PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 7532249-9 1994 However, cycloheximide alone resulted in a significant up-regulation of DAF mRNA levels on HMC. Cycloheximide 9-22 CD55 molecule (Cromer blood group) Homo sapiens 72-75 9737666-8 1998 Actinomycin D, cycloheximide, and brefeldin A inhibited the increase in DAF release induced by IL-4 and IL-1beta stimulation. Cycloheximide 15-28 CD55 molecule (Cromer blood group) Homo sapiens 72-75 7507966-7 1994 The histamine-induced DAF expression was inhibited by actinomycin D and cycloheximide and accompanied by an increase in the DAF mRNA level, indicating that both transcription and translation are required. Cycloheximide 72-85 CD55 molecule (Cromer blood group) Homo sapiens 22-25 1688581-12 1990 The increased expression of DAF only was evident 8 h after PMA addition and was blocked by the RNA and protein synthesis inhibitors, actinomycin D and cycloheximide, indicating that both transcription and translation are required for DAF synthesis induced by phorbol esters. Cycloheximide 151-164 CD55 molecule (Cromer blood group) Homo sapiens 28-31 1688581-12 1990 The increased expression of DAF only was evident 8 h after PMA addition and was blocked by the RNA and protein synthesis inhibitors, actinomycin D and cycloheximide, indicating that both transcription and translation are required for DAF synthesis induced by phorbol esters. Cycloheximide 151-164 CD55 molecule (Cromer blood group) Homo sapiens 234-237 1719094-8 1991 Thus, both cycloheximide and actinomycin D blocked the increase on MC DAF observed after incubation with IC and serum. Cycloheximide 11-24 CD55 molecule (Cromer blood group) Homo sapiens 70-73