PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 16688761-7 2006 Dopamine, SKF 38393, and quinpirole increased the level of p44/42 mitogen-activated protein kinases (MAPKs), p38 MAPK, and stress-activated protein kinase/Jun-N-terminal kinase (SAPK/JNK) phosphorylation. Dopamine 0-8 mitogen-activated protein kinase 3 Mus musculus 59-62 17029599-6 2006 FBPA blocked dopamine-induced extracellular signal-regulated kinase 1/2 phosphorylation, and it specifically affected mitogen-activated protein kinase kinase (MEK)1/2 activity in hippocampal HN33 cells. Dopamine 13-21 mitogen-activated protein kinase 3 Mus musculus 30-71 16688761-7 2006 Dopamine, SKF 38393, and quinpirole increased the level of p44/42 mitogen-activated protein kinases (MAPKs), p38 MAPK, and stress-activated protein kinase/Jun-N-terminal kinase (SAPK/JNK) phosphorylation. Dopamine 0-8 mitogen-activated protein kinase 3 Mus musculus 101-105 16688761-10 2006 The dopamine-induced decrease in cyclin E, cyclin-dependent protein kinase-2 (CDK-2), and cyclin D1, CDK-4 were blocked by pertussis toxin (G protein inhibitor), SQ 22536, neomycin, bisindolylmaleimide I (PKC inhibitor), SB 203580 (p38 MAPK inhibitor), PD 98059 (p44/42 inhibitor), and SP 600125 (SAPK/JNK inhibitor). Dopamine 4-12 mitogen-activated protein kinase 3 Mus musculus 263-266 25100942-0 2014 Dopamine-induced tyrosine phosphorylation of NR2B (Tyr1472) is essential for ERK1/2 activation and processing of novel taste information. Dopamine 0-8 mitogen-activated protein kinase 3 Mus musculus 77-83 33781778-0 2021 Dopamine and glutamate receptors control social stress-induced striatal ERK1/2 activation. Dopamine 0-8 mitogen-activated protein kinase 3 Mus musculus 72-78 33228965-8 2021 These results suggest that LET during adolescence completely improves schizophrenia-like abnormal behaviors associated with improved glutamate uptake and the dopamine-induced ERK1/2 signaling pathway in the PFC. Dopamine 158-166 mitogen-activated protein kinase 3 Mus musculus 175-181 16139809-7 2006 L-DOPA also induces activation of ERK1/2 in the dopamine-depleted striatum. Dopamine 48-56 mitogen-activated protein kinase 3 Mus musculus 34-40 25100942-5 2014 Here, we tested the hypothesis that tyrosine phosphorylation of NR2B at Y1472 is correlated with ERK1/2 activation by dopamine and necessary for it as well. Dopamine 118-126 mitogen-activated protein kinase 3 Mus musculus 97-103 24406899-9 2014 In addition, we show that a single treatment of DNSP-11 in the MN9D dopaminergic neuronal cell line results in phosphorylation of ERK1/2, which suggests a novel cellular mechanism responsible for increases in DA function. Dopamine 209-211 mitogen-activated protein kinase 3 Mus musculus 130-136 24672017-1 2014 In animal models of Parkinson"s disease, striatal overactivation of ERK1/2 via dopamine (DA) D1 receptors is the hallmark of a supersensitive molecular response associated with dyskinetic behaviors. Dopamine 79-87 mitogen-activated protein kinase 3 Mus musculus 68-74 24672017-1 2014 In animal models of Parkinson"s disease, striatal overactivation of ERK1/2 via dopamine (DA) D1 receptors is the hallmark of a supersensitive molecular response associated with dyskinetic behaviors. Dopamine 89-91 mitogen-activated protein kinase 3 Mus musculus 68-74 24672017-3 2014 Application of the dopamine D1-like receptor agonist SKF38393 induced ERK1/2 phosphorylation and downstream signaling in the DA-denervated but not the intact striatum. Dopamine 19-27 mitogen-activated protein kinase 3 Mus musculus 70-76 24672017-3 2014 Application of the dopamine D1-like receptor agonist SKF38393 induced ERK1/2 phosphorylation and downstream signaling in the DA-denervated but not the intact striatum. Dopamine 125-127 mitogen-activated protein kinase 3 Mus musculus 70-76 24672017-10 2014 Our results reveal that D1-dependent ERK1/2 activation in the DA-denervated striatum depends on a complex interaction between PKA- and Ca(2+)-dependent signaling pathways that is critically modulated by striatal mGluR5. Dopamine 62-64 mitogen-activated protein kinase 3 Mus musculus 37-43 19446794-6 2009 RESULTS: Phosphorylation of ERK1/2 in response to glutamate, dopamine D1 agonist, or both stimuli simultaneously is impaired in Ras-GRF1-deficient striatal cells and organotypic slices of the striatonigral MSN compartment. Dopamine 61-69 mitogen-activated protein kinase 3 Mus musculus 28-34 22393477-4 2012 Furthermore, dopamine-related striatal signaling was analyzed by assessing the phosphorylation of AKT/GSK3beta and ERK1/2. Dopamine 13-21 mitogen-activated protein kinase 3 Mus musculus 115-121 18614680-4 2008 Loss of dopamine input to the striatum, as in models of Parkinson"s disease (PD), also results in a sensitization of responses to dopamine agonists that is manifest by increased activation of ERK1/2 in the dorsal striatum. Dopamine 8-16 mitogen-activated protein kinase 3 Mus musculus 192-198 18614680-4 2008 Loss of dopamine input to the striatum, as in models of Parkinson"s disease (PD), also results in a sensitization of responses to dopamine agonists that is manifest by increased activation of ERK1/2 in the dorsal striatum. Dopamine 130-138 mitogen-activated protein kinase 3 Mus musculus 192-198 18340382-5 2008 In addition, we found that DA, through D2 receptors, exerted its inhibitory effect on EPC mobilization through suppression of VEGFA-induced ERK1/ERK2 phosphorylation and MMP-9 synthesis. Dopamine 27-29 mitogen-activated protein kinase 3 Mus musculus 140-144