PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32052927-6	2020	Both lnc-MST1P2 silence and miR-133b overexpression could resensitize DDP-resistant bladder cancer cells to DDP treatment.	2-dimethylaminoethyl(dimethylamido)phosphonofluoridate	70-73	macrophage stimulating 1 pseudogene 2	Homo sapiens	9-15	
32052927-6	2020	Both lnc-MST1P2 silence and miR-133b overexpression could resensitize DDP-resistant bladder cancer cells to DDP treatment.	2-dimethylaminoethyl(dimethylamido)phosphonofluoridate	108-111	macrophage stimulating 1 pseudogene 2	Homo sapiens	9-15	
32052927-8	2020	Inc-MST1P2/miR-133b axis affected the resistance of bladder cancer cells to DDP via Sirt1/p53 signaling.	2-dimethylaminoethyl(dimethylamido)phosphonofluoridate	76-79	macrophage stimulating 1 pseudogene 2	Homo sapiens	4-10	
32052927-9	2020	In conclusion, MST1P2 serves as a competing endogenous RNA for miR-133b to counteract miR-133b-induced suppression on Sirt1, therefore enhancing the resistance of bladder cancer cells to DDP.	2-dimethylaminoethyl(dimethylamido)phosphonofluoridate	187-190	macrophage stimulating 1 pseudogene 2	Homo sapiens	15-21	
32052927-10	2020	MST1P2/miR-133b axis affects the resistance of bladder cancer cells to DDP via downstream Sirt1/p53 signaling.	2-dimethylaminoethyl(dimethylamido)phosphonofluoridate	71-74	macrophage stimulating 1 pseudogene 2	Homo sapiens	0-6