PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22686561-4	2012	A critical point is that the LKB1/AMPK network remains functional in a wide range of cancers and could be stimulated by drugs, such as N,N-dimethylimidodicarbonimidic diamide (metformin) or 5-aminoimidazole-4-carboxamide 1-beta-D-ribofuranoside (AICAR).	acadesine	190-244	serine/threonine kinase 11	Homo sapiens	29-33	
16880506-5	2006	In contrast, downregulation of LKB1 did not affect thrombin-induced AMPK activation but abolished phosphorylation of AMPK with 5-aminoimidazole-4-carboxamide ribonucleoside.	acadesine	127-172	serine/threonine kinase 11	Homo sapiens	31-35	
26506235-1	2016	We analyzed the mechanism underlying 5-aminoimidazole-4-carboxamide riboside (AICAR) mediated apoptosis in LKB1-null non-small cell lung cancer (NSCLC) cells.	acadesine	37-76	serine/threonine kinase 11	Homo sapiens	107-111	
26506235-1	2016	We analyzed the mechanism underlying 5-aminoimidazole-4-carboxamide riboside (AICAR) mediated apoptosis in LKB1-null non-small cell lung cancer (NSCLC) cells.	acadesine	78-83	serine/threonine kinase 11	Homo sapiens	107-111	
26506235-6	2016	Furthermore, ectopic expression of LKB1 was capable of attenuating AICAR-induced death in AMPK-null cells.	acadesine	67-72	serine/threonine kinase 11	Homo sapiens	35-39	
22686561-4	2012	A critical point is that the LKB1/AMPK network remains functional in a wide range of cancers and could be stimulated by drugs, such as N,N-dimethylimidodicarbonimidic diamide (metformin) or 5-aminoimidazole-4-carboxamide 1-beta-D-ribofuranoside (AICAR).	acadesine	246-251	serine/threonine kinase 11	Homo sapiens	29-33