PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20495578-11	2010	In vitro data suggested that low concentrations of As2O3 could induce only a small degree of apoptosis in primary pulmonary cells but could significantly inhibit the secretion of eotaxin by these cells.	Arsenic Trioxide	51-56	chemokine (C-C motif) ligand 11	Mus musculus	179-186	
20495578-9	2010	We found that administration of As2O3 in OVA-immunized mice abrogated airway eosinophil recruitment by downregulating eotaxin expression but did not alter serum IgE or IL-5 levels in bronchoalveolar lavage fluid (BALF).	Arsenic Trioxide	32-37	chemokine (C-C motif) ligand 11	Mus musculus	118-125	
17178007-8	2006	RESULTS: As2O3 attenuated the allergen-induced serum IgE, chemokine expression of eotaxin and RANTES, and the EOS recruitment in bronchoalveolar lavage fluid, which is associated with an increased IkappaBalpha expression as well as a decreased NF-kappaB activation.	Arsenic Trioxide	9-14	chemokine (C-C motif) ligand 11	Mus musculus	82-89