PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32260979-2	2013	Here, we engineered the surface of poly(caprolactone) (PCL) scaffolds, first with bone mineral hydroxyapatite (HA), and then with fibronectin-osteocalcin (FN-OCN) bi-functional protein by means of affinity binding between OCN and HA.	polycaprolactone	35-53	fibronectin 1	Rattus norvegicus	130-141	
24657671-0	2014	Umbilical-cord-blood-derived mesenchymal stem cells seeded onto fibronectin-immobilized polycaprolactone nanofiber improve cardiac function.	polycaprolactone	88-104	fibronectin 1	Rattus norvegicus	64-75	
24657671-2	2014	We investigated whether umbilical-cord-blood-derived mesenchymal stem cell (UCB-MSC)-seeded fibronectin (FN)-immobilized polycaprolactone (PCL) nanofibers could improve cardiac function and inhibit left ventricle (LV) remodeling in a rat model of myocardial infarction (MI).	polycaprolactone	121-137	fibronectin 1	Rattus norvegicus	92-103	
24657671-2	2014	We investigated whether umbilical-cord-blood-derived mesenchymal stem cell (UCB-MSC)-seeded fibronectin (FN)-immobilized polycaprolactone (PCL) nanofibers could improve cardiac function and inhibit left ventricle (LV) remodeling in a rat model of myocardial infarction (MI).	polycaprolactone	121-137	fibronectin 1	Rattus norvegicus	105-107	
24657671-2	2014	We investigated whether umbilical-cord-blood-derived mesenchymal stem cell (UCB-MSC)-seeded fibronectin (FN)-immobilized polycaprolactone (PCL) nanofibers could improve cardiac function and inhibit left ventricle (LV) remodeling in a rat model of myocardial infarction (MI).	polycaprolactone	139-142	fibronectin 1	Rattus norvegicus	92-103	
24657671-2	2014	We investigated whether umbilical-cord-blood-derived mesenchymal stem cell (UCB-MSC)-seeded fibronectin (FN)-immobilized polycaprolactone (PCL) nanofibers could improve cardiac function and inhibit left ventricle (LV) remodeling in a rat model of myocardial infarction (MI).	polycaprolactone	139-142	fibronectin 1	Rattus norvegicus	105-107	
24657671-5	2014	Furthermore, genes related to angiogenesis and mesenchymal differentiations were up-regulated in the FN-immobilized PCL nanofibers in comparison to control PCL nanofibers in vitro.	polycaprolactone	116-119	fibronectin 1	Rattus norvegicus	101-103	
24657671-6	2014	4 weeks after the transplantation in the rat MI model, the echocardiogram showed that the UCB-MSC-seeded FN-immobilized PCL nanofiber group increased LV ejection fraction and fraction shortening as compared to the non-treated control and acellular FN-immobilized PCL nanofiber groups.	polycaprolactone	120-123	fibronectin 1	Rattus norvegicus	105-107	
24657671-6	2014	4 weeks after the transplantation in the rat MI model, the echocardiogram showed that the UCB-MSC-seeded FN-immobilized PCL nanofiber group increased LV ejection fraction and fraction shortening as compared to the non-treated control and acellular FN-immobilized PCL nanofiber groups.	polycaprolactone	120-123	fibronectin 1	Rattus norvegicus	248-250	
24657671-6	2014	4 weeks after the transplantation in the rat MI model, the echocardiogram showed that the UCB-MSC-seeded FN-immobilized PCL nanofiber group increased LV ejection fraction and fraction shortening as compared to the non-treated control and acellular FN-immobilized PCL nanofiber groups.	polycaprolactone	263-266	fibronectin 1	Rattus norvegicus	105-107	
24657671-7	2014	Histological analysis indicated that the implantation of UCB-MSCs with FN-immobilized PCL nanofibers induced a decrease in MI size and fibrosis, and an increase in scar thickness.	polycaprolactone	86-89	fibronectin 1	Rattus norvegicus	71-73