PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12909734-10	2003	A number of environmental and lifestyle factors, such as smoking, alcohol, coffee consumption, and exposure to organochlorine or hydrocarbon solvent, have been associated with the frequency and spectrum of K-ras mutation in pancreatic tumors.	Hydrocarbons	129-140	KRAS proto-oncogene, GTPase	Homo sapiens	206-211	
11756230-1	2002	Occupational exposure to hydrocarbon solvents has been found to be associated with an increased risk of exocrine pancreatic cancer (EPC), the human tumor with the highest prevalence of K-ras mutations.	Hydrocarbons	25-36	KRAS proto-oncogene, GTPase	Homo sapiens	185-190	
11756230-3	2002	We analysed the relationship between past occupational exposure to hydrocarbon solvents and mutations in codon 12 of the K-ras gene in 107 incident cases of EPC.	Hydrocarbons	67-78	KRAS proto-oncogene, GTPase	Homo sapiens	121-126	
11756230-12	2002	The results raise the possibility that hydrocarbon solvents might be involved in the pathogenesis of EPC, possibly through indirect modulation of K-ras activation.	Hydrocarbons	39-50	KRAS proto-oncogene, GTPase	Homo sapiens	146-151	
22189140-3	2012	Isothermal calorimetric characterization of the role of hydrocarbon substitution of this six-membered ring, upon binding the HCV NS3 protease, was found to be exclusively entropic in nature.	Hydrocarbons	56-67	KRAS proto-oncogene, GTPase	Homo sapiens	129-132	
25624485-0	2015	Direct inhibition of oncogenic KRAS by hydrocarbon-stapled SOS1 helices.	Hydrocarbons	39-50	KRAS proto-oncogene, GTPase	Homo sapiens	31-35