C-glucose.	14c-glucose	60-71	parathyroid hormone	Homo sapiens	13-16
28435144-4	2017	The results also suggest the effect of hSTC-1 on de novo fatty acid synthesis from pyruvate generated by 14C-glucose in the glycolysis pathway.	14c-glucose	105-116	stanniocalcin 1	Homo sapiens	39-45
955377-0	1976	The in vivo metabolism of 14C-glucose and 14C-glycine in insulin-treated northern pike (Esox lucius L.).	14c-glucose	26-37	insulin	Esox lucius	57-64
27730346-4	2016	The effect of hSTC-1 on de novo fatty acid synthesis and on glucose oxidation in WRAT is supported by an 85 % increase in 14CO2 production from 14C-glucose.	14c-glucose	144-155	stanniocalcin 1	Homo sapiens	14-20
9794244-10	1998	Insulin-stimulated incorporation of 14C-glucose into glycogen was inhibited by PDE 3- and PDE 4-specific inhibitors as well as by the unspecific PDE inhibitor IMBX.	14c-glucose	36-47	phosphodiesterase 4D, cAMP-specific-like 1	Rattus norvegicus	79-84
26608612-9	2016	The 14C-glucose incorporation into fatty acids was reduced by ~25% by AGP treatment for 24 h with 1000 ng AGP/ml medium (P<0.05).	14c-glucose	4-15	alpha-1-acid glycoprotein	Sus scrofa	70-73
26608612-9	2016	The 14C-glucose incorporation into fatty acids was reduced by ~25% by AGP treatment for 24 h with 1000 ng AGP/ml medium (P<0.05).	14c-glucose	4-15	alpha-1-acid glycoprotein	Sus scrofa	106-109
10336421-7	1999	A consistent relationship was observed between the expression of individual HXT genes and the glucose transport kinetics determined from zero-trans influx of 14C-glucose during 5 s. This relationship was in broad agreement with the transport kinetics of Hxt1-Hxt7 and Gal2 deduced in previous studies on single-HXT strains.	14c-glucose	158-169	hexose transporter HXT1	Saccharomyces cerevisiae S288C	254-258
10336421-7	1999	A consistent relationship was observed between the expression of individual HXT genes and the glucose transport kinetics determined from zero-trans influx of 14C-glucose during 5 s. This relationship was in broad agreement with the transport kinetics of Hxt1-Hxt7 and Gal2 deduced in previous studies on single-HXT strains.	14c-glucose	158-169	hexose transporter HXT7	Saccharomyces cerevisiae S288C	259-263
10336421-7	1999	A consistent relationship was observed between the expression of individual HXT genes and the glucose transport kinetics determined from zero-trans influx of 14C-glucose during 5 s. This relationship was in broad agreement with the transport kinetics of Hxt1-Hxt7 and Gal2 deduced in previous studies on single-HXT strains.	14c-glucose	158-169	galactose permease GAL2	Saccharomyces cerevisiae S288C	268-272