PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34754326-6	2021	DMF treatment significantly decreased the accumulation of ROS, downregulated inflammatory cytokines (p-NF-kappaB, IL-1beta, and TNF-alpha), and ER stress-associated apoptosis proteins (Bip, calpain-1, caspase-12, caspase-3, and Bax) in LPS-challenged NPCs.	Dimethyl Fumarate	0-3	BCL2 associated X, apoptosis regulator	Homo sapiens	228-231	
25443417-8	2014	In addition, DMF suppressed B-cell lymphoma extra-large (Bcl-xL) and X-linked inhibitor of apoptosis (XIAP) expression whereas Bcl-2, survivin, Bcl-2-associated X protein (Bax), and Bim levels did not change.	Dimethyl Fumarate	13-16	BCL2 associated X, apoptosis regulator	Homo sapiens	172-175	
24112621-6	2013	DMF-mediated apoptosis was associated with increased expression of Bax and Bak and activation of caspase-9 and caspase-6.	Dimethyl Fumarate	0-3	BCL2 associated X, apoptosis regulator	Homo sapiens	67-70	
34025440-3	2021	Our data have demonstrated that DMF treatment attenuated HG-induced apoptosis, as confirmed by reduction of BAX/Bcl-2 ratio.	Dimethyl Fumarate	32-35	BCL2 associated X, apoptosis regulator	Homo sapiens	108-111