PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22217991-3	2011	Afterwards, cells were exposed for 3 days to subtoxic concentrations of lead (Pb, 100 muM) or vanadium (+ 5, V, 2 muM) or cadmium (Cd, 3 muM), slightly reducing, by themselves, cell proliferation and unaffecting cell viability and apoptosis.	Vanadium	94-102	latexin	Homo sapiens	114-117	
22246913-3	2011	After 48 h incubation with 10-40 muM for NaVO3 or 20-40 muM for the other three vanadium salts, the results were statistically significantly lower (0.001 < p < 0.01) as compared to the controls (without vanadium in the medium).	Vanadium	80-88	latexin	Homo sapiens	56-59	
22246913-4	2011	A vanadium concentration higher than 40 muM resulted in cell destruction or death in all cells.	Vanadium	2-10	latexin	Homo sapiens	40-43	
22217991-3	2011	Afterwards, cells were exposed for 3 days to subtoxic concentrations of lead (Pb, 100 muM) or vanadium (+ 5, V, 2 muM) or cadmium (Cd, 3 muM), slightly reducing, by themselves, cell proliferation and unaffecting cell viability and apoptosis.	Vanadium	94-102	latexin	Homo sapiens	114-117	
31552528-8	2019	The results showed that the viability of HUVECs was decreased by treatment with vanadium compounds 50-400 muM in a concentration-dependent manner, while 0.01-1 muM BBR effectively protected HUVECs from the inhibitory effects of vanadium compounds on cell viability.	Vanadium	228-236	latexin	Homo sapiens	160-163	
22725619-10	2012	The presence of zoledronate or vanadium correlated with the most potent activity (IC(50)~1-5 muM) against three human tumor cell lines.	Vanadium	31-39	latexin	Homo sapiens	93-96	
31552528-8	2019	The results showed that the viability of HUVECs was decreased by treatment with vanadium compounds 50-400 muM in a concentration-dependent manner, while 0.01-1 muM BBR effectively protected HUVECs from the inhibitory effects of vanadium compounds on cell viability.	Vanadium	80-88	latexin	Homo sapiens	106-109