PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
3955844-4	1986	This effect of cycloheximide lends support to the hypothesis that the liver enzyme increases after CCl4 are probably due to increased synthesis, in addition to the classically held mechanisms of leakage from necrotic or damaged hepatocytes.	Cycloheximide	15-28	C-C motif chemokine ligand 4	Rattus norvegicus	99-103	
131362-4	1976	Aminotriazole or cycloheximide pretreatment but not pyrazole administration decrease the intensity of polysome rupture caused by CCl4.	Cycloheximide	17-30	C-C motif chemokine ligand 4	Rattus norvegicus	129-133	
6301476-2	1983	The administration of either cycloheximide or actinomycin D completely blocked the CCl4-mediated induction of choline kinase activity, indicating that the elevated activity could be due to the change in the enzyme level.	Cycloheximide	29-42	C-C motif chemokine ligand 4	Rattus norvegicus	83-87	
6696388-1	1984	The previously reported increases in liver and serum aspartate aminotransferase (ASAT) activities and liver protein content 24 hours after the administration of carbon tetrachloride (CCl4) were reduced by administering multiple doses of the protein synthesis inhibitor cycloheximide (CH).	Cycloheximide	269-282	C-C motif chemokine ligand 4	Rattus norvegicus	183-187	
6696388-1	1984	The previously reported increases in liver and serum aspartate aminotransferase (ASAT) activities and liver protein content 24 hours after the administration of carbon tetrachloride (CCl4) were reduced by administering multiple doses of the protein synthesis inhibitor cycloheximide (CH).	Cycloheximide	284-286	C-C motif chemokine ligand 4	Rattus norvegicus	183-187