PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24955652-4	2014	The results of qPCR and competitive binding test indicated that the expression level of galectin-3 in two metastatic prostate carcinoma cell lines (PC-3 and DU145 cells) could be significantly suppressed by the addition of G3-C12-modified HPMA copolymers (PG1 and PG2), demonstrating the high affinity of PG1 and PG2 to galectin-3.	copolymers	244-254	galectin 3	Homo sapiens	88-98	
26393405-8	2015	Initially, mitochondrial galectin-3 weakened Dox-induced mitochondrial damage; however, as time progressed, G3-C12 active-mediation allowed increasing amounts of Dox to be delivered to the mitochondria, which eventually induced higher level of apoptosis than nontargeted copolymers.	copolymers	271-281	galectin 3	Homo sapiens	25-35	
22189143-2	2012	The aim of this study was to develop a G3-C12-mediated drug delivery system based on N-(2-hydroxypropyl) methacrylamide (HPMA) copolymers targeting to Gal-3-expressed human PC-3 prostate carcinoma cells.	copolymers	127-137	galectin 3	Homo sapiens	151-156	
22074249-3	2011	The targetability of the G3-C12 bearing copolymers towards galectin-3 was further compared to that of galactose-containing copolymers.	copolymers	40-50	galectin 3	Homo sapiens	59-69