PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23207895-8	2012	V cells treated with RLX displayed a similar response to that observed in P cells, suggesting that RLX can modulate the reactivity of the MCs to Ang II.	Relaxin	21-24	angiotensinogen	Rattus norvegicus	145-151	
10021461-7	1999	In contrast, the renal vasoconstrictory response to angiotensin II was attenuated by the RLX treatment.	Relaxin	89-92	angiotensinogen	Rattus norvegicus	52-66	
10021461-10	1999	In conclusion, the renal and osmoregulatory effects of chronic RLX administration to conscious rats resemble the physiological changes of pregnancy in several respects: (a) marked increases in ERPF and GFR with a mediatory role for nitric oxide; (b) attenuation of the renal circulatory response to angiotensin II; and (c) reduction in plasma osmolality.	Relaxin	63-66	angiotensinogen	Rattus norvegicus	299-313	
23207895-8	2012	V cells treated with RLX displayed a similar response to that observed in P cells, suggesting that RLX can modulate the reactivity of the MCs to Ang II.	Relaxin	99-102	angiotensinogen	Rattus norvegicus	145-151	
23207895-10	2012	CONCLUSION: Results suggest that RLX is a mediator of the refractoriness of the MCs to Ang II during pregnancy.	Relaxin	33-36	angiotensinogen	Rattus norvegicus	87-93