PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 25116192-5 2014 RESULTS: Compared to C, STZ-WT mice had significantly increased macrophage and TLR4 immunostaining in kidney, significant increases in MyD88, Interferon Regulatory Factor-3, NFKappaB activity, TNF-Alpha, IL-6, and MCP-1; all these were significantly decreased in the STZ-TLR4KO compared to STZ-WT mice. Streptozocin 24-27 myeloid differentiation primary response gene 88 Mus musculus 135-140 30146809-2 2018 METHODS: : Diabetes was induced by feeding high-fat/high-sugar diet and injection of low-dose streptozotocin in MyD88-knockout mice. Streptozocin 94-108 myeloid differentiation primary response gene 88 Mus musculus 112-117 27650857-7 2016 Bladder expression of TLR4 and MyD88 and serum expression of HMGB1 were increased in STZ compared with control mice. Streptozocin 85-88 myeloid differentiation primary response gene 88 Mus musculus 31-36 34552217-5 2022 We showed that MyD88 was significantly activated in the kidney of STZ-induced type 1 diabetic mice in tubular epithelial cells as well as in high glucose-treated rat tubular epithelial cells NRK-52E. Streptozocin 66-69 myeloid differentiation primary response gene 88 Mus musculus 15-20 21617141-6 2011 Macrophage TLR2 expression and MyD88-dependent signaling were increased in diabetic mice (WT+STZ) compared with nondiabetic WT mice. Streptozocin 93-96 myeloid differentiation primary response gene 88 Mus musculus 31-36 19357791-5 2009 In contrast, after low-dose streptozotocin (STZ) challenge, Myd88-/-mice had an abnormal IPGTT relative to WT controls. Streptozocin 28-42 myeloid differentiation primary response gene 88 Mus musculus 60-65 19357791-5 2009 In contrast, after low-dose streptozotocin (STZ) challenge, Myd88-/-mice had an abnormal IPGTT relative to WT controls. Streptozocin 44-47 myeloid differentiation primary response gene 88 Mus musculus 60-65 19357791-6 2009 Furthermore, Myd88-/- mice suffer enhanced beta-cell apoptosis and have enhanced hepatic damage with delayed recovery upon low-dose STZ treatment. Streptozocin 132-135 myeloid differentiation primary response gene 88 Mus musculus 13-18 20733560-11 2010 Wounds of TLR2(-/-)+STZ mice showed less oxidative stress, decreased MyD88 signaling, NF-kappaB activation, and cytokine secretion. Streptozocin 20-23 myeloid differentiation primary response gene 88 Mus musculus 69-74