PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26613789-4	2016	The results showed that compared with the control group, NaF significantly increased malondialdehyde (MDA) level and reduced catalase (CAT) activity, while no statistical difference was observed in activities and mRNA expressions of superoxide dismutase (SOD), glutathione peroxidase (GPx), and glutathione reductase (GR).	Sodium Fluoride	57-60	catalase	Mus musculus	125-133	
20637250-7	2010	On the other hand, while the plasma MDA level was determined to have increased significantly, erythrocyte SOD and CAT activities were ascertained to have decreased significantly in the group, which was administered sodium fluoride alone on day 30.	Sodium Fluoride	215-230	catalase	Mus musculus	114-117	
20637250-8	2010	In the groups, which were administered N-acetylcysteine prior to sodium fluoride, however, it was observed that, after sodium fluoride administration, plasma MDA levels and erythrocyte SOD and CAT activities drew closer to the values of the control group.	Sodium Fluoride	119-134	catalase	Mus musculus	193-196	
28657544-4	2017	NaF-caused oxidative stress was accompanied by increasing reactive oxygen species (ROS) and malondialdehyde (MDA) levels, and decreasing mRNA expression levels and activities of superoxide dismutase (SOD), catalase (CAT), glutathione (GSH), glutathione peroxidase (GSH-PX) and glutathione-s-transferase (GST).	Sodium Fluoride	0-3	catalase	Mus musculus	206-214	
28657544-4	2017	NaF-caused oxidative stress was accompanied by increasing reactive oxygen species (ROS) and malondialdehyde (MDA) levels, and decreasing mRNA expression levels and activities of superoxide dismutase (SOD), catalase (CAT), glutathione (GSH), glutathione peroxidase (GSH-PX) and glutathione-s-transferase (GST).	Sodium Fluoride	0-3	catalase	Mus musculus	216-219	
18187247-3	2008	The combined treatment of NaF and As(2)O(3) impaired significantly (p<0.001) the production of free radical scavengers such as glutathione and ascorbic acid as well as antioxidant enzymes, namely, glutathione peroxidase (GSH-Px), superoxide dismutase (SOD) and catalase (Cat), thereby increasing ovarian lipid peroxides (LPO) which might have rendered the ovary susceptible to injury.	Sodium Fluoride	26-29	catalase	Mus musculus	264-272	
18187247-3	2008	The combined treatment of NaF and As(2)O(3) impaired significantly (p<0.001) the production of free radical scavengers such as glutathione and ascorbic acid as well as antioxidant enzymes, namely, glutathione peroxidase (GSH-Px), superoxide dismutase (SOD) and catalase (Cat), thereby increasing ovarian lipid peroxides (LPO) which might have rendered the ovary susceptible to injury.	Sodium Fluoride	26-29	catalase	Mus musculus	274-277	
28990137-8	2018	Compared to the control group, the activities of both SOD1 and CAT in testis and epididymis were significantly reduced by 50 or 100 mg NaF exposure.	Sodium Fluoride	135-138	catalase	Mus musculus	63-66	
28990137-9	2018	In addition, the mRNA expressions of testicular SOD1 and CAT were also decreased significantly in 100 mg NaF/L group, while the SOD1 and CAT mRNA expressions in epididymides were significantly reduced in all F treatment groups.	Sodium Fluoride	105-108	catalase	Mus musculus	57-60	
26613789-4	2016	The results showed that compared with the control group, NaF significantly increased malondialdehyde (MDA) level and reduced catalase (CAT) activity, while no statistical difference was observed in activities and mRNA expressions of superoxide dismutase (SOD), glutathione peroxidase (GPx), and glutathione reductase (GR).	Sodium Fluoride	57-60	catalase	Mus musculus	135-138	
22285274-8	2012	Catalase (CAT) treatment significantly inhibited the NaF-mediated cell death and also suppressed the NaF-mediated increase in phospho-c-Jun N-terminal kinase (p-JNK) levels.	Sodium Fluoride	53-56	catalase	Mus musculus	0-8	
22285274-8	2012	Catalase (CAT) treatment significantly inhibited the NaF-mediated cell death and also suppressed the NaF-mediated increase in phospho-c-Jun N-terminal kinase (p-JNK) levels.	Sodium Fluoride	53-56	catalase	Mus musculus	10-13	
22285274-8	2012	Catalase (CAT) treatment significantly inhibited the NaF-mediated cell death and also suppressed the NaF-mediated increase in phospho-c-Jun N-terminal kinase (p-JNK) levels.	Sodium Fluoride	101-104	catalase	Mus musculus	0-8	
22285274-8	2012	Catalase (CAT) treatment significantly inhibited the NaF-mediated cell death and also suppressed the NaF-mediated increase in phospho-c-Jun N-terminal kinase (p-JNK) levels.	Sodium Fluoride	101-104	catalase	Mus musculus	10-13	
22285274-12	2012	Further, NaF-mediated loss of mitochondrial membrane potential was apparently inhibited by pifithrin-alpha or CAT inhibitor.	Sodium Fluoride	9-12	catalase	Mus musculus	110-113	
23201538-11	2013	We found an increase in lipid peroxidation, and catalase activity as well as decrease in glutathione activity (GSH and GST) in liver of mice respectively which were exposed to sodium fluoride.	Sodium Fluoride	176-191	catalase	Mus musculus	48-56