PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23828841-11	2013	We hypothesize that the deletion of one GSTP1 allele (an isoform highly expressed in embryonic tissues), associated with specific environmental factors, such as tobacco and alcohol, could cause the esophageal atresia observed in our patient.	Alcohols	173-180	glutathione S-transferase pi 1	Homo sapiens	40-45	
26768611-9	2016	An elevated risk for GC was observed in smokers and alcohol consumers carrying GSTP1 Ile/Val +Val/Val genotype (p = 0.041; odds ratio = 3.71; 95 % CI 0.98-14.12; p = 0.002; odds ratio = 12.31; 95 % CI 1.71-88.59).	Alcohols	52-59	glutathione S-transferase pi 1	Homo sapiens	79-84	
22853296-4	2012	When adjusted for age, sex, smoking status, and alcohol use, the increased risk for ESCC was significantly associated with reduced expression of GSTP1, MTHFR, and NQO1, and GSTP1 mRNA showed a steady association with the risk for ESCC (OR=2.640) in the model of stepwise regression analysis.	Alcohols	48-55	glutathione S-transferase pi 1	Homo sapiens	145-150	
15479622-5	2004	A significant interaction was observed between the GSTP1 genotype and alcohol consumption (p for interaction = 0.01); compared with never-drinking women with Ile/Ile genotype, ever-drinking women with the GSTP1 Val allele had almost a three-fold risk of breast cancer (OR = 2.9, 95 % CI = 1.05-7.85), whereas never-drinking women with Val allele had half this risk (OR = 0.5, 95 % CI = 0.27-0.93).	Alcohols	70-77	glutathione S-transferase pi 1	Homo sapiens	51-56	
23077566-0	2012	Interaction between GSTP1 Val allele and H. pylori infection, smoking and alcohol consumption and risk of gastric cancer among the Chinese population.	Alcohols	74-81	glutathione S-transferase pi 1	Homo sapiens	20-25	
23077566-10	2012	The GSTP1 Val allele shows an interaction with smoking, alcohol consumption, and especially H. pylori infection for increasing the risk of gastric cancer.	Alcohols	56-63	glutathione S-transferase pi 1	Homo sapiens	4-9	
16338071-11	2006	However, interaction of smoking or alcohol with GSTM1 null or GSTP1 ile/ile moderately increases the risk for esophageal cancer in North Indian population.	Alcohols	35-42	glutathione S-transferase pi 1	Homo sapiens	62-67	
20391126-9	2010	GSTP1 protein levels also showed significant decrease in ESCC when adjusted for age, gender, smoking status, and alcohol use.	Alcohols	113-120	glutathione S-transferase pi 1	Homo sapiens	0-5	
19388510-3	2009	The most widely studied group were genes encoded molecules engaged in biotransformations of xenobiotics, in particular potential carcinogens, like alcohol (ADH2) and aldehyde (ALDH2) dehydrogenases, various isoenzymes of cytochrome P450 (CYP1A1, CYP2E1) and glutathione S-transferase (GSTM1, GSTT1, GSTP1).	Alcohols	147-154	glutathione S-transferase pi 1	Homo sapiens	299-304	
15479622-6	2004	Our findings suggest that the GSTP1 polymorphism influences individual susceptibility to breast cancer in the Korean women and this effect may be modified by alcohol consumption.	Alcohols	158-165	glutathione S-transferase pi 1	Homo sapiens	30-35	
15479622-5	2004	A significant interaction was observed between the GSTP1 genotype and alcohol consumption (p for interaction = 0.01); compared with never-drinking women with Ile/Ile genotype, ever-drinking women with the GSTP1 Val allele had almost a three-fold risk of breast cancer (OR = 2.9, 95 % CI = 1.05-7.85), whereas never-drinking women with Val allele had half this risk (OR = 0.5, 95 % CI = 0.27-0.93).	Alcohols	70-77	glutathione S-transferase pi 1	Homo sapiens	205-210	
32650499-13	2020	Interactions between polymorphic variants As3MT*GSTM1 and GSTO2*GSTP1 could be potential modifiers of urinary excretion of arsenic and covariates as age, LADD, and alcohol consumption contribute to largely vary the arsenic individual metabolic capacity in exposed people.	Alcohols	164-171	glutathione S-transferase pi 1	Homo sapiens	64-69	
26680864-6	2002	When the data were stratified, by the known risk factors of breast cancer, a significant interaction was observed between the GSTP1 genotype and alcohol consumption (p for interaction = 0.01); women with GSTP1 Val allele, that drank regularly, had a 3.0-fold increased risk of breast cancer (95% CI=1.1~7.9), whereas women with GSTP1 Val allele, that never drink, had protective effects (OR=0.4, 95% CI=0.2~0.8).	Alcohols	145-152	glutathione S-transferase pi 1	Homo sapiens	126-131	
26680864-6	2002	When the data were stratified, by the known risk factors of breast cancer, a significant interaction was observed between the GSTP1 genotype and alcohol consumption (p for interaction = 0.01); women with GSTP1 Val allele, that drank regularly, had a 3.0-fold increased risk of breast cancer (95% CI=1.1~7.9), whereas women with GSTP1 Val allele, that never drink, had protective effects (OR=0.4, 95% CI=0.2~0.8).	Alcohols	145-152	glutathione S-transferase pi 1	Homo sapiens	204-209	
26680864-6	2002	When the data were stratified, by the known risk factors of breast cancer, a significant interaction was observed between the GSTP1 genotype and alcohol consumption (p for interaction = 0.01); women with GSTP1 Val allele, that drank regularly, had a 3.0-fold increased risk of breast cancer (95% CI=1.1~7.9), whereas women with GSTP1 Val allele, that never drink, had protective effects (OR=0.4, 95% CI=0.2~0.8).	Alcohols	145-152	glutathione S-transferase pi 1	Homo sapiens	204-209	
26680864-7	2002	CONCLUSION: Our findings suggest that GSTP1 Ile105Val polymorphism influences the individual susceptibility to breast cancer, and that this effect may be modified by alcohol consumption.	Alcohols	166-173	glutathione S-transferase pi 1	Homo sapiens	38-43	
11008209-6	2000	Among alcohol drinkers, borderline significance was also found for GSTP1 Ile/Ile genotype (OR 2.0, 95% CI 0.9-4.4).	Alcohols	6-13	glutathione S-transferase pi 1	Homo sapiens	67-72	
31886888-5	2020	The main aim of the current study was to compare the frequency of SNP polymorphisms in XRCC1 (rs7997782) and GSTP1 (rs1695) genes involved in DNA repair of single strand breaks (SSB) and xenobiotic detoxification between alcohol addicts and a control group comprised of non-drinkers.	Alcohols	221-228	glutathione S-transferase pi 1	Homo sapiens	109-114