PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28633807-0	2017	Differential induction of ATF3 and HO-1 in myeloid cells and keratinocytes via Dimethylfumarate or Cyclosporine A.	Dimethyl Fumarate	79-95	activating transcription factor 3	Homo sapiens	26-30	
28633807-9	2017	RESULTS: In the present study, we could show that ATF3 is induced in PBMCs by DMF and weakly by Ebselen, while CsA is the most prominent inducer of ATF3 in keratinocytes without enhancing HO-1 transcription.	Dimethyl Fumarate	78-81	activating transcription factor 3	Homo sapiens	50-54	
28633807-13	2017	CONCLUSIONS: Taken together, our results shed light into the different carcinogenic potential of CsA and DMF, which both target ATF3.	Dimethyl Fumarate	105-108	activating transcription factor 3	Homo sapiens	128-132	
28633807-14	2017	Collectively our data demonstrate that CsA strongly induces pro-carcinogenic ATF3 in keratinocytes, whereas ATF3 induction by DMF in myeloid cells acts anti-inflammatory.	Dimethyl Fumarate	126-129	activating transcription factor 3	Homo sapiens	108-112