PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31597940-3	2019	The mutation of sec59-1 caused a protein N-glycosylation defect mediated ER stress resulting in increased levels of phospholipid, neutral lipid and sterol, whereas growth was reduced.	Sterols	148-154	dolichol kinase	Saccharomyces cerevisiae S288C	16-21	
31597940-7	2019	The microarray data also revealed increased expression of genes involved in phospholipid, TAG, fatty acid, sterol synthesis, and phospholipid transport resulting in dysregulation of lipid homeostasis in the sec59-1  cell.	Sterols	107-113	dolichol kinase	Saccharomyces cerevisiae S288C	207-212