PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 12808005-6 2003 The D1 receptor antagonist R-(+)-7-chloro-8-hydroxy-3-methyl-1-phenyl-2,3,4,5-tetrahydro-1H-3-benzazepine (SCH 23390) increased and the D1 receptor agonist 2,3,4,5-tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine (SKF 33939) decreased anandamide content. 1,2,3,4-tetrahydroisoquinoline-7-sulfonamide 216-219 dopamine receptor D1 Mus musculus 4-15 1974856-3 1990 D1 receptor stimulation with SKF 38393 produced up to 90%, dose-related increases in neostriatal cGMP, whereas D1 antagonism with SCH 23390 decreased cGMP by 30% and blocked the increase induced by SKF 38393. 1,2,3,4-tetrahydroisoquinoline-7-sulfonamide 29-32 dopamine receptor D1 Mus musculus 0-11 1974856-3 1990 D1 receptor stimulation with SKF 38393 produced up to 90%, dose-related increases in neostriatal cGMP, whereas D1 antagonism with SCH 23390 decreased cGMP by 30% and blocked the increase induced by SKF 38393. 1,2,3,4-tetrahydroisoquinoline-7-sulfonamide 198-201 dopamine receptor D1 Mus musculus 0-11 18045908-6 2007 The D1-like agonist SKF 82958 (2,3,4,5,-tetrahydro-6-chloro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine hydrobromide) and the D2-like agonist quinelorane both functioned as positive reinforcers in wild-type mice but not in D1 receptor mutant mice, whereas food and intravenous injections of the opioid agonist remifentanil functioned as positive reinforcers in both genotypes. 1,2,3,4-tetrahydroisoquinoline-7-sulfonamide 20-23 dopamine receptor D1 Mus musculus 219-230 10340822-1 1999 OBJECTIVE: We previously reported that a two week treatment with SKF 38393 (SKF, a dopamine D1 receptor agonist), plus bromocriptine (BC, a dopamine D2 receptor agonist) acted synergistically to normalize hyperphagia, body fat, hyperglycaemia and hyperlipidaemia in ob/ob mice. 1,2,3,4-tetrahydroisoquinoline-7-sulfonamide 65-68 dopamine receptor D1 Mus musculus 83-103 10340822-1 1999 OBJECTIVE: We previously reported that a two week treatment with SKF 38393 (SKF, a dopamine D1 receptor agonist), plus bromocriptine (BC, a dopamine D2 receptor agonist) acted synergistically to normalize hyperphagia, body fat, hyperglycaemia and hyperlipidaemia in ob/ob mice. 1,2,3,4-tetrahydroisoquinoline-7-sulfonamide 76-79 dopamine receptor D1 Mus musculus 83-103