PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7532249-9	1994	However, cycloheximide alone resulted in a significant up-regulation of DAF mRNA levels on HMC.	Cycloheximide	9-22	CD55 molecule (Cromer blood group)	Homo sapiens	72-75	
9737666-8	1998	Actinomycin D, cycloheximide, and brefeldin A inhibited the increase in DAF release induced by IL-4 and IL-1beta stimulation.	Cycloheximide	15-28	CD55 molecule (Cromer blood group)	Homo sapiens	72-75	
7507966-7	1994	The histamine-induced DAF expression was inhibited by actinomycin D and cycloheximide and accompanied by an increase in the DAF mRNA level, indicating that both transcription and translation are required.	Cycloheximide	72-85	CD55 molecule (Cromer blood group)	Homo sapiens	22-25	
1719094-8	1991	Thus, both cycloheximide and actinomycin D blocked the increase on MC DAF observed after incubation with IC and serum.	Cycloheximide	11-24	CD55 molecule (Cromer blood group)	Homo sapiens	70-73	
1688581-12	1990	The increased expression of DAF only was evident 8 h after PMA addition and was blocked by the RNA and protein synthesis inhibitors, actinomycin D and cycloheximide, indicating that both transcription and translation are required for DAF synthesis induced by phorbol esters.	Cycloheximide	151-164	CD55 molecule (Cromer blood group)	Homo sapiens	28-31	
1688581-12	1990	The increased expression of DAF only was evident 8 h after PMA addition and was blocked by the RNA and protein synthesis inhibitors, actinomycin D and cycloheximide, indicating that both transcription and translation are required for DAF synthesis induced by phorbol esters.	Cycloheximide	151-164	CD55 molecule (Cromer blood group)	Homo sapiens	234-237