PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19387251-8	2009	CONCLUSION: The combination of elevated CRP and cTnI increased the risk of adverse cardiac events, demonstrating the additive impacts of active inflammation and myocardial injury on prognosis after SES implantation.	ses	198-201	C-reactive protein	Homo sapiens	40-43	
16923447-9	2006	The same relation was present in the SES group, i.e., patients with a higher CRP level had a higher incidence of 12-month death or myocardial infarction compared with patients with a low CRP level (6.3% vs 1.0%, p = 0.005) and a higher 12-month mortality (5.2% vs 0%, p = 0.001).	ses	37-40	C-reactive protein	Homo sapiens	77-80	
19245380-8	2009	At 24 hours, the mean plasma CRP concentration in the SES group was 20.21 mg/dL versus 8.95 mg/dL in the BMS group (P = 0.15).	ses	54-57	C-reactive protein	Homo sapiens	29-32	
16923447-9	2006	The same relation was present in the SES group, i.e., patients with a higher CRP level had a higher incidence of 12-month death or myocardial infarction compared with patients with a low CRP level (6.3% vs 1.0%, p = 0.005) and a higher 12-month mortality (5.2% vs 0%, p = 0.001).	ses	37-40	C-reactive protein	Homo sapiens	187-190	
16923447-11	2006	In conclusion, PCI in the SES era causes a smaller increase in CRP compared with the BMS era.	ses	26-29	C-reactive protein	Homo sapiens	63-66