PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
35211416-5	2022	However, in our clinical practice, we found that disease progressed rapidly even after venetoclax+azacitidine (AZA) therapy in two relapsed t(8;21) AML patients with KIT mutations.	aza	111-114	KIT proto-oncogene, receptor tyrosine kinase	Homo sapiens	166-169
24777031-9	2016	At baseline, the greatest C4d urinary excretion was noticed in patients treated with CSA + AZA, 199.5 +- 175.9 ng/mL (5.3 +- 7.1 ng/mgCr) and the lowest in those in whom tacrolimus and mycophenolate mophetil was administered, 166.6 +- 186.3 ng/mL (3.9 +- 6.2 ng/mgCr).	aza	91-94	MN1 proto-oncogene, transcriptional regulator	Homo sapiens	132-136
30356025-11	2018	Furthermore, ASCEMS pre-treated with AZA/RES displayed anti-inflammatory properties, as decreased levels of TNF-alpha, nitric oxide (NO), and IL-6 were observed in those cells in comparison with their untreated counterparts in the co-culture with RAW264.7 macrophages.	aza	37-40	tumor necrosis factor	Equus caballus	108-117
30356025-11	2018	Furthermore, ASCEMS pre-treated with AZA/RES displayed anti-inflammatory properties, as decreased levels of TNF-alpha, nitric oxide (NO), and IL-6 were observed in those cells in comparison with their untreated counterparts in the co-culture with RAW264.7 macrophages.	aza	37-40	interleukin 6	Equus caballus	142-146
29999247-11	2018	Furthermore, we proved that AZA/RES exerts its beneficial effects by modulating autophagy and mitochondrial dynamics through PARKIN and RUNX-2 activity.	aza	28-31	RUNX family transcription factor 2	Equus caballus	136-142
29771984-8	2018	Demethylation using 5-aza-2"-deoxycitidine (AZA) increased TFR2 mRNA expression in Huh7.	aza	44-47	transferrin receptor 2	Homo sapiens	59-63
29771984-8	2018	Demethylation using 5-aza-2"-deoxycitidine (AZA) increased TFR2 mRNA expression in Huh7.	aza	44-47	MIR7-3 host gene	Homo sapiens	83-87
29771984-10	2018	However, HAMP mRNA expression in Huh7 was increased by AZA treatment, suggesting that methylation of one or more iron sensing genes may indirectly influence HAMP expression.	aza	55-58	hepcidin antimicrobial peptide	Homo sapiens	9-13
29771984-10	2018	However, HAMP mRNA expression in Huh7 was increased by AZA treatment, suggesting that methylation of one or more iron sensing genes may indirectly influence HAMP expression.	aza	55-58	MIR7-3 host gene	Homo sapiens	33-37
29771984-10	2018	However, HAMP mRNA expression in Huh7 was increased by AZA treatment, suggesting that methylation of one or more iron sensing genes may indirectly influence HAMP expression.	aza	55-58	hepcidin antimicrobial peptide	Homo sapiens	157-161
27904681-7	2016	MiR-330-3p expression was increased in gastric cancer cells after treated with histone deacetylase inhibitor trichostatin A (TSA) and DNA methylation inhibitor 5-aza-CdR (AZA).	aza	171-174	microRNA 330	Homo sapiens	0-7
24777031-9	2016	At baseline, the greatest C4d urinary excretion was noticed in patients treated with CSA + AZA, 199.5 +- 175.9 ng/mL (5.3 +- 7.1 ng/mgCr) and the lowest in those in whom tacrolimus and mycophenolate mophetil was administered, 166.6 +- 186.3 ng/mL (3.9 +- 6.2 ng/mgCr).	aza	91-94	MN1 proto-oncogene, transcriptional regulator	Homo sapiens	262-266
25500741-10	2015	AZA administration partly increased mRNA and protein expressions of COX II, and COX activity decreased by CSE and attenuated the toxic effects of CSE.	aza	0-3	mitochondrially encoded cytochrome c oxidase II	Homo sapiens	68-74
21799893-3	2011	Treatment of BPCs with Aza and TSA induced expression of pluripotent genes Oct4, Nanog, Sox2, and thereafter culturing these cells in defined cardiac myocyte-conditioned medium resulted in their differentiation into cardiomyocyte progenitors and subsequently into cardiac myocytes.	aza	23-26	POU domain, class 5, transcription factor 1, related sequence 1	Mus musculus	75-79
21799893-3	2011	Treatment of BPCs with Aza and TSA induced expression of pluripotent genes Oct4, Nanog, Sox2, and thereafter culturing these cells in defined cardiac myocyte-conditioned medium resulted in their differentiation into cardiomyocyte progenitors and subsequently into cardiac myocytes.	aza	23-26	Nanog homeobox	Mus musculus	81-86
21799893-3	2011	Treatment of BPCs with Aza and TSA induced expression of pluripotent genes Oct4, Nanog, Sox2, and thereafter culturing these cells in defined cardiac myocyte-conditioned medium resulted in their differentiation into cardiomyocyte progenitors and subsequently into cardiac myocytes.	aza	23-26	SRY (sex determining region Y)-box 2	Mus musculus	88-92
20060001-6	2010	This expression was potentiated by first enriching for CD117/Sca-1 cells followed by differentiation (AZA, 39% and LS, 28%).	aza	102-105	KIT proto-oncogene receptor tyrosine kinase	Mus musculus	55-60
20060001-6	2010	This expression was potentiated by first enriching for CD117/Sca-1 cells followed by differentiation (AZA, 39% and LS, 28%).	aza	102-105	ataxin 1	Mus musculus	61-66
20060001-9	2010	In conclusion, IP CD117/Sca-1(+) murine BM-MSCs display robust cardiac muscle lineage development that can be induced independent of AZA but is diminished under higher serum concentrations.	aza	133-136	KIT proto-oncogene receptor tyrosine kinase	Mus musculus	18-23
20060001-9	2010	In conclusion, IP CD117/Sca-1(+) murine BM-MSCs display robust cardiac muscle lineage development that can be induced independent of AZA but is diminished under higher serum concentrations.	aza	133-136	ataxin 1	Mus musculus	24-29
15816871-6	2005	Interestingly, AZA decreased reelin promoter methylation without concomittantly increasing histone acetylation.	aza	15-18	reelin	Homo sapiens	29-35
15816871-9	2005	Our data indicate that while TSA and VPA act to increase histone acetylation and reduce promoter methylation, AZA acts only to decrease the amount of reelin promoter methylation.	aza	110-113	reelin	Homo sapiens	150-156