PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32718084-0	2020	13-Acetoxysarcocrassolide Exhibits Cytotoxic Activity Against Oral Cancer Cells Through the Interruption of the Keap1/Nrf2/p62/SQSTM1 Pathway: The Need to Move Beyond Classical Concepts.	13-acetoxysarcocrassolide	0-25	sequestosome 1	Homo sapiens	123-126	
32718084-0	2020	13-Acetoxysarcocrassolide Exhibits Cytotoxic Activity Against Oral Cancer Cells Through the Interruption of the Keap1/Nrf2/p62/SQSTM1 Pathway: The Need to Move Beyond Classical Concepts.	13-acetoxysarcocrassolide	0-25	sequestosome 1	Homo sapiens	127-133	
32718084-9	2020	The induction of the Keap1-Nrf2 pathway and the promotion of p62/SQSTM1 were observed in Ca9-22 cells treated with 13-AC.	13-acetoxysarcocrassolide	115-120	sequestosome 1	Homo sapiens	61-64	
32718084-9	2020	The induction of the Keap1-Nrf2 pathway and the promotion of p62/SQSTM1 were observed in Ca9-22 cells treated with 13-AC.	13-acetoxysarcocrassolide	115-120	sequestosome 1	Homo sapiens	65-71	
32718084-10	2020	The knockdown of p62 expression by siRNA transfection significantly attenuated the effect of 13-AC on the inhibition of cell viability.	13-acetoxysarcocrassolide	93-98	sequestosome 1	Homo sapiens	17-20	
32718084-12	2020	The apoptotic effect of 13-AC was found to be mediated through the interruption of the Keap1/Nrf2/p62/SQSTM1 pathway, suggesting its potential future application as an anticancer agent.	13-acetoxysarcocrassolide	24-29	sequestosome 1	Homo sapiens	98-101	
32718084-12	2020	The apoptotic effect of 13-AC was found to be mediated through the interruption of the Keap1/Nrf2/p62/SQSTM1 pathway, suggesting its potential future application as an anticancer agent.	13-acetoxysarcocrassolide	24-29	sequestosome 1	Homo sapiens	102-108